Panning transfected cells for electrophysiological studies.
about
Human RSPO1/R-spondin1 is expressed during early ovary development and augments β-catenin signalingCell membrane expression of cardiac sodium channel Na(v)1.5 is modulated by alpha-actinin-2 interactionA novel mutation in DAX1 causes delayed-onset adrenal insufficiency and incomplete hypogonadotropic hypogonadismNuclear receptor corepressors activate rather than suppress basal transcription of genes that are negatively regulated by thyroid hormoneMechanism of nicotinic acetylcholine receptor cluster formation by rapsynInteraction of batrachotoxin with the local anesthetic receptor site in transmembrane segment IVS6 of the voltage-gated sodium channelModulation of CaV1.2 channels by Mg2+ acting at an EF-hand motif in the COOH-terminal domainIdentification with a recombinant antibody of an inner-ear cytokeratin, a marker for hair-cell differentiation.Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutationStudies of alpha-helicity and intersegmental interactions in voltage-gated Na+ channels: S2D4.Reversed voltage-dependent gating of a bacterial sodium channel with proline substitutions in the S6 transmembrane segment.Molecular determinants of drug access to the receptor site for antiarrhythmic drugs in the cardiac Na+ channel.Role of the C-terminal domain in inactivation of brain and cardiac sodium channelsTruncated K+ channel DNA sequences specifically suppress lymphocyte K+ channel gene expression.Time-resolved rhodopsin activation currents in a unicellular expression system.The Drosophila nicotinic acetylcholine receptor subunits Dα5 and Dα7 form functional homomeric and heteromeric ion channelsThe β1-subunit of Na(v)1.5 cardiac sodium channel is required for a dominant negative effect through α-α interaction.Functional properties and differential neuromodulation of Na(v)1.6 channels.Roscovitine is a proteostasis regulator that corrects the trafficking defect of F508del-CFTR by a CDK-independent mechanismStructure and function of the voltage sensor of sodium channels probed by a beta-scorpion toxin.Direct evidence that scorpion α-toxins (site-3) modulate sodium channel inactivation by hindrance of voltage-sensor movementsA constitutively active mutant of the human lutropin receptor (hLHR-L457R) escapes lysosomal targeting and degradation.Allosteric modulation of Ca2+ channels by G proteins, voltage-dependent facilitation, protein kinase C, and Ca(v)beta subunits.Inhibition of cardiac L-type calcium channels by protein kinase C phosphorylation of two sites in the N-terminal domainStructural Analysis and Deletion Mutagenesis Define Regions of QUIVER/SLEEPLESS that Are Responsible for Interactions with Shaker-Type Potassium Channels and Nicotinic Acetylcholine Receptors.Molecular determinants of inactivation and G protein modulation in the intracellular loop connecting domains I and II of the calcium channel alpha1A subunit.A trapped intracellular cation modulates K+ channel recovery from slow inactivation.Cross talk between activation and slow inactivation gates of Shaker potassium channelsProbing the cavity of the slow inactivated conformation of shaker potassium channels.Glutamine substitution at alanine1649 in the S4-S5 cytoplasmic loop of domain 4 removes the voltage sensitivity of fast inactivation in the human heart sodium channelInactivation and secondary structure in the D4/S4-5 region of the SkM1 sodium channel.Anomalous effect of permeant ion concentration on peak open probability of cardiac Na+ channels.Movement and crevices around a sodium channel S3 segment.DAX-1 inhibits SF-1-mediated transactivation via a carboxy-terminal domain that is deleted in adrenal hypoplasia congenita.Endoplasmic reticulum chaperones stabilize nicotinic receptor subunits and regulate receptor assembly.N-linked glycosylation is required for nicotinic receptor assembly but not for subunit associations with calnexinConstruction of a high-affinity receptor site for dihydropyridine agonists and antagonists by single amino acid substitutions in a non-L-type Ca2+ channelbeta1-integrin cytoplasmic subdomains involved in dominant negative function.Interactions of methoxyacetic acid with androgen receptor.Sodium channel selectivity and conduction: prokaryotes have devised their own molecular strategy.
P2860
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P2860
Panning transfected cells for electrophysiological studies.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Panning transfected cells for electrophysiological studies.
@en
type
label
Panning transfected cells for electrophysiological studies.
@en
prefLabel
Panning transfected cells for electrophysiological studies.
@en
P2093
P1433
P1476
Panning transfected cells for electrophysiological studies.
@en
P2093
B McHendry-Rinde
R F Margolskee
P304
P577
1993-11-01T00:00:00Z