High levels of oncomiR-21 contribute to the senescence-induced growth arrest in normal human cells and its knock-down increases the replicative lifespan.
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Non-Neuronal Acetylcholine: The Missing Link Between Sepsis, Cancer, and Delirium?Milk--A Nutrient System of Mammalian Evolution Promoting mTORC1-Dependent TranslationHuman miR-221/222 in Physiological and Atherosclerotic Vascular RemodelingExtracellular microRNAs and endothelial hyperglycaemic memory: a therapeutic opportunity?Age-associated pro-inflammatory remodeling and functional phenotype in the heart and large arteriesIdentification of microRNAs specific for high producer CHO cell lines using steady-state cultivation.Circulating MicroRNAs as easy-to-measure aging biomarkers in older breast cancer patients: correlation with chronological age but not with fitness/frailty status.miR-125a-5p impairs endothelial cell angiogenesis in aging mice via RTEF-1 downregulation.MicroRNA-21 regulates hTERT via PTEN in hypertrophic scar fibroblasts.Dependence of intracellular and exosomal microRNAs on viral E6/E7 oncogene expression in HPV-positive tumor cells.Molecular and cellular effects of in vitro shockwave treatment on lymphatic endothelial cells.Mir-23a induces telomere dysfunction and cellular senescence by inhibiting TRF2 expression.Age-Associated Differences in MiRNA Signatures Are Restricted to CD45RO Negative T Cells and Are Associated with Changes in the Cellular Composition, Activation and Cellular AgeingHigh Throughput Sequencing of Extracellular RNA from Human Plasma.Systematic analysis of the p53-related microRNAs in breast cancer revealing their essential roles in the cell cycleLong noncoding RNAs in diseases of aging.MiR-21-5p and miR-126a-3p levels in plasma and circulating angiogenic cells: relationship with type 2 diabetes complications.Identification of microRNAs dysregulated in cellular senescence driven by endogenous genotoxic stress.Identify potential drugs for cardiovascular diseases caused by stress-induced genes in vascular smooth muscle cells.MiR-23a-depressed autophagy is a participant in PUVA- and UVB-induced premature senescence.AU-1 from Agavaceae plants causes transient increase in p21/Cip1 expression in renal adenocarcinoma ACHN cells in an miR-34-dependent manner.Advances in the Application and Impact of MicroRNAs as Therapies for Skin DiseaseIntegrative analysis of miRNA and mRNA paired expression profiling of primary fibroblast derived from diabetic foot ulcers reveals multiple impaired cellular functions.Identification of an lncRNA‑miRNA‑mRNA interaction mechanism in breast cancer based on bioinformatic analysis.Genome-Wide miRNA Screening for Genes Bypassing Oncogene-Induced Senescence.Regulatory Roles of miRNAs in Aging.miR-10a rejuvenates aged human mesenchymal stem cells and improves heart function after myocardial infarction through KLF4.MicroRNAs mediate the senescence-associated decline of NRF2 in endothelial cells.Diosgenin Glucoside Protects against Spinal Cord Injury by Regulating Autophagy and Alleviating Apoptosis
P2860
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P2860
High levels of oncomiR-21 contribute to the senescence-induced growth arrest in normal human cells and its knock-down increases the replicative lifespan.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
High levels of oncomiR-21 cont ...... ases the replicative lifespan.
@en
type
label
High levels of oncomiR-21 cont ...... ases the replicative lifespan.
@en
prefLabel
High levels of oncomiR-21 cont ...... ases the replicative lifespan.
@en
P2093
P2860
P50
P921
P356
P1433
P1476
High levels of oncomiR-21 cont ...... ases the replicative lifespan.
@en
P2093
Barbara Preschitz-Kammerhofer
Carina Schreiner
Hanna Dellago
Johannes Grillari
Lucia Terlecki-Zaniewicz
Martina W-F Chang
Matthias Wieser
Rossella Monteforte
P2860
P304
P356
10.1111/ACEL.12069
P50
P577
2013-04-19T00:00:00Z