Ultraviolet light-induced keratinocyte apoptosis: a potential mechanism for the induction of skin lesions and autoantibody production in LE. - Wikidata - awgldk - TriplyDB

Ultraviolet light-induced keratinocyte apoptosis: a potential mechanism for the induction of skin lesions and autoantibody production in LE.

Ultraviolet light-induced keratinocyte apoptosis: a potential mechanism for the induction of skin lesions and autoantibody production in LE.

http://www.wikidata.org/entity/Q41375803

about

Goodpasture antigen-binding protein, the kinase that phosphorylates the goodpasture antigen, is an alternatively spliced variant implicated in autoimmune pathogenesis Pathogenesis of systemic lupus erythematosus IFN-gamma transgenic mice: clues to the pathogenesis of systemic lupus erythematosus?Death, autoantigen modifications, and tolerance.Photosensitivity in cutaneous lupus erythematosus Caspase-mediated cleavage of DNA topoisomerase I at unconventional sites during apoptosis The biological significance of evolution in autoimmune phenomena Rapidly evolving purpuric lesions to massive hemorrhagic bullae, with rapid improvement by Prednisolone: as a coetaneous manifestation of Systemic Lupus Erythematosus: a case report.The role of apoptosis in systemic lupus erythematosus.Abnormal production of pro- and anti-inflammatory cytokines by lupus monocytes in response to apoptotic cells.Cytokines and systemic lupus erythematosus Disruption of the cytoskeleton after apoptosis induction with autoantibodies.The in vivo expression of actin/salt-resistant hyperactive DNase I inhibits the development of anti-ssDNA and anti-histone autoantibodies in a murine model of systemic lupus erythematosus The heterogeneity of juvenile myositis Antimalarial lichenoid tissue reactions in patients with pre-existing lupus erythematosus.Increased risk of systemic lupus erythematosus in 29,000 patients with biopsy-verified celiac disease.Environment and systemic lupus erythematosus: an overview.Delayed apoptotic cell clearance and lupus-like autoimmunity in mice lacking the c-mer membrane tyrosine kinase.Histidyl-tRNA synthetase and asparaginyl-tRNA synthetase, autoantigens in myositis, activate chemokine receptors on T lymphocytes and immature dendritic cells.The cellular location of self-antigen determines the positive and negative selection of autoreactive B cells Seasonal variation in the activity of systemic lupus erythematosus Aminoacyl tRNA synthetases and their connections to disease.Autoantigens in systemic autoimmunity: critical partner in pathogenesis Complement in the immunopathogenesis of rheumatic disease.Cutaneous lupus erythematosus: clinical aspects and molecular pathogenesis.Sweet but dangerous - the role of immunoglobulin G glycosylation in autoimmunity and inflammation.Trigger factors of cutaneous lupus erythematosus: a review of current literature.Defective prevention of immune precipitation in autoimmune diseases is independent of C4A*Q0.Induction of apoptosis in monocytes and lymphocytes by serum from patients with systemic lupus erythematosus - an additional mechanism to increased autoantigen load?Time between onset of apoptosis and release of nucleosomes from apoptotic cells: putative implications for systemic lupus erythematosus.Homozygous single nucleotide polymorphism of the complement C1QA gene is associated with decreased levels of C1q in patients with subacute cutaneous lupus erythematosus.Posttranslational protein modifications, apoptosis, and the bypass of tolerance to autoantigens.Autologous tolerogenic dendritic cells derived from monocytes of systemic lupus erythematosus patients and healthy donors show a stable and immunosuppressive phenotype.Apoptosis of keratinocytes and serum DNase I activity in patients with cutaneous lupus erythematosus: relationship with clinical and immunoserological parameters.CD4 positive peripheral T cells from patients with systemic lupus erythematosus (SLE) are clonally expanded.Prevalence of celiac disease in Iranian patients with rheumatologic disorders

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Ultraviolet light-induced keratinocyte apoptosis: a potential mechanism for the induction of skin lesions and autoantibody production in LE.

http://www.wikidata.org/entity/Q41375803

description

1997 nî lūn-bûn

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1997年論文

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1997年论文

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1997年论文

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1997年论文

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name

Ultraviolet light-induced kera ...... autoantibody production in LE.

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Ultraviolet light-induced kera ...... autoantibody production in LE.

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Ultraviolet light-induced kera ...... autoantibody production in LE.

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Ultraviolet light-induced kera ...... autoantibody production in LE.

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A Rosen

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P2860

In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

How can cryptic epitopes trigger autoimmunity?

Novel packages of viral and self-antigens are generated during apoptosis

The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1 beta-converting enzyme

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

The CED-3/ICE-like protease Mch2 is activated during apoptosis and cleaves the death substrate lamin A

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scholarly article

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10.1177/096120339700600213

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2

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