Herpes simplex virus 1 U(L)31 and U(L)34 gene products promote the late maturation of viral replication compartments to the nuclear periphery.
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Herpesviruses and intermediate filaments: close encounters with the third typeAlpha-herpesvirus infection induces the formation of nuclear actin filamentsThe Herpes Simplex Virus Protein pUL31 Escorts Nucleocapsids to Sites of Nuclear Egress, a Process Coordinated by Its N-Terminal Domain.Electron tomography of nascent herpes simplex virus virionsHerpes simplex virus 1 induces egress channels through marginalized host chromatinHerpes simplex virus type I disrupts the ATR-dependent DNA-damage response during lytic infection.Role for A-type lamins in herpesviral DNA targeting and heterochromatin modulationRole of chromatin during herpesvirus infections.Viral mimicry of Cdc2/cyclin-dependent kinase 1 mediates disruption of nuclear lamina during human cytomegalovirus nuclear egress.Construction and properties of a herpes simplex virus 2 dl5-29 vaccine candidate strain encoding an HSV-1 virion host shutoff protein.Chromatin at the intersection of viral infection and DNA damageIdentification of an essential domain in the herpes simplex virus 1 UL34 protein that is necessary and sufficient to interact with UL31 protein.Chromatin organization regulates viral egress dynamics.Herpes simplex virus type 1 infection induces activation and recruitment of protein kinase C to the nuclear membrane and increased phosphorylation of lamin B.Herpes simplex virus 1-encoded protein kinase UL13 phosphorylates viral Us3 protein kinase and regulates nuclear localization of viral envelopment factors UL34 and UL31Roles for herpes simplex virus type 1 UL34 and US3 proteins in disrupting the nuclear lamina during herpes simplex virus type 1 egress.U(S)3 and U(S)3.5 protein kinases of herpes simplex virus 1 differ with respect to their functions in blocking apoptosis and in virion maturation and egress.p32 is a novel target for viral protein ICP34.5 of herpes simplex virus type 1 and facilitates viral nuclear egress.Herpes simplex virus ICP0 promotes both histone removal and acetylation on viral DNA during lytic infection.Herpesviral replication compartments move and coalesce at nuclear speckles to enhance export of viral late mRNA.ORF18 is a transfactor that is essential for late gene transcription of a gammaherpesvirus.Roles of the nuclear lamina in stable nuclear association and assembly of a herpesviral transactivator complex on viral immediate-early genes.Herpes simplex virus glycoproteins gB and gH function in fusion between the virion envelope and the outer nuclear membrane.US3 of herpes simplex virus type 1 encodes a promiscuous protein kinase that phosphorylates and alters localization of lamin A/C in infected cellsHerpes simplex virus 1 VP22 regulates translocation of multiple viral and cellular proteins and promotes neurovirulence.Emerin is hyperphosphorylated and redistributed in herpes simplex virus type 1-infected cells in a manner dependent on both UL34 and US3.Expression of gamma interferon-dependent genes is blocked independently by virion host shutoff RNase and by US3 protein kinaseEffects of lamin A/C, lamin B1, and viral US3 kinase activity on viral infectivity, virion egress, and the targeting of herpes simplex virus U(L)34-encoded protein to the inner nuclear membraneNuclear pore composition and gating in herpes simplex virus-infected cellsChromatin control of herpes simplex virus lytic and latent infection.Selective uptake of small RNA molecules in the virion of murine gammaherpesvirus 68.Herpesvirus gB-induced fusion between the virion envelope and outer nuclear membrane during virus egress is regulated by the viral US3 kinase.Gene Editing for Treatment of Neurological Infections.Expression and characterization of the UL31 protein from duck enteritis virusNuclear Exodus: Herpesviruses Lead the Way.Herpes Simplex Virus-Type1 (HSV-1) Impairs DNA Repair in Cortical Neurons.Role of tegument proteins in herpesvirus assembly and egress.Herpesviruses remodel host membranes for virus egress.Breach of the nuclear lamina during assembly of herpes simplex viruses.Nuclear actin and lamins in viral infections.
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P2860
Herpes simplex virus 1 U(L)31 and U(L)34 gene products promote the late maturation of viral replication compartments to the nuclear periphery.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
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2004年學術文章
@zh-hant
name
Herpes simplex virus 1 U(L)31 ...... ents to the nuclear periphery.
@en
type
label
Herpes simplex virus 1 U(L)31 ...... ents to the nuclear periphery.
@en
prefLabel
Herpes simplex virus 1 U(L)31 ...... ents to the nuclear periphery.
@en
P2093
P2860
P1433
P1476
Herpes simplex virus 1 U(L)31 ...... ents to the nuclear periphery.
@en
P2093
David M Knipe
Joel Baines
Martha Simpson-Holley
Richard Roller
P2860
P304
P356
10.1128/JVI.78.11.5591-5600.2004
P407
P577
2004-06-01T00:00:00Z