Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines.
about
Regulation of p16CDKN2 expression and its implications for cell immortalization and senescenceBRAF and FBXW7 (CDC4, FBW7, AGO, SEL10) mutations in distinct subsets of pancreatic cancer: potential therapeutic targetsComprehensive molecular profiling of lung adenocarcinoma.Role of cell cycle regulators in lung carcinogenesisDisrupting the Rb-Raf-1 interaction: a potential therapeutic target for cancer.Requirement for p27(KIP1) in retinoblastoma protein-mediated senescence.A gene-alteration profile of human lung cancer cell lines.CDK4/6 inhibitors have potent activity in combination with pathway selective therapeutic agents in models of pancreatic cancer.Inhibition of T-cell acute lymphoblastic leukemia proliferation in vivo by re-expression of the p16INK4a tumor suppressor geneExpression of p16 gene and Rb protein in gastric carcinoma and their clinicopathological significanceCASZ1, a candidate tumor-suppressor gene, suppresses neuroblastoma tumor growth through reprogramming gene expression.E2F inhibition synergizes with paclitaxel in lung cancer cell linesReduced skin tumor development in cyclin D1-deficient mice highlights the oncogenic ras pathway in vivoClinicopathological significance of p16 gene expression in the surgical treatment of head and neck squamous cell carcinomas.HPV analysis in distinguishing second primary tumors from lung metastases in patients with head and neck squamous cell carcinoma.Active cyclin A-CDK2 complex, a possible critical factor for cell proliferation in human primary lung carcinomas.Expression status of p16 protein is associated with human papillomavirus oncogenic potential in cervical and genital lesionsPrognostic significance of CCND1 (cyclin D1) overexpression in primary resected non-small-cell lung cancer.Frequent alterations of cell cycle regulators in early-stage breast lesions as detected by immunohistochemistryAberrant methylation of p16(INK4a) is an early event in lung cancer and a potential biomarker for early diagnosisExpression, deletion [was deleton] and mutation of p16 gene in human gastric cancerDetection of transcriptionally active high-risk HPV in patients with head and neck squamous cell carcinoma as visualized by a novel E6/E7 mRNA in situ hybridization method.Abnormal expression of CCND1 and RB1 in resection margin epithelia of lung cancer patients.Alterations of the p16-pRb pathway and the chromosome locus 9p21-22 in non-small-cell lung carcinomas: relationship with p53 and MDM2 protein expression.The Rb family contains a conserved cyclin-dependent-kinase-regulated transcriptional repressor motifLoss of functional pRB is not a ubiquitous feature of B-cell malignanciesCyclin D1 expression in non-small-cell lung cancers: its association with altered p53 expression, cell proliferation and clinical outcomeG1 checkpoint protein and p53 abnormalities occur in most invasive transitional cell carcinomas of the urinary bladder.Predictive value of expression of p16INK4A, retinoblastoma and p53 proteins for the prognosis of non-small-cell lung cancers.p16 protein expression is associated with a poor prognosis in squamous cell carcinoma of the lung.Cyclin E, a redundant cyclin in breast cancer.Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients.Epstein-Barr virus infection correlates with the expression of COX-2, p16(INK4A) and p53 in classic Hodgkin lymphoma.Peperomin E reactivates silenced tumor suppressor genes in lung cancer cells by inhibition of DNA methyltransferase.Expression ratio of CCND1 to CDKN2A mRNA predicts RB1 status of cultured cancer cell lines and clinical tumor samples.Frequent aberrant methylation of p16INK4a in primary rat lung tumors.p16INK4 expression is associated with the increased sensitivity of human non-small cell lung cancer cells to DNA topoisomerase I inhibitors.Ectopic expression of dE2F and dDP induces cell proliferation and death in the Drosophila eyeThe genomics of lung adenocarcinoma: opportunities for targeted therapies.Retinoblastoma family proteins have distinct functions in pulmonary epithelial cells in vivo critical for suppressing cell growth and tumorigenesis
P2860
Q24650807-B375978E-F214-4872-97AF-2D4E2F242EF6Q24685106-6BF65D31-5CFE-45BB-89E9-77FF428F6F7DQ28244995-A3D7576B-30DF-47B6-99D9-1DC631AADAC4Q28394676-462E8B4B-BD00-42C7-8157-8C0AB000C34FQ33566343-620EFD20-3F24-4CC5-ADCA-5DCCDA754B21Q33968178-68D4D618-9D5E-440D-AE43-29E3F99EAC21Q33978771-B08DEF8E-38EB-433C-B458-D3F0F6625FA9Q34227791-38718B4E-9F65-47F2-BC07-B60805A707C0Q34786251-EE05CE95-CC0E-4683-9322-1D0028775B6AQ35010835-61BD2AED-1217-40E0-8099-66D81BD58DD5Q35092733-D7465969-4B35-47D0-A52F-4FE58C7F1DF7Q35169158-AF2A9711-637A-4DFA-A7BB-1E65453E5017Q35207717-A10B02B1-0DEA-4296-BD24-42DDE2D91B4DQ35585922-EBB12F2B-8475-4F1E-B581-6253A92F7234Q35740820-38EB5B8A-D9BA-477C-8463-4D06A3890B9DQ35753340-06E97DE8-DF6B-46A8-AF38-C047E6A3B978Q35785959-D5BF8F1C-550C-4CAF-99E5-14762B9B2987Q36134992-396BAB8C-5774-459B-A767-311426BA6C4AQ36292511-D2AC2994-2DF8-4630-A50E-4233ACB0CC82Q36324689-B26A87D2-C609-4441-AAA8-5FEB75E705E9Q36397218-C5BA7797-9290-4D51-9A73-B89BFC5E9165Q36405912-95D60CEF-422E-4F8F-A097-D6DE0EBB70AEQ36422016-94F9E04E-739D-4C52-B2FB-7F5EBB8DA240Q36445614-F57E6B04-AA92-4CA5-863C-5FE5AA74DBFAQ36564503-4D28DA41-7CE2-4228-844F-2CAD75F30A9AQ36617740-51BF2DCD-3EF5-4F3A-A281-8D9A8D366ABDQ36617988-184875F1-9444-4626-A421-8247BAE85AE6Q36618000-1F3E75DA-5A6B-4950-A229-8ED2D7D7ACA4Q36619686-42031E6D-0DAD-4E20-B896-FE8FBDB2770DQ36621070-49DDCBBA-1A80-4BAA-B17B-3E41B9757E0BQ37089382-5A5CC1A8-E3CD-40CF-AE87-817B53FD4FD0Q37230186-864D4FCC-62FA-4F0A-A516-CBA8A67E4079Q37349459-5D3A0ADC-D813-4531-B35F-83E6E8AB2CB9Q38736229-62D0FFD8-AA35-473A-8510-6DFDE2F1F54BQ39568745-0691B5D4-037D-4FE2-8734-F4CEDD48389DQ40021115-F94E6E7A-9706-4C2C-9AA4-C4BAA815EA34Q41073992-9B442169-403C-4025-8EC3-0EE48AF9C4AAQ41075838-BBDA4CCE-A81A-4A72-87AF-F2CFD5A2785CQ41891513-EC84432C-07CA-4EFC-9A18-A527B4C6DD03Q42645431-BD9B000C-268C-4086-AF71-5E74DE0DB833
P2860
Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines.
@en
type
label
Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines.
@en
prefLabel
Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines.
@en
P2093
P1433
P1476
Reciprocal Rb inactivation and p16INK4 expression in primary lung cancers and cell lines
@en
P2093
Edwards CD
Godleski J
Richards W
Rollins BJ
Shapiro GI
Sugarbaker DJ
P304
P407
P577
1995-02-01T00:00:00Z