Antibodies against granule proteins activate neutrophils in vitro.
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Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in miceThe Prevalence and Management of Pauci-Immune Glomerulonephritis and Vasculitis in Western CountriesPauci-Immune Crescentic Glomerulonephritis: An ANCA-Associated VasculitisThe role of myeloperoxidase and myeloperoxidase-antineutrophil cytoplasmic antibodies (MPO-ANCAs) in the pathogenesis of human MPO-ANCA-associated glomerulonephritisAnimal models of anti-neutrophil cytoplasmic antibody-associated vasculitisLack of effect of antineutrophil cytoplasmic antibodies associated with ulcerative colitis on superoxide anion production from neutrophilsRare diseases.3: Wegener's granulomatosisRare diseases bullet 11: Churg-Strauss syndromeThe immunopathology of ANCA-associated vasculitisThe role of neutrophils and NETosis in autoimmune and renal diseases.p38MAPK, ERK and PI3K signaling pathways are involved in C5a-primed neutrophils for ANCA-mediated activation.Anti-lactoferrin antibodies and other types of ANCA in ulcerative colitis, primary sclerosing cholangitis, and Crohn's disease.The interaction between C5a and sphingosine-1-phosphate in neutrophils for antineutrophil cytoplasmic antibody mediated activation.A pro-inflammatory role of C5L2 in C5a-primed neutrophils for ANCA-induced activation.The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies.High mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4.Monocyte activation in patients with Wegener's granulomatosis.Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodiesIdentification of 4-Trimethylaminobutyraldehyde Dehydrogenase (TMABA-DH) as a Candidate Serum Autoantibody Target for Kawasaki Disease.Autoantibodies developing to myeloperoxidase and proteinase 3 in systemic vasculitis stimulate neutrophil cytotoxicity toward cultured endothelial cellsB cell epitope specificity in ANCA-associated vasculitis: does it matter?Antineutrophil cytoplasmic autoantibodies interact with primary granule constituents on the surface of apoptotic neutrophils in the absence of neutrophil primingAntineutrophil cytoplasmic antibodies induce monocyte IL-8 release. Role of surface proteinase-3, alpha1-antitrypsin, and Fcgamma receptors.Role of neutrophils in systemic autoimmune diseasesExperimental models of vasculitis and glomerulonephritis induced by antineutrophil cytoplasmic autoantibodies.Pathogenesis of antineutrophil cytoplasmic autoantibody vasculitis.How anti-neutrophil cytoplasmic autoantibodies activate neutrophils.Pathogenesis of antineutrophil cytoplasmic autoantibody-associated small-vessel vasculitis.B cell-mediated pathogenesis of ANCA-mediated vasculitis.Comparative effects of antilactoferrin antibodies and tumor necrosis factor on neutrophil adherence to matrix proteins.Neutrophil activation by anti-proteinase 3 antibodies in Wegener's granulomatosis: role of exogenous arachidonic acid and leukotriene B4 generation.Wegener's granulomatosis: anti-proteinase 3 antibodies are potent inductors of human endothelial cell signaling and leakage response.Drug-induced neutropenia associated with anti-neutrophil cytoplasmic antibodies (ANCA): possible involvement of complement in granulocyte cytotoxicity.Interference of PR3-ANCA with the enzymatic activity of PR3: differences in patients during active disease or remission of Wegener's granulomatosis.Selective killing of B-cell hybridomas targeting proteinase 3, Wegener's autoantigen.T lymphocyte responses to anti-neutrophil cytoplasmic autoantibody (ANCA) antigens are present in patients with ANCA-associated systemic vasculitis and persist during disease remission.The C3aR promotes macrophage infiltration and regulates ANCA production but does not affect glomerular injury in experimental anti-myeloperoxidase glomerulonephritis.Complement Factor H Inhibits Anti-Neutrophil Cytoplasmic Autoantibody-Induced Neutrophil Activation by Interacting With Neutrophils.Impaired granulocyte oxidative burst and decreased expression of leucocyte adhesion molecule-1 (LAM-1) in patients with Wegener's granulomatosis.Anti-neutrophil extracellular trap antibody in a patient with relapse of anti-neutrophil cytoplasmic antibody-associated vasculitis: a case report.
P2860
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P2860
Antibodies against granule proteins activate neutrophils in vitro.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Antibodies against granule proteins activate neutrophils in vitro.
@en
type
label
Antibodies against granule proteins activate neutrophils in vitro.
@en
prefLabel
Antibodies against granule proteins activate neutrophils in vitro.
@en
P2093
P1476
Antibodies against granule proteins activate neutrophils in vitro.
@en
P2093
P304
P577
1991-12-01T00:00:00Z