Mechanism of gram-positive shock: identification of peptidoglycan and lipoteichoic acid moieties essential in the induction of nitric oxide synthase, shock, and multiple organ failure.
about
Recognition of Staphylococcus aureus by the innate immune system.Bacteriophage lysins as effective antibacterialsStaphylococcal peptidoglycans induce arthritisMonoacyl lipoteichoic acid from pneumococci stimulates human cells but not mouse cellsBlood-brain barrier invasion by group B Streptococcus depends upon proper cell-surface anchoring of lipoteichoic acid.Bacillus anthracis peptidoglycan stimulates an inflammatory response in monocytes through the p38 mitogen-activated protein kinase pathway.The role of antimicrobial peptides in animal defenses.Commercial preparations of lipoteichoic acid contain endotoxin that contributes to activation of mouse macrophages in vitroDifferential tumor necrosis factor alpha expression and release from peritoneal mouse macrophages in vitro in response to proliferating gram-positive versus gram-negative bacteria.Enzyme degradation and proinflammatory activity in arthritogenic and nonarthritogenic Eubacterium aerofaciens cell wallsBacteriophage endolysins: a novel anti-infective to control Gram-positive pathogensLipoteichoic acid-induced nitric oxide production depends on the activation of platelet-activating factor receptor and Jak2.Lipoteichoic acid from Staphylococcus aureus induces lung endothelial cell barrier dysfunction: role of reactive oxygen and nitrogen speciesPurified cell wall from the probiotic bacterium Lactobacillus gasseri activates systemic inflammation and, at higher doses, produces lethality in a rat modelModulation of release of proinflammatory bacterial compounds by antibacterials: potential impact on course of inflammation and outcome in sepsis and meningitis.Structure-function relationship of cytokine induction by lipoteichoic acid from Staphylococcus aureus.Toll receptors: a central element in innate immune responsesMechanisms of tissue injury in sepsis: contrasts between gram positive and gram negative infection.Nucleotide oligomerization domain-2 (NOD2)-induced uveitis: dependence on IFN-gamma.Endogenous versus exogenous glucocorticoid responses to experimental bacterial sepsis.Teichoic acids are not required for Streptococcus pneumoniae and Staphylococcus aureus cell walls to trigger the release of tumor necrosis factor by peripheral blood monocytesReceptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Proinflammatory activity of cell-wall constituents from gram-positive bacteria.A continuum of anionic charge: structures and functions of D-alanyl-teichoic acids in gram-positive bacteriaA myeloid hypoxia-inducible factor 1α-Krüppel-like factor 2 pathway regulates gram-positive endotoxin-mediated sepsisImmunotherapeutics for nosocomial infections.Functional Roles of Aromatic Residues and Helices of Papiliocin in its Antimicrobial and Anti-inflammatory Activities.Bacillus anthracis cell wall produces injurious inflammation but paradoxically decreases the lethality of anthrax lethal toxin in a rat model.Evaluation of the immunomodulatory effects of a South African commercial traditional immune booster in human peripheral blood mononuclear cellsAnti-peptidoglycan antibodies and Fcγ receptors are the key mediators of inflammation in Gram-positive sepsisGeneration of inflammatory stimuli: how bacteria set up inflammatory responses in the gingiva.Both anti-TNF and CTLA4 Ig treatments attenuate the disease severity of staphylococcal dermatitis in mice.TCA cycle inactivation in Staphylococcus aureus alters nitric oxide production in RAW 264.7 cells.Systemic infections in multiple sclerosis and experimental autoimmune encephalomyelitis.Bacillus anthracis peptidoglycan activates human platelets through FcγRII and complement.Interaction between periodontitis and liver diseasesLipoprotein lipase and hydrofluoric acid deactivate both bacterial lipoproteins and lipoteichoic acids, but platelet-activating factor-acetylhydrolase degrades only lipoteichoic acidsPeriodontitis as a risk factor of atherosclerosis.Lysophosphatidylcholine reduces the organ injury and dysfunction in rodent models of gram-negative and gram-positive shock.Lipoteichoic acids as a major virulence factor causing inflammatory responses via Toll-like receptor 2.
P2860
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P2860
Mechanism of gram-positive shock: identification of peptidoglycan and lipoteichoic acid moieties essential in the induction of nitric oxide synthase, shock, and multiple organ failure.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Mechanism of gram-positive sho ...... k, and multiple organ failure.
@en
type
label
Mechanism of gram-positive sho ...... k, and multiple organ failure.
@en
prefLabel
Mechanism of gram-positive sho ...... k, and multiple organ failure.
@en
P2093
P2860
P356
P1476
Mechanism of gram-positive sho ...... k, and multiple organ failure.
@en
P2093
C Thiemermann
K M Kengatharan
S De Kimpe
S J Foster
P2860
P304
P356
10.1084/JEM.188.2.305
P407
P577
1998-07-01T00:00:00Z