dNTP pools determine fork progression and origin usage under replication stress. - Wikidata - awgldk - TriplyDB

dNTP pools determine fork progression and origin usage under replication stress.

dNTP pools determine fork progression and origin usage under replication stress.

http://www.wikidata.org/entity/Q41166217

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The FHIT gene product: tumor suppressor and genome "caretaker"Rescuing stalled or damaged replication forks Rad53-Mediated Regulation of Rrm3 and Pif1 DNA Helicases Contributes to Prevention of Aberrant Fork Transitions under Replication Stress.Sen1p contributes to genomic integrity by regulating expression of ribonucleotide reductase 1 (RNR1) in Saccharomyces cerevisiae.ATR-like kinase Mec1 facilitates both chromatin accessibility at DNA replication forks and replication fork progression during replication stress.Replication fork instability and the consequences of fork collisions from rereplication (Ubi)quitin' the h2bit: recent insights into the roles of H2B ubiquitylation in DNA replication and transcription Replication stress in Mammalian cells and its consequences for mitosis Genetic instability in budding and fission yeast-sources and mechanisms APOBEC3A and APOBEC3B Preferentially Deaminate the Lagging Strand Template during DNA Replication Depletion of cellular iron by curcumin leads to alteration in histone acetylation and degradation of Sml1p in Saccharomyces cerevisiae The mutation spectrum in genomic late replication domains shapes mammalian GC content Yeast DNA polymerase ζ maintains consistent activity and mutagenicity across a wide range of physiological dNTP concentrations Domain within the helicase subunit Mcm4 integrates multiple kinase signals to control DNA replication initiation and fork progression.MYC and the control of DNA replication.DNA replication and transcription programs respond to the same chromatin cues.A mammalian-like DNA damage response of fission yeast to nucleoside analogs Causes and consequences of replication stress.SAMHD1 prevents autoimmunity by maintaining genome stability.Continued DNA synthesis in replication checkpoint mutants leads to fork collapse.Rescue from replication stress during mitosis.Single molecule analysis of Trypanosoma brucei DNA replication dynamics.Replication fork integrity and intra-S phase checkpoint suppress gene amplification.Whole genome RNAi screens reveal a critical role of REV3 in coping with replication stress.Increased Rrm2 gene dosage reduces fragile site breakage and prolongs survival of ATR mutant mice.H2B mono-ubiquitylation facilitates fork stalling and recovery during replication stress by coordinating Rad53 activation and chromatin assembly.Endogenous DNA replication stress results in expansion of dNTP pools and a mutator phenotype Singlet Oxygen-Mediated Oxidation during UVA Radiation Alters the Dynamic of Genomic DNA Replication.Slow Replication Fork Velocity of Homologous Recombination-Defective Cells Results from Endogenous Oxidative Stress A Checkpoint-Related Function of the MCM Replicative Helicase Is Required to Avert Accumulation of RNA:DNA Hybrids during S-phase and Ensuing DSBs during G2/M Rtt107 Is a Multi-functional Scaffold Supporting Replication Progression with Partner SUMO and Ubiquitin Ligases.A Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression.Mutations in the Non-Catalytic Subunit Dpb2 of DNA Polymerase Epsilon Affect the Nrm1 Branch of the DNA Replication Checkpoint.The human lagging strand DNA polymerase δ holoenzyme is distributive.Nucleoside salvage pathway kinases regulate hematopoiesis by linking nucleotide metabolism with replication stress.S phase block following MEC1ATR inactivation occurs without severe dNTP depletion.Mec1, INO80, and the PAF1 complex cooperate to limit transcription replication conflicts through RNAPII removal during replication stress Concerted activities of Mcm4, Sld3, and Dbf4 in control of origin activation and DNA replication fork progression Mammalian RAD51 paralogs protect nascent DNA at stalled forks and mediate replication restart.Single-molecule analysis of DNA replication reveals novel features in the divergent eukaryotes Leishmania and Trypanosoma brucei versus mammalian cells.

P2860

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P2860

dNTP pools determine fork progression and origin usage under replication stress.

http://www.wikidata.org/entity/Q41166217

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

dNTP pools determine fork progression and origin usage under replication stress.

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type

label

dNTP pools determine fork progression and origin usage under replication stress.

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dNTP pools determine fork progression and origin usage under replication stress.

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P1433

The EMBO Journal

P1476

dNTP pools determine fork progression and origin usage under replication stress.

@en

P2093

Andrea Keszthelyi

http://www.w3.org/2001/XMLSchema#string

Armelle Lengronne

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Jérôme Poli

http://www.w3.org/2001/XMLSchema#string

Laure Crabbé

http://www.w3.org/2001/XMLSchema#string

Olga Tsaponina

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Véronique Pantesco

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P2860

Galaxy: a comprehensive approach for supporting accessible, reproducible, and transparent computational research in the life sciences

The Integrated Genome Browser: free software for distribution and exploration of genome-scale datasets

OriDB: a DNA replication origin database

Genome-wide analysis of Rad52 foci reveals diverse mechanisms impacting recombination.

NORF5/HUG1 is a component of the MEC1-mediated checkpoint response to DNA damage and replication arrest in Saccharomyces cerevisiae.

The novel DNA damage checkpoint protein ddc1p is phosphorylated periodically during the cell cycle and in response to DNA damage in budding yeast.

The N- and C-termini of Elg1 contribute to the maintenance of genome stability.

The Dun1 checkpoint kinase phosphorylates and regulates the ribonucleotide reductase inhibitor Sml1.

Mrc1 transduces signals of DNA replication stress to activate Rad53.

Mrc1 and Tof1 promote replication fork progression and recovery independently of Rad53.

P304

883-894

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scholarly article

P356

10.1038/EMBOJ.2011.470

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P407

P433

4

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P478

31

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Philippe Pasero

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2012-01-10T00:00:00Z

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P5875

221737553

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22234185

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3280562

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