about
The leading causes of death after burn injury in a single pediatric burn centerLong-term persistance of the pathophysiologic response to severe burn injury.Propranolol decreases cardiac work in a dose-dependent manner in severely burned children.Influence of inhalation injury on energy expenditure in severely burned children.Long-term oxandrolone treatment increases muscle protein net deposition via improving amino acid utilization in pediatric patients 6 months after burn injury.Nutritional and pharmacological modulation of the metabolic response of severely burned patients: review of the literature (part 1).Changes in cardiac physiology after severe burn injurySurvivors versus nonsurvivors postburn: differences in inflammatory and hypermetabolic trajectoriesHistory of burns: The past, present and the future.Modulation of the hypermetabolic response to trauma: temperature, nutrition, and drugsThe hypermetabolic response to burn injury and interventions to modify this responseAbnormal insulin sensitivity persists up to three years in pediatric patients post-burnInsulin resistance postburn: underlying mechanisms and current therapeutic strategies.The hepatic response to thermal injury: is the liver important for postburn outcomes?Is propranolol of benefit in pediatric burn patients?What, how, and how much should patients with burns be fed?Burn injury: review of pathophysiology and therapeutic modalities in major burns.Gene expression profiles and protein balance in skeletal muscle of burned children after beta-adrenergic blockade.
P2860
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P2860
description
1956 nî lūn-bûn
@nan
1956年の論文
@ja
1956年論文
@yue
1956年論文
@zh-hant
1956年論文
@zh-hk
1956年論文
@zh-mo
1956年論文
@zh-tw
1956年论文
@wuu
1956年论文
@zh
1956年论文
@zh-cn
name
The metabolic response to burns
@en
type
label
The metabolic response to burns
@en
prefLabel
The metabolic response to burns
@en
P2093
P356
P1476
The metabolic response to burns
@en
P2093
P2860
P356
10.1172/JCI103253
P407
P577
1956-01-01T00:00:00Z