Tumor necrosis factor-alpha increases ATP content in metabolically inhibited L929 cells preceding cell death.
about
Rat liver myofibroblasts and hepatic stellate cells differ in CD95-mediated apoptosis and response to TNF-alphaPerforin-independent CD8(+) T-cell-mediated cytotoxicity of alveolar epithelial cells is preferentially mediated by tumor necrosis factor-alpha: relative insensitivity to Fas ligand.A role of reactive oxygen species in apoptotic activation of volume-sensitive Cl(-) channel.In vitro viability and cytotoxicity testing and same-well multi-parametric combinations for high throughput screeningMonoubiquitination of nuclear RelA negatively regulates NF-κB activity independent of proteasomal degradationLoss of estrogen-related receptor α promotes hepatocarcinogenesis development via metabolic and inflammatory disturbances.Apoptotic microtubule network organization and maintenance depend on high cellular ATP levels and energized mitochondria.Binding and molecular dynamic studies of sesquiterpenes (2R-acetoxymethyl-1,3,3-trimethyl-4t-(3-methyl-2-buten-1-yl)-1t-cyclohexanol) derived from marine Streptomyces sp. VITJS8 as potential anticancer agent.Tumor necrosis factor-alpha selectively induces MnSOD expression via mitochondria-to-nucleus signaling, whereas interleukin-1beta utilizes an alternative pathway.The effect of heat- and auto-polymerized denture base polymers on clonogenicity, apoptosis, and necrosis in fibroblasts: denture base polymers induce apoptosis and necrosis.Overexpression of catalase in the mitochondrial or cytosolic compartment increases sensitivity of HepG2 cells to tumor necrosis factor-alpha-induced apoptosis.Metabolic effects of photodynamically induced apoptosis in an erythroleukemic cell line. A (31)P NMR spectroscopic study of Victoria-Blue-BO-sensitized TF-1 cells.Long-chain ceramide is a potent inhibitor of the mitochondrial permeability transition pore.Alloxan-induced mitochondrial permeability transition triggered by calcium, thiol oxidation, and matrix ATP.Update on in vitro cytotoxicity assays for drug development.Tumor Necrosis Factor-α Increases the Steady-state Reduction of Cytochromebof the Mitochondrial Respiratory Chain in Metabolically Inhibited L929 Cells
P2860
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P2860
Tumor necrosis factor-alpha increases ATP content in metabolically inhibited L929 cells preceding cell death.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh
1997年學術文章
@zh-hant
name
Tumor necrosis factor-alpha in ...... 29 cells preceding cell death.
@en
type
label
Tumor necrosis factor-alpha in ...... 29 cells preceding cell death.
@en
prefLabel
Tumor necrosis factor-alpha in ...... 29 cells preceding cell death.
@en
P2093
P2860
P50
P356
P1476
Tumor necrosis factor-alpha in ...... 929 cells preceding cell death
@en
P2093
E Siles-Rivas
I Hernández-Muñoz
J A Solís-Herruzo
M T Muñoz-Yagüe
P de la Torre
P2860
P304
30167-30177
P356
10.1074/JBC.272.48.30167
P407
P577
1997-11-01T00:00:00Z