Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1. - Wikidata - awgldk - TriplyDB

Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.

Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.

http://www.wikidata.org/entity/Q41253971

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MicroRNA Regulation of Brain Tumour Initiating Cells in Central Nervous System Tumours GSK-3 as potential target for therapeutic intervention in cancer Targeting SOX2 as a Therapeutic Strategy in Glioblastoma EMT, CTCs and CSCs in tumor relapse and drug-resistance The enforced expression of c-Myc in pig fibroblasts triggers mesenchymal-epithelial transition (MET) via F-actin reorganization and RhoA/Rock pathway inactivation Loss of miR-638 in vitro promotes cell invasion and a mesenchymal-like transition by influencing SOX2 expression in colorectal carcinoma cells.Actin cytoskeleton regulator Arp2/3 complex is required for DLL1 activating Notch1 signaling to maintain the stem cell phenotype of glioma initiating cells.An integrated analysis of the SOX2 microRNA response program in human pluripotent and nullipotent stem cell lines.High-throughput chemical screens identify disulfiram as an inhibitor of human glioblastoma stem cells.Sox2 suppresses the invasiveness of breast cancer cells via a mechanism that is dependent on Twist1 and the status of Sox2 transcription activity.Autologous anti-SOX2 antibody responses reflect intensity but not frequency of antigen expression in small cell lung cancer.KITENIN promotes glioma invasiveness and progression, associated with the induction of EMT and stemness markers.MiR-203 downregulation is responsible for chemoresistance in human glioblastoma by promoting epithelial-mesenchymal transition via SNAI2 Urokinase-type plasminogen activator receptor (uPAR)-mediated regulation of WNT/β-catenin signaling is enhanced in irradiated medulloblastoma cells.The homing of human cord blood stem cells to sites of inflammation: unfolding mysteries of a novel therapeutic paradigm for glioblastoma multiforme.Serine Protease Activation Essential for Endothelial-Mesenchymal Transition in Vascular Calcification.Autocrine IL-8 promotes F-actin polymerization and mediate mesenchymal transition via ELMO1-NF-κB-Snail signaling in glioma Mutations and deregulation of Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR cascades which alter therapy response.Epigenetic regulation of miRNA-211 by MMP-9 governs glioma cell apoptosis, chemosensitivity and radiosensitivity.Multifunctional roles of urokinase plasminogen activator (uPA) in cancer stemness and chemoresistance of pancreatic cancer.CXXC5 (retinoid-inducible nuclear factor, RINF) is a potential therapeutic target in high-risk human acute myeloid leukemia Human-derived normal mesenchymal stem/stromal cells in anticancer therapies.Clinical significance of putative markers of cancer stem cells in gastric cancer: A retrospective cohort study The hitchhikers guide to cancer stem cell theory: markers, pathways and therapy.Overexpression of microRNA-194 suppresses the epithelial-mesenchymal transition in targeting stem cell transcription factor Sox3 in endometrial carcinoma stem cells.miR-429 inhibits glioma invasion through BMK1 suppression.Down regulation of Akirin-2 increases chemosensitivity in human glioblastomas more efficiently than Twist-1.The Epithelial-to-Mesenchymal Transition-Like Process in Glioblastoma: An Updated Systematic Review and In Silico Investigation.Mesenchymal stem cells enhance the oncolytic effect of Newcastle disease virus in glioma cells and glioma stem cells via the secretion of TRAIL.The EZH2 inhibitor GSK343 suppresses cancer stem-like phenotypes and reverses mesenchymal transition in glioma cells.MiR-21: A key player in glioblastoma pathogenesis.Signaling pathways and mesenchymal transition in pediatric high-grade glioma.CDA-2 induces cell differentiation through suppressing Twist/SLUG signaling via miR-124 in glioma.Conditioned Medium from Adipose-Derived Stem Cells (ADSCs) Promotes Epithelial-to-Mesenchymal-Like Transition (EMT-Like) in Glioma Cells In vitro.Pericytes/vessel-associated mural cells (VAMCs) are the major source of key epithelial-mesenchymal transition (EMT) factors SLUG and TWIST in human glioma.

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Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.

http://www.wikidata.org/entity/Q41253971

description

2011 nî lūn-bûn

@nan

2011年の論文

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2011年学术文章

@wuu

2011年学术文章

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2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

@zh-sg

2011年學術文章

@yue

2011年學術文章

@zh

2011年學術文章

@zh-hant

name

Cord blood stem cells revert g ...... activation of Sox2 and Twist1.

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type

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Cord blood stem cells revert g ...... activation of Sox2 and Twist1.

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Cord blood stem cells revert g ...... activation of Sox2 and Twist1.

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Cord blood stem cells revert g ...... activation of Sox2 and Twist1.

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Andrew J Tsung

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Dzung H Dinh

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Jasti S Rao

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Kiran Kumar Velpula

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Venkata Ramesh Dasari

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P2860

Aberrant expression of SOX2 upregulates MUC5AC gastric foveolar mucin in mucinous cancers of the colorectum and related lesions

The molecular mechanism governing the oncogenic potential of SOX2 in breast cancer

Core transcriptional regulatory circuitry in human embryonic stem cells

Cancerous stem cells can arise from pediatric brain tumors

The epithelial-mesenchymal transition generates cells with properties of stem cells

Epithelial-mesenchymal transitions in tumour progression

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

Identification of human brain tumour initiating cells

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

Epithelial-mesenchymal transition: at the crossroads of development and tumor metastasis

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1028-1042

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10.18632/ONCOTARGET.367

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22184289

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3282065

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