TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries. - Wikidata - awgldk - TriplyDB

TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

http://www.wikidata.org/entity/Q41148336

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Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle Disorders The bestrophin- and TMEM16A-associated Ca(2+)- activated Cl(–) channels in vascular smooth muscles Cellular functions of TMEM16/anoctamin Renal autoregulation in health and disease Inhibition of TMEM16A expression suppresses growth and invasion in human colorectal cancer cells Mechanical activation of angiotensin II type 1 receptors causes actin remodelling and myogenic responsiveness in skeletal muscle arterioles.Disruption of vascular Ca2+-activated chloride currents lowers blood pressure.A PLCγ1-dependent, force-sensitive signaling network in the myogenic constriction of cerebral arteries.Evaluation of Arteriolar Smooth Muscle Cell Function in an Ex Vivo Microvascular Network Model.Calcium-calmodulin does not alter the anion permeability of the mouse TMEM16A calcium-activated chloride channel.Vascular TRP channels: performing under pressure and going with the flow Eugenol dilates rat cerebral arteries by inhibiting smooth muscle cell voltage-dependent calcium channels.Modulating Ca²⁺ signals: a common theme for TMEM16, Ist2, and TMC.Calcium-activated chloride channels anoctamin 1 and 2 promote murine uterine smooth muscle contractility.TRPM4 channels couple purinergic receptor mechanoactivation and myogenic tone development in cerebral parenchymal arterioles.Molecular and functional significance of Ca(2+)-activated Cl(-) channels in pulmonary arterial smooth muscle.Membrane potential and Ca2+ concentration dependence on pressure and vasoactive agents in arterial smooth muscle: A model.Anoctamin Calcium-Activated Chloride Channels May Modulate Inhibitory Transmission in the Cerebellar Cortex.Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels.Location, Location, Location: Juxtaposed calcium-signaling microdomains as a novel model of the vascular smooth muscle myogenic response.Ca(V)3.2 channels and the induction of negative feedback in cerebral arteries.Activation of Ca(2+) -activated Cl(-) channel ANO1 by localized Ca(2+) signals.Mechanosensitive Cl- secretion in biliary epithelium mediated through TMEM16A.Regulation of cerebral artery smooth muscle membrane potential by Ca²⁺-activated cation channels.The molecular basis of the genesis of basal tone in internal anal sphincter.βENaC is required for whole cell mechanically gated currents in renal vascular smooth muscle cells.Rab25 influences functional Cav1.2 channel surface expression in arterial smooth muscle cells.Local coupling of TRPC6 to ANO1/TMEM16A channels in smooth muscle cells amplifies vasoconstriction in cerebral arteries.Subdued, a TMEM16 family Ca²⁺-activated Cl⁻channel in Drosophila melanogaster with an unexpected role in host defense Cl⁻ channels in smooth muscle cells.Activation of the Ano1 (TMEM16A) chloride channel by calcium is not mediated by calmodulin.Transport and function of chloride in vascular smooth muscles.T-type Ca²⁺ channels in cerebral arteries: approaches, hypotheses, and speculation.Functions of volume-sensitive and calcium-activated chloride channels.Regulation of vascular tone and arterial blood pressure: role of chloride transport in vascular smooth muscle.TRPV4 Channels Contribute to Renal Myogenic Autoregulation in Neonatal Pigs.Effects of new-generation TMEM16A inhibitors on calcium-activated chloride currents in rabbit urethral interstitial cells of Cajal.Negative News: Cl- and HCO3- in the Vascular Wall.Endothelial-smooth muscle cell interactions in the regulation of vascular tone in skeletal muscle.New Insights on the Regulation of Ca2+ -Activated Chloride Channel TMEM16A.

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P2860

TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

http://www.wikidata.org/entity/Q41148336

description

2012 nî lūn-bûn

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2012年の論文

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2012年论文

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2012年论文

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2012年论文

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name

TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

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type

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TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

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TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

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CIRCRESAHA.112.277145

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Circulation Research

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TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteries.

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Candice M Thomas-Gatewood

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John P Bannister

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Jonathan H Jaggar

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Sarah K Burris

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Simon Bulley

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Wanchana Jangsangthong

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Zachary P Neeb

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P2860

International Union of Pharmacology. XLIX. Nomenclature and structure-function relationships of transient receptor potential channels

Measurement of chloride flux associated with the myogenic response in rat cerebral arteries

TMEM16A/anoctamin 1 protein mediates calcium-activated chloride currents in pulmonary arterial smooth muscle cells

TMEM16A confers receptor-activated calcium-dependent chloride conductance

Excitatory actions of gaba during development: the nature of the nurture

Reversible permeabilization. A novel technique for the intracellular introduction of antisense oligodeoxynucleotides into intact smooth muscle.

Expression cloning of TMEM16A as a calcium-activated chloride channel subunit.

Chloride channels and their functional roles in smooth muscle tone in the vasculature.

Expression profile and protein translation of TMEM16A in murine smooth muscle.

Invited review: arteriolar smooth muscle mechanotransduction: Ca(2+) signaling pathways underlying myogenic reactivity.

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1027-1036

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10.1161/CIRCRESAHA.112.277145

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8

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111

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2012-08-07T00:00:00Z

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22872152

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3666568

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