The Microdamage and Expression of Sclerostin in Peri-implant Bone under One-time Shock Force Generated by Impact.

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The Microdamage and Expression of Sclerostin in Peri-implant Bone under One-time Shock Force Generated by Impact.

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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The Microdamage and Expression ...... ock Force Generated by Impact.

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Item

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The Microdamage and Expression ...... ock Force Generated by Impact.

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The Microdamage and Expression ...... ock Force Generated by Impact.

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P1476

The Microdamage and Expression ...... ock Force Generated by Impact.

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P2093

Baili An

http://www.w3.org/2001/XMLSchema#string

Chenyun Dou

http://www.w3.org/2001/XMLSchema#string

Fan Feng

http://www.w3.org/2001/XMLSchema#string

Haitao Xin

http://www.w3.org/2001/XMLSchema#string

Kai Li

http://www.w3.org/2001/XMLSchema#string

Xiaoou Diao

http://www.w3.org/2001/XMLSchema#string

Yulu Wu

http://www.w3.org/2001/XMLSchema#string

Zhirui Li

http://www.w3.org/2001/XMLSchema#string

P2860

Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST)

SOST is a ligand for LRP5/LRP6 and a Wnt signaling inhibitor

Bone dysplasia sclerosteosis results from loss of the SOST gene product, a novel cystine knot-containing protein

Characterization of the Structural Features and Interactions of Sclerostin: MOLECULAR INSIGHT INTO A KEY REGULATOR OF Wnt-MEDIATED BONE FORMATION

Matrix-embedded cells control osteoclast formation

Mechanical stimulation of bone in vivo reduces osteocyte expression of Sost/sclerostin

Wnt but not BMP signaling is involved in the inhibitory action of sclerostin on BMP-stimulated bone formation

Sclerostin is a delayed secreted product of osteocytes that inhibits bone formation

Osteocytes mediate the anabolic actions of canonical Wnt/β-catenin signaling in bone.

Sclerostin stimulates osteocyte support of osteoclast activity by a RANKL-dependent pathway

P2888

s41598-017-06867-9

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scholarly article

P356

10.1038/S41598-017-06867-9

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P407

English

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-07-26T00:00:00Z

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28747741

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P932

5529451

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