Muscarinic regulation of Alzheimer's disease amyloid precursor protein secretion and amyloid beta-protein production in human neuronal NT2N cells.
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Drug Development in Alzheimer's Disease: The Contribution of PET and SPECTTrafficking regulation of proteins in Alzheimer's diseaseCholinergic treatments with emphasis on m1 muscarinic agonists as potential disease-modifying agents for Alzheimer's diseaseA predictive in vitro model of the impact of drugs with anticholinergic properties on human neuronal and astrocytic systemsThe copper transport protein Atox1 promotes neuronal survivalSerotonin 5-HT2a and 5-HT2c receptors stimulate amyloid precursor protein ectodomain secretionP2Y2 nucleotide receptor-mediated responses in brain cells.The secretases: enzymes with therapeutic potential in Alzheimer disease.Amyloid precursor protein processing is stimulated by metabotropic glutamate receptors.Implications of amyloid precursor protein and subsequent beta-amyloid production to the pharmacotherapy of Alzheimer's disease.Induction of tumor necrosis factor alpha in human neuronal cells by extracellular human T-cell lymphotropic virus type 1 Tax.Cystatin C Shifts APP Processing from Amyloid-β Production towards Non-Amyloidgenic Pathway in Brain Endothelial Cells.Acetylcholinergic neurotransmission and the beta-amyloid cascade: implications for Alzheimer's disease.Lysophosphatidic acid induces increased BACE1 expression and Aβ formation.Unique neuronal functions of cathepsin L and cathepsin B in secretory vesicles: biosynthesis of peptides in neurotransmission and neurodegenerative disease.Specific mechanism for blood inflow stimulation in brain area prone to Alzheimer's disease lesions.Effects of TNFalpha-converting enzyme inhibition on amyloid beta production and APP processing in vitro and in vivo.Pharmacotherapeutic targets in Alzheimer's diseaseCholinergic modulation of amyloid precursor protein processing with emphasis on M1 muscarinic receptor: perspectives and challenges in treatment of Alzheimer's disease.Wnt signaling: role in Alzheimer disease and schizophrenia.M1 muscarinic acetylcholine receptor in Alzheimer's diseaseCholinergic System and Post-translational Modifications: An Insight on the Role in Alzheimer's DiseasePPARα activation attenuates amyloid-β-dependent neurodegeneration by modulating Endo G and AIF translocation.Development of a human neuronal cell model for human immunodeficiency virus (HIV)-infected macrophage-induced neurotoxicity: apoptosis induced by HIV type 1 primary isolates and evidence for involvement of the Bcl-2/Bcl-xL-sensitive intrinsic apoptoFunctional expression of L-, N-, P/Q-, and R-type calcium channels in the human NT2-N cell line.Mechanisms of spontaneous cytosolic Ca2+ transients in differentiated human neuronal cells.Regulation of amyloid precursor protein secretion by glutamate receptors in human Ntera 2 neurons.Acetylcholinesterase expression in NTera 2 human neuronal cells: a model for developmental expression in the nervous system.Constitutive overactivation of protein kinase C in guinea pig brain increases alpha-secretory APP processing without decreasing beta-amyloid generation.Sortilin, SorCS1b, and SorLA Vps10p sorting receptors, are novel gamma-secretase substrates.Elevated intracellular calcium concentration increases secretory processing of the amyloid precursor protein by a tyrosine phosphorylation-dependent mechanism.Rapid stimulation of amyloid precursor protein release by epidermal growth factor: role of protein kinase C.Bradykinin-induced amyloid precursor protein secretion: a protein kinase C-independent mechanism that is not altered in fibroblasts from patients with sporadic Alzheimer's disease.Cortico-hippocampal APP and NGF levels are dynamically altered by cholinergic muscarinic antagonist or M1 agonist treatment in normal mice.Fructose-derived carbohydrates from Alisma orientalis.Differential expression of LMO4 protein in Alzheimer's disease.Muscarinic therapies in Alzheimer's disease; from palliative treatments to disease modification.Release of beta-amyloid from high-density platelets: implications for Alzheimer's disease pathology.Increased neuronal and glial expression of protein kinase C isoforms in neocortex of transgenic Tg2576 mice with amyloid pathology.Differentiated human NT2-N neurons possess a high intracellular content of myo-inositol.
P2860
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P2860
Muscarinic regulation of Alzheimer's disease amyloid precursor protein secretion and amyloid beta-protein production in human neuronal NT2N cells.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Muscarinic regulation of Alzhe ...... in human neuronal NT2N cells.
@en
type
label
Muscarinic regulation of Alzhe ...... in human neuronal NT2N cells.
@en
prefLabel
Muscarinic regulation of Alzhe ...... in human neuronal NT2N cells.
@en
P2093
P2860
P356
P1476
Muscarinic regulation of Alzhe ...... in human neuronal NT2N cells.
@en
P2093
A M Wertkin
J R Williamson
R J Konrad
R P Yasuda
P2860
P304
P356
10.1074/JBC.270.9.4916
P407
P577
1995-03-01T00:00:00Z