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Activation of mu opioid receptors sensitizes transient receptor potential vanilloid type 1 (TRPV1) via β-arrestin-2-mediated cross-talkPersistent Nociception Triggered by Nerve Growth Factor (NGF) Is Mediated by TRPV1 and Oxidative Mechanisms.Regulation of ROS production and vascular function by carbon monoxideRepeat low-level blast exposure increases transient receptor potential vanilloid 1 (TRPV1) and endothelin-1 (ET-1) expression in the trigeminal ganglion.Phosphorylation regulates TRPV1 association with β-arrestin-2.
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Elaine D. Por
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12789976800
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0000-0001-8025-5713
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2000-01-01T00:00:00Z