Pathogenesis of Burkitt lymphoma: expression of an activated c-myc oncogene causes the tumorigenic conversion of EBV-infected human B lymphoblasts.
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Coordinated regulation of iron-controlling genes, H-ferritin and IRP2, by c-MYCRox, a novel bHLHZip protein expressed in quiescent cells that heterodimerizes with Max, binds a non-canonical E box and acts as a transcriptional repressor.Myc-Max heterodimers activate a DEAD box gene and interact with multiple E box-related sites in vivoIdentification of putative c-Myc-responsive genes: characterization of rcl, a novel growth-related geneChanging perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agentsThe transcription factor, Nm23H2, binds to and activates the translocated c-myc allele in Burkitt's lymphomac-Myc suppression of miR-23a/b enhances mitochondrial glutaminase expression and glutamine metabolismTransfection of the c-myc oncogene into normal Epstein-Barr virus-harboring B cells results in new phenotypic and functional features resembling those of Burkitt lymphoma cells and normal centroblastsMyc-mediated transformation: the repression connection.Glycyrrhizic acid alters Kaposi sarcoma-associated herpesvirus latency, triggering p53-mediated apoptosis in transformed B lymphocytes.Induction of ectopic Myc target gene JAG2 augments hypoxic growth and tumorigenesis in a human B-cell model.Epstein-Barr virus nuclear antigen 1 does not induce lymphoma in transgenic FVB mice.Overexpression of the human BCL-2 gene product results in growth enhancement of Epstein-Barr virus-immortalized B cells.Growth- and tumor-promoting effects of deregulated BCL2 in human B-lymphoblastoid cells.The Epstein-Barr virus and its association with human cancers.HMG-I/Y, a new c-Myc target gene and potential oncogeneBurkitt's lymphoma: historical background and recent insights into classification and pathogenesis.Burkitt's lymphoma: molecular pathogenesis and treatment.MYC overexpression imposes a nonimmunogenic phenotype on Epstein-Barr virus-infected B cells.Biology and treatment of adult acute lymphoblastic leukemiaConstitutive expression of c-myc oncogene confers hormone independence and enhanced growth-factor responsiveness on cells transformed by human papilloma virus type 16.Cell-specific expression of human beta-galactoside alpha 2,6-sialyltransferase transcripts differing in the 5' untranslated region.Human tumor-associated viruses and new insights into the molecular mechanisms of cancer.Tumour growth inhibition by an imidazoquinoline is associated with c-Myc down-regulation in urothelial cell carcinoma.c-myc hypermutation in Burkitt's lymphoma.The pathogenesis of AIDS lymphomas: a foundation for addressing the challenges of therapy and prevention.Cytogenetic abnormalities in childhood acute lymphoblastic leukemia correlates with clinical features and treatment outcome.Pathogenesis of AIDS-related lymphomas: molecular and histogenetic heterogeneity.Localization of the cell proliferation and apoptosis-associated CAS protein in lymphoid neoplasmsAzidothymidine inhibits NF-kappaB and induces Epstein-Barr virus gene expression in Burkitt lymphomaStrong transcriptional activation of translocated c-myc genes occurs without a strong nearby enhancer or promoterAnalysis of the tumorigenic potential of common marmoset lymphoblastoid cells expressing a constitutively activated c-myc gene.A role for deregulated c-Myc expression in apoptosis of Epstein-Barr virus-immortalized B cells.Cellular interactions in the germinal center: role of adhesion receptors and significance for the pathogenesis of AIDS and malignant lymphoma.Opposite regulation of gene transcription and cell proliferation by c-Myc and Max.Differential inhibition of nerve growth factor responses by purine analogues: correlation with inhibition of a nerve growth factor-activated protein kinase.c-Myc transactivation of LDH-A: implications for tumor metabolism and growth.Expression of an exogenous interleukin 6 gene in human Epstein Barr virus B cells confers growth advantage and in vivo tumorigenicitySomatic mutation and transcriptional deregulation of myc in endemic Burkitt's lymphoma disease: heptamer-nonamer recognition mistakes?Augmentation of retrovirus-induced lymphoid leukosis by Marek's disease herpesviruses in White Leghorn chickens.
P2860
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P2860
Pathogenesis of Burkitt lymphoma: expression of an activated c-myc oncogene causes the tumorigenic conversion of EBV-infected human B lymphoblasts.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Pathogenesis of Burkitt lympho ...... infected human B lymphoblasts.
@en
type
label
Pathogenesis of Burkitt lympho ...... infected human B lymphoblasts.
@en
prefLabel
Pathogenesis of Burkitt lympho ...... infected human B lymphoblasts.
@en
P2093
P1433
P1476
Pathogenesis of Burkitt lympho ...... infected human B lymphoblasts.
@en
P2093
Dalla-Favera R
Lombardi L
Newcomb EW
P304
P356
10.1016/0092-8674(87)90556-3
P407
P577
1987-04-01T00:00:00Z