Establishment of the human papillomavirus type 16 (HPV-16) life cycle in an immortalized human foreskin keratinocyte cell line.
about
Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6Human papillomavirus infection is inhibited by host autophagy in primary human keratinocytesRole of the PDZ domain-binding motif of the oncoprotein E6 in the pathogenesis of human papillomavirus type 31Immortalization of Primary Keratinocytes and Its Application to Skin ResearchEstablishment of human papillomavirus infection requires cell cycle progressionAnalysis of the papillomavirus E2 and bromodomain protein Brd4 interaction using bimolecular fluorescence complementationThe transcription map of human papillomavirus type 18 during genome replication in U2OS cellsThe papillomavirus E1 helicase activates a cellular DNA damage response in viral replication fociReplication and encapsidation of papillomaviruses in Saccharomyces cerevisiaeRecurrent respiratory papillomatosis: an overview of current thinking and treatmentEstablishment of Immortalized Primary Human Foreskin Keratinocytes and Their Application to Toxicity Assessment and Three Dimensional Skin Culture Construction.Variant upstream regulatory region sequences differentially regulate human papillomavirus type 16 DNA replication throughout the viral life cycle.Human papillomavirus E7 oncoprotein overrides the tumor suppressor activity of p21Cip1 in cervical carcinogenesis.High-Risk Human Papillomavirus E7 Alters Host DNA Methylome and Represses HLA-E Expression in Human Keratinocytes.Tumourigenesis driven by the human papillomavirus type 16 Asian-American e6 variant in a three-dimensional keratinocyte modelDisplacement of YY1 by differentiation-specific transcription factor hSkn-1a activates the P(670) promoter of human papillomavirus type 16.Early polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.In vitro progression of human papillomavirus 16 episome-associated cervical neoplasia displays fundamental similarities to integrant-associated carcinogenesis.Production of infectious human papillomavirus independently of viral replication and epithelial cell differentiation.Human papillomavirus 16 variants from Zambian women with normal pap smears.HPV episome levels are potently decreased by pyrrole-imidazole polyamides.A splicing enhancer in the E4 coding region of human papillomavirus type 16 is required for early mRNA splicing and polyadenylation as well as inhibition of premature late gene expression.Human papillomavirus type 16 E1circumflexE4 contributes to multiple facets of the papillomavirus life cycle.Distinct human papillomavirus type 16 methylomes in cervical cells at different stages of premalignancy.Novel antivirals inhibit early steps in HPV infection.Interactions with pocket proteins contribute to the role of human papillomavirus type 16 E7 in the papillomavirus life cycle.Human papillomavirus 16 E6 variants differ in their dysregulation of human keratinocyte differentiation and apoptosis.Genetic analysis of high-risk e6 in episomal maintenance of human papillomavirus genomes in primary human keratinocytes.Saccharomyces cerevisiae is permissive for replication of bovine papillomavirus type 1.Human papillomavirus and cervical cancer.Different modes of human papillomavirus DNA replication during maintenanceQuantitative role of the human papillomavirus type 16 E5 gene during the productive stage of the viral life cycle.Human papillomaviruses and centrosome duplication errors: modeling the origins of genomic instability.Role of Cdk1 in the p53-independent abrogation of the postmitotic checkpoint by human papillomavirus E6Replication and partitioning of papillomavirus genomes.The E7 open reading frame acts in cis and in trans to mediate differentiation-dependent activities in the human papillomavirus type 16 life cycle.Targeting the human papillomavirus E6 and E7 oncogenes through expression of the bovine papillomavirus type 1 E2 protein stimulates cellular motility.The transcription map of HPV11 in U2OS cells adequately reflects the initial and stable replication phases of the viral genome.Influence of physiologic folate deficiency on human papillomavirus type 16 (HPV16)-harboring human keratinocytes in vitro and in vivo.Cervical keratinocytes containing stably replicating extrachromosomal HPV-16 are refractory to transformation by oncogenic H-Ras.
P2860
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P2860
Establishment of the human papillomavirus type 16 (HPV-16) life cycle in an immortalized human foreskin keratinocyte cell line.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Establishment of the human pap ...... reskin keratinocyte cell line.
@en
type
label
Establishment of the human pap ...... reskin keratinocyte cell line.
@en
prefLabel
Establishment of the human pap ...... reskin keratinocyte cell line.
@en
P2093
P356
P1433
P1476
Establishment of the human pap ...... reskin keratinocyte cell line.
@en
P2093
B L Allen-Hoffmann
C A Sattler
E R Flores
P F Lambert
P304
P356
10.1006/VIRO.1999.9868
P407
P577
1999-09-01T00:00:00Z