c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK.
about
Selective interaction of JNK protein kinase isoforms with transcription factorsHepatocyte nuclear factor 3/fork head homolog 11 is expressed in proliferating epithelial and mesenchymal cells of embryonic and adult tissuesDown-regulation of c-Fos/c-Jun AP-1 dimer activity by sumoylation.Comparative analysis of the ternary complex factors Elk-1, SAP-1a and SAP-2 (ERP/NET)ATF-2 is preferentially activated by stress-activated protein kinases to mediate c-jun induction in response to genotoxic agentsIsolation of an AP-1 repressor by a novel method for detecting protein-protein interactionsMolecular characterization of a novel transcription factor that controls stromelysin expressionPhylogenetic and genomewide analyses suggest a functional relationship between kayak, the Drosophila fos homolog, and fig, a predicted protein phosphatase 2c nested within a kayak intronSchnurri-3 (KRC) interacts with c-Jun to regulate the IL-2 gene in T cellsTargeted therapy for hepatocellular carcinoma: novel agents on the horizonDifferential regulation of leukemia inhibitory factor-stimulated neuronal gene expression by protein phosphatases SHP-1 and SHP-2 through mitogen-activated protein kinase-dependent and -independent pathwaysThe AP-1 repressor, JDP2, is a bona fide substrate for the c-Jun N-terminal kinaseMood stabilizers, glycogen synthase kinase-3beta and cell survivalHIV-1 envelope glycoproteins induce activation of activated protein-1 in CD4+ T cellsExtracellular signal-regulated kinase 7 (ERK7), a novel ERK with a C-terminal domain that regulates its activity, its cellular localization, and cell growth.The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsRas-mediated phosphorylation of a conserved threonine residue enhances the transactivation activities of c-Ets1 and c-Ets2Selective induction of p38 mitogen-activated protein kinase activity following A6H co-stimulation in primary human CD4(+) T cells.Maternal hypothyroxinemia impairs spatial learning and synaptic nature and function in the offspringCIB1 contributes to oncogenic signalling by Ras via modulating the subcellular localisation of sphingosine kinase 1Heterodimerization with different Jun proteins controls c-Fos intranuclear dynamics and distributionTargeting signal transduction pathways to eliminate chemotherapeutic drug resistance and cancer stem cells.Essential role of RSK2 in c-Fos-dependent osteosarcoma development.Activation of the Src/p21ras/Erk pathway by progesterone receptor via cross-talk with estrogen receptor.Helicobacter pylori induces ERK-dependent formation of a phospho-c-Fos c-Jun activator protein-1 complex that causes apoptosis in macrophagesKRAS and YAP1 converge to regulate EMT and tumor survivalSignal transduction and the Ets family of transcription factors.c-Fos as a proapoptotic agent in TRAIL-induced apoptosis in prostate cancer cellsPAK1, a gene that can regulate p53 activity in yeast.Transcriptional regulation of CXC-ELR chemokines KC and MIP-2 in mouse pancreatic acini.Antitumor promotion by phenolic antioxidants: inhibition of AP-1 activity through induction of Fra expression.Transactivation of Fra-1 and consequent activation of AP-1 occur extracellular signal-regulated kinase dependently.Transcription factor activation in response to cutaneous injury: role of AP-1 in reepithelialization.CD69 is a TGF-β/1α,25-dihydroxyvitamin D3 target gene in monocytes.Rotational coupling of the transmembrane and kinase domains of the Neu receptor tyrosine kinase.The role of JNK and p38 MAPK activities in UVA-induced signaling pathways leading to AP-1 activation and c-Fos expression.Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.Uses for JNK: the many and varied substrates of the c-Jun N-terminal kinases.Rho GTPases as therapeutic targets for the treatment of inflammatory diseases.Regulation of keratin expression by retinoids.
P2860
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P2860
c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
c-Fos transcriptional activity ...... ase distinct from JNK and ERK.
@en
type
label
c-Fos transcriptional activity ...... ase distinct from JNK and ERK.
@en
prefLabel
c-Fos transcriptional activity ...... ase distinct from JNK and ERK.
@en
P356
P1433
P1476
c-Fos transcriptional activity ...... ase distinct from JNK and ERK.
@en
P2888
P304
P356
10.1038/371171A0
P407
P577
1994-09-01T00:00:00Z
P6179
1004155267