Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.
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MicroRNA Regulation of Brain Tumour Initiating Cells in Central Nervous System TumoursGSK-3 as potential target for therapeutic intervention in cancerTargeting SOX2 as a Therapeutic Strategy in GlioblastomaEMT, CTCs and CSCs in tumor relapse and drug-resistanceThe enforced expression of c-Myc in pig fibroblasts triggers mesenchymal-epithelial transition (MET) via F-actin reorganization and RhoA/Rock pathway inactivationLoss of miR-638 in vitro promotes cell invasion and a mesenchymal-like transition by influencing SOX2 expression in colorectal carcinoma cells.Actin cytoskeleton regulator Arp2/3 complex is required for DLL1 activating Notch1 signaling to maintain the stem cell phenotype of glioma initiating cells.An integrated analysis of the SOX2 microRNA response program in human pluripotent and nullipotent stem cell lines.High-throughput chemical screens identify disulfiram as an inhibitor of human glioblastoma stem cells.Sox2 suppresses the invasiveness of breast cancer cells via a mechanism that is dependent on Twist1 and the status of Sox2 transcription activity.Autologous anti-SOX2 antibody responses reflect intensity but not frequency of antigen expression in small cell lung cancer.KITENIN promotes glioma invasiveness and progression, associated with the induction of EMT and stemness markers.MiR-203 downregulation is responsible for chemoresistance in human glioblastoma by promoting epithelial-mesenchymal transition via SNAI2Urokinase-type plasminogen activator receptor (uPAR)-mediated regulation of WNT/β-catenin signaling is enhanced in irradiated medulloblastoma cells.The homing of human cord blood stem cells to sites of inflammation: unfolding mysteries of a novel therapeutic paradigm for glioblastoma multiforme.Serine Protease Activation Essential for Endothelial-Mesenchymal Transition in Vascular Calcification.Autocrine IL-8 promotes F-actin polymerization and mediate mesenchymal transition via ELMO1-NF-κB-Snail signaling in gliomaMutations and deregulation of Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR cascades which alter therapy response.Epigenetic regulation of miRNA-211 by MMP-9 governs glioma cell apoptosis, chemosensitivity and radiosensitivity.Multifunctional roles of urokinase plasminogen activator (uPA) in cancer stemness and chemoresistance of pancreatic cancer.CXXC5 (retinoid-inducible nuclear factor, RINF) is a potential therapeutic target in high-risk human acute myeloid leukemiaHuman-derived normal mesenchymal stem/stromal cells in anticancer therapies.Clinical significance of putative markers of cancer stem cells in gastric cancer: A retrospective cohort studyThe hitchhikers guide to cancer stem cell theory: markers, pathways and therapy.Overexpression of microRNA-194 suppresses the epithelial-mesenchymal transition in targeting stem cell transcription factor Sox3 in endometrial carcinoma stem cells.miR-429 inhibits glioma invasion through BMK1 suppression.Down regulation of Akirin-2 increases chemosensitivity in human glioblastomas more efficiently than Twist-1.The Epithelial-to-Mesenchymal Transition-Like Process in Glioblastoma: An Updated Systematic Review and In Silico Investigation.Mesenchymal stem cells enhance the oncolytic effect of Newcastle disease virus in glioma cells and glioma stem cells via the secretion of TRAIL.The EZH2 inhibitor GSK343 suppresses cancer stem-like phenotypes and reverses mesenchymal transition in glioma cells.MiR-21: A key player in glioblastoma pathogenesis.Signaling pathways and mesenchymal transition in pediatric high-grade glioma.CDA-2 induces cell differentiation through suppressing Twist/SLUG signaling via miR-124 in glioma.Conditioned Medium from Adipose-Derived Stem Cells (ADSCs) Promotes Epithelial-to-Mesenchymal-Like Transition (EMT-Like) in Glioma Cells In vitro.Pericytes/vessel-associated mural cells (VAMCs) are the major source of key epithelial-mesenchymal transition (EMT) factors SLUG and TWIST in human glioma.
P2860
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P2860
Cord blood stem cells revert glioma stem cell EMT by down regulating transcriptional activation of Sox2 and Twist1.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Cord blood stem cells revert g ...... activation of Sox2 and Twist1.
@en
type
label
Cord blood stem cells revert g ...... activation of Sox2 and Twist1.
@en
prefLabel
Cord blood stem cells revert g ...... activation of Sox2 and Twist1.
@en
P2093
P2860
P356
P1433
P1476
Cord blood stem cells revert g ...... activation of Sox2 and Twist1.
@en
P2093
Andrew J Tsung
Dzung H Dinh
Jasti S Rao
Kiran Kumar Velpula
Venkata Ramesh Dasari
P2860
P304
P356
10.18632/ONCOTARGET.367
P407
P577
2011-12-01T00:00:00Z