Differential selectivity of CIITA promoter activation by IFN-gamma and IRF-1 in astrocytes and macrophages: CIITA promoter activation is not affected by TNF-alpha.
about
Interferon-gamma induces major histocompatibility class II transactivator (CIITA), which mediates collagen repression and major histocompatibility class II activation by human aortic smooth muscle cellsClass II transactivator: mastering the art of major histocompatibility complex expressionGlial grafting for demyelinating disease.Self-recognition of the endothelium enables regulatory T-cell trafficking and defines the kinetics of immune regulation.CNS-specific expression of C3a and C5a exacerbate demyelination severity in the cuprizone model.TNF-alpha induces macroautophagy and regulates MHC class II expression in human skeletal muscle cells.Expression of the MHC class II transactivator (CIITA) type IV promoter in B lymphocytes and regulation by IFN-gamma.Genetic analysis of intracranial tumors in a murine model of glioma demonstrate a shift in gene expression in response to host immunity.Maturation of dendritic cells is accompanied by rapid transcriptional silencing of class II transactivator (CIITA) expression.Selective abrogation of major histocompatibility complex class II expression on extrahematopoietic cells in mice lacking promoter IV of the class II transactivator gene.Immune players in the CNS: the astrocyte.Impaired class II transactivator expression in mice lacking interferon regulatory factor-2.Sequential modifications in class II transactivator isoform 1 induced by lipopolysaccharide stimulate major histocompatibility complex class II transcription in macrophages.Inhibition of the JAK/STAT Pathway Protects Against α-Synuclein-Induced Neuroinflammation and Dopaminergic Neurodegeneration.Increased binding of IFN regulating factor 1 mediates the synergistic induction of CIITA by IFN-gamma and tumor necrosis factor-alpha in human thyroid carcinoma cells.Protection from autoimmune brain inflammation in mice lacking IFN-regulatory factor-1 is associated with Th2-type cytokines.Upregulation of transcription factors controlling MHC expression in multiple sclerosis lesions.Malignant glioma cells use MHC class II transactivator (CIITA) promoters III and IV to direct IFN-gamma-inducible CIITA expression and can function as nonprofessional antigen presenting cells in endocytic processing and CD4(+) T-cell activation.
P2860
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P2860
Differential selectivity of CIITA promoter activation by IFN-gamma and IRF-1 in astrocytes and macrophages: CIITA promoter activation is not affected by TNF-alpha.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Differential selectivity of CI ...... is not affected by TNF-alpha.
@en
type
label
Differential selectivity of CI ...... is not affected by TNF-alpha.
@en
prefLabel
Differential selectivity of CI ...... is not affected by TNF-alpha.
@en
P2093
P1476
Differential selectivity of CI ...... is not affected by TNF-alpha.
@en
P2093
Hellendall RP
Nikcevich KM
Piskurich JF
P304
P356
10.1016/S0165-5728(99)00117-4
P577
1999-10-01T00:00:00Z