about
The role of chromosomal instability in tumor initiation.Twist modulates breast cancer stem cells by transcriptional regulation of CD24 expressionDifferent mutator phenotypes in Mlh1- versus Pms2-deficient miceIntra-organ variation in age-related mutation accumulation in the mouse.Physical and functional interactions of the tumor suppressor protein p53 and DNA polymerase alpha-primase.Cell cycle control, checkpoint mechanisms, and genotoxic stress.Mutational spectrum analysis of RNase H(35) deficient Saccharomyces cerevisiae using fluorescence-based directed termination PCR.Drug resistance in cancer: principles of emergence and prevention.Negative clonal selection in tumor evolution.Biology of colorectal cancer.A Peptide mimicking a region in proliferating cell nuclear antigen specific to key protein interactions is cytotoxic to breast cancer.Understanding tumor heterogeneity as functional compartments--superorganisms revisited.Mutator phenotypes of yeast strains heterozygous for mutations in the MSH2 gene.CGB and GNRH1 expression analysis as a method of tumor cells metastatic spread detection in patients with gynecological malignances.Development of a 'clickable' non-natural nucleotide to visualize the replication of non-instructional DNA lesions.Embryonic reversions and lineage infidelities in tumour cells: genome-based models and role of genetic instability.Probing DNA bulges with designed helical spirocyclic molecules.Epigenetics and colorectal cancer pathogenesisMolecular changes in the bronchial epithelium of patients with small cell lung cancer.Genomic instability and carcinogenesis: an update.Non-natural nucleotides as probes for the mechanism and fidelity of DNA polymerases.Breast cancer causes and treatment: where are we going wrong?Size Does Matter: Why Polyploid Tumor Cells are Critical Drug Targets in the War on Cancer.The involvement of a Nanog, Klf4 and c-Myc transcriptional circuitry in the intertwining between neoplastic progression and reprogramming.Mutator phenotype induced by aberrant replicationMechanisms of dinucleotide repeat instability in Escherichia coli.Hypoxia and miscoupling between reduced energy efficiency and signaling to cell proliferation drive cancer to grow increasingly faster.Stochastic tunnels in evolutionary dynamics.Effect of cell cycle duration on somatic evolutionary dynamics.Recombination and its roles in DNA repair, cellular immortalization and cancer
P2860
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P2860
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Cancer cells exhibit a mutator phenotype.
@en
type
label
Cancer cells exhibit a mutator phenotype.
@en
prefLabel
Cancer cells exhibit a mutator phenotype.
@en
P1476
Cancer cells exhibit a mutator phenotype.
@en
P2093
P577
1998-01-01T00:00:00Z