G0 function of BCL2 and BCL-xL requires BAX, BAK, and p27 phosphorylation by Mirk, revealing a novel role of BAX and BAK in quiescence regulation.
about
Bcl-xL phosphorylation at Ser49 by polo kinase 3 during cell cycle progression and checkpointsMolecular mechanisms involved in farnesol-induced apoptosisEscherichia coli subtilase cytotoxin induces apoptosis regulated by host Bcl-2 family proteins Bax/Bak.The BCL2L1 and PGAM5 axis defines hypoxia-induced receptor-mediated mitophagy.Mirk kinase inhibition blocks the in vivo growth of pancreatic cancer cells.Inactivation of mirk/dyrk1b kinase targets quiescent pancreatic cancer cells.Intermediate progenitors are increased by lengthening of the cell cycle through calcium signaling and p53 expression in human neural progenitorsBCL-xL/MCL-1 inhibition and RARγ antagonism work cooperatively in human HL60 leukemia cells.MicroRNAs and Glucocorticoid-Induced Apoptosis in Lymphoid MalignanciesMirk regulates the exit of colon cancer cells from quiescence.Protect and serve: Bcl-2 proteins as guardians and rulers of cancer cell survival.Biomarkers of therapeutic response to BCL2 antagonists in cancer.Pharmacological inhibition of Bcl-xL sensitizes osteosarcoma to doxorubicin.Cell cycle progression dictates the requirement for BCL2 in natural killer cell survival.Transient arrest in a quiescent state allows ovarian cancer cells to survive suboptimal growth conditions and is mediated by both Mirk/dyrk1b and p130/RB2.Mirk/dyrk1B Kinase in Ovarian Cancer.The pro-apoptotic BAX protein influences cell growth and differentiation from the nucleus in healthy interphasic cells.Bcl-2 delays cell cycle through mitochondrial ATP and ROS.Enantiomerically pure β-dipeptide derivative induces anticancer activity against human hormone-refractory prostate cancer through both PI3K/Akt-dependent and -independent pathways.Transcriptional Profiling of Quiescent Muscle Stem Cells In Vivo.When BAX doesn't kill.Bax, Bcl-2, and Bax/Bcl-2 as prognostic markers in acute myeloid leukemia: are we ready for Bcl-2-directed therapy?Expression of Bcl-2 is a favorable prognostic biomarker in lung squamous cell carcinoma.PGC-1α coordinates with Bcl-2 to control the cell cycle in U251 cells through reducing ROS* #.
P2860
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P2860
G0 function of BCL2 and BCL-xL requires BAX, BAK, and p27 phosphorylation by Mirk, revealing a novel role of BAX and BAK in quiescence regulation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
G0 function of BCL2 and BCL-xL ...... BAK in quiescence regulation.
@en
type
label
G0 function of BCL2 and BCL-xL ...... BAK in quiescence regulation.
@en
prefLabel
G0 function of BCL2 and BCL-xL ...... BAK in quiescence regulation.
@en
P2093
P2860
P356
P1476
G0 function of BCL2 and BCL-xL ...... BAK in quiescence regulation.
@en
P2093
Courtney G Sansam
Elizabeth Yang
Mayda Valentin
Qinghua Cui
Yelena Janumyan
P2860
P304
34108-34120
P356
10.1074/JBC.M806294200
P407
P577
2008-09-25T00:00:00Z