Defining the regulatory networks for muscle development.
about
Cardiovascular basic helix loop helix factor 1, a novel transcriptional repressor expressed preferentially in the developing and adult cardiovascular systemInvolvement of Ras and Ral in chemotactic migration of skeletal myoblastsPC4 coactivates MyoD by relieving the histone deacetylase 4-mediated inhibition of myocyte enhancer factor 2C.Sumoylated SnoN represses transcription in a promoter-specific mannerA novel synaptobrevin/VAMP homologous protein (VAMP5) is increased during in vitro myogenesis and present in the plasma membraneEvolution of Na,K-ATPase beta m-subunit into a coregulator of transcription in placental mammalsMuscle LIM protein promotes myogenesis by enhancing the activity of MyoDRIP2, a checkpoint in myogenic differentiation.Myogenic basic helix-loop-helix proteins and Sp1 interact as components of a multiprotein transcriptional complex required for activity of the human cardiac alpha-actin promoterAn artificial cell-cycle inhibitor isolated from a combinatorial libraryThe bHLH transcription factor Mist1 is required to maintain exocrine pancreas cell organization and acinar cell identityThe basic helix-loop-helix transcription factors dHAND and eHAND exhibit dimerization characteristics that suggest complex regulation of functionHES6 acts as a transcriptional repressor in myoblasts and can induce the myogenic differentiation programTime- and exercise-dependent gene regulation in human skeletal muscleEndocrine regulation of fetal skeletal muscle growth: impact on future metabolic healthNew roles for old enzymes: killer caspases as the engine of cell behavior changesRegulation of myogenic terminal differentiation by the hairy-related transcription factor CHF2TGF-β-activated kinase 1 (TAK1) and apoptosis signal-regulating kinase 1 (ASK1) interact with the promyogenic receptor Cdo to promote myogenic differentiation via activation of p38MAPK pathwaySyndecan-1 expression is down-regulated during myoblast terminal differentiation. Modulation by growth factors and retinoic acidThe transcription factors GATA4 and dHAND physically interact to synergistically activate cardiac gene expression through a p300-dependent mechanismDisruption of striated preferentially expressed gene locus leads to dilated cardiomyopathy in miceThe homeodomain protein Barx2 promotes myogenic differentiation and is regulated by myogenic regulatory factorsMyogenin induces a shift of enzyme activity from glycolytic to oxidative metabolism in muscles of transgenic miceEsophageal muscle physiology and morphogenesis require assembly of a collagen XIX-rich basement membrane zoneMyogenin protein stability is decreased by BMP-2 through a mechanism implicating Id1MyoR: a muscle-restricted basic helix-loop-helix transcription factor that antagonizes the actions of MyoDCooperative synergy between NFAT and MyoD regulates myogenin expression and myogenesisDecoding hematopoietic specificity in the helix-loop-helix domain of the transcription factor SCL/Tal-1CDO, a robo-related cell surface protein that mediates myogenic differentiationRNA-Binding Protein AUF1 Promotes Myogenesis by Regulating MEF2C Expression LevelscDNA cloning and characterization of 5' upstream promoter region of porcine mef2c gene.The histone methyltransferase Set7/9 promotes myoblast differentiation and myofibril assembly.Antisense inhibition of decorin expression in myoblasts decreases cell responsiveness to transforming growth factor beta and accelerates skeletal muscle differentiation.Inferring biological functions and associated transcriptional regulators using gene set expression coherence analysisMyogenic Differentiation from MYOGENIN-Mutated Human iPS Cells by CRISPR/Cas9.Myogenin regulates exercise capacity and skeletal muscle metabolism in the adult mouse.Dysregulation of nuclear receptor COUP-TFII impairs skeletal muscle development.Protein kinase D2 is an essential regulator of murine myoblast differentiation.Synergistic regulation of vertebrate muscle development by Dach2, Eya2, and Six1, homologs of genes required for Drosophila eye formation.Targeted disruption of the MYC antagonist MAD1 inhibits cell cycle exit during granulocyte differentiation.
P2860
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P2860
Defining the regulatory networks for muscle development.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Defining the regulatory networks for muscle development.
@en
type
label
Defining the regulatory networks for muscle development.
@en
prefLabel
Defining the regulatory networks for muscle development.
@en
P1476
Defining the regulatory networks for muscle development
@en
P2093
P304
P356
10.1016/S0959-437X(96)80066-9
P577
1996-08-01T00:00:00Z