Obstruction of extrahepatic bile ducts by lymphocytes is regulated by IFN-gamma in experimental biliary atresia. - Wikidata - awgldk - TriplyDB

Obstruction of extrahepatic bile ducts by lymphocytes is regulated by IFN-gamma in experimental biliary atresia.

Obstruction of extrahepatic bile ducts by lymphocytes is regulated by IFN-gamma in experimental biliary atresia.

http://www.wikidata.org/entity/Q41345437

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Pathogenesis of biliary atresia: defining biology to understand clinical phenotypes B cell deficient mice are protected from biliary obstruction in the rotavirus-induced mouse model of biliary atresia.Loss of interleukin-12 modifies the pro-inflammatory response but does not prevent duct obstruction in experimental biliary atresia.Urokinase-type plasminogen activator supports liver repair independent of its cellular receptor Interactions between Th1 cells and Tregs affect regulation of hepatic fibrosis in biliary atresia through the IFN-γ/STAT1 pathway.Differential expression of hepatic fibrosis mediators in sick and spontaneously recovered mice with experimental biliary atresia High-dose IgG therapy mitigates bile duct-targeted inflammation and obstruction in a mouse model of biliary atresia Identification of intramural epithelial networks linked to peribiliary glands that express progenitor cell markers and proliferate after injury in mice.Loss of guanylyl cyclase C (GCC) signaling leads to dysfunctional intestinal barrier Gene expression signature for biliary atresia and a role for interleukin-8 in pathogenesis of experimental disease Nuclear receptors: mediators and modifiers of inflammation-induced cholestasis.Biliary wound healing, ductular reactions, and IL-6/gp130 signaling in the development of liver disease Biliary atresia: will blocking inflammation tame the disease?Staging of biliary atresia at diagnosis by molecular profiling of the liver Genome-wide association study identifies a susceptibility locus for biliary atresia on 10q24.2.Silencing of the rotavirus NSP4 protein decreases the incidence of biliary atresia in murine model Expansion of prominin-1-expressing cells in association with fibrosis of biliary atresia Macrophages are targeted by rotavirus in experimental biliary atresia and induce neutrophil chemotaxis by Mip2/Cxcl2.Systemic rotavirus infection.The pathogenesis of biliary atresia: evidence for a virus-induced autoimmune disease.DNA hypomethylation causes bile duct defects in zebrafish and is a distinguishing feature of infantile biliary atresia.Integrative genomics identifies candidate microRNAs for pathogenesis of experimental biliary atresia.HMGB1-promoted and TLR2/4-dependent NK cell maturation and activation take part in rotavirus-induced murine biliary atresia.Osteopontin upregulation in rotavirus-induced murine biliary atresia requires replicating virus but is not necessary for development of biliary atresia.Th2 signals induce epithelial injury in mice and are compatible with the biliary atresia phenotype.Natural killer cells promote long-term hepatobiliary inflammation in a low-dose rotavirus model of experimental biliary atresia Effect of rotavirus strain on the murine model of biliary atresia Recent advances in the pathogenesis and management of biliary atresia MicroRNA profiling identifies miR-29 as a regulator of disease-associated pathways in experimental biliary atresia What Causes Biliary Atresia? Unique Aspects of the Neonatal Immune System Provide Clues to Disease Pathogenesis Cytomegalovirus-specific T-cell reactivity in biliary atresia at the time of diagnosis is associated with deficits in regulatory T cells.Cellular and humoral autoimmunity directed at bile duct epithelia in murine biliary atresia.Armed CD4+ Th1 effector cells and activated macrophages participate in bile duct injury in murine biliary atresia.Methylation Microarray Studies Highlight PDGFA Expression as a Factor in Biliary Atresia Progressive biliary destruction is independent of a functional tumor necrosis factor-alpha pathway in a rhesus rotavirus-induced murine model of biliary atresia Effector role of neonatal hepatic CD8+ lymphocytes in epithelial injury and autoimmunity in experimental biliary atresia Rhesus rotavirus VP4 sequence-specific activation of mononuclear cells is associated with cholangiopathy in murine biliary atresia.Unraveling the pathogenesis and etiology of biliary atresia.Rotavirus infection of human cholangiocytes parallels the murine model of biliary atresia Potential etiologies of biliary atresia.

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P2860

Obstruction of extrahepatic bile ducts by lymphocytes is regulated by IFN-gamma in experimental biliary atresia.

http://www.wikidata.org/entity/Q41345437

description

2004 nî lūn-bûn

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2004年论文

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2004年论文

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2004年论文

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Obstruction of extrahepatic bi ...... experimental biliary atresia.

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Obstruction of extrahepatic bi ...... experimental biliary atresia.

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Obstruction of extrahepatic bi ...... experimental biliary atresia.

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Journal of Clinical Investigation

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Obstruction of extrahepatic bi ...... experimental biliary atresia.

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P2093

Alexander Miethke

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Greg Tiao

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Gregg E Sabla

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Jorge A Bezerra

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Kathleen M Campbell

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Monica M McNeal

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Pranavkumar Shivakumar

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Richard L Ward

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P2860

Chemokine and chemokine receptor interactions provide a mechanism for selective T cell recruitment to specific liver compartments within hepatitis C-infected liver

Loss-of-function mutations in the EGF-CFC gene CFC1 are associated with human left-right laterality defects

Interferon-inducible T cell alpha chemoattractant (I-TAC): a novel non-ELR CXC chemokine with potent activity on activated T cells through selective high affinity binding to CXCR3

Multiple defects of immune cell function in mice with disrupted interferon-gamma genes

Identification, genomic organization, chromosomal mapping and mutation analysis of the human INV gene, the ortholog of a murine gene implicated in left-right axis development and biliary atresia.

Reversal of left-right asymmetry: a situs inversus mutation.

Etiopathogenesis of biliary atresia.

Cytokines and autoimmunity.

Extrahepatic biliary atresia: a disease or a phenotype?

Utilization of sialic acid as a coreceptor is required for reovirus-induced biliary disease

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322-329

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10.1172/JCI200421153

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3

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114

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15286798

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484981

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