RhoA-ROCK Inhibition Reverses Synaptic Remodeling and Motor and Cognitive Deficits Caused by Traumatic Brain Injury.

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RhoA-ROCK Inhibition Reverses Synaptic Remodeling and Motor and Cognitive Deficits Caused by Traumatic Brain Injury.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

@zh-hk

2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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RhoA-ROCK Inhibition Reverses ...... sed by Traumatic Brain Injury.

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Item

label

RhoA-ROCK Inhibition Reverses ...... sed by Traumatic Brain Injury.

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prefLabel

RhoA-ROCK Inhibition Reverses ...... sed by Traumatic Brain Injury.

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P1476

RhoA-ROCK Inhibition Reverses ...... used by Traumatic Brain Injury

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Claudia Robertson

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Karen Firozi

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Mauro Costa-Mattioli

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Mohammad Danish Uddin

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Raymond J Grill

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Shalaka Mulherkar

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Wei Huang

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P2860

Suppression of PKR promotes network excitability and enhanced cognition by interferon-γ-mediated disinhibition

Peripheral delivery of a ROCK inhibitor improves learning and working memory.

Cofilin phosphorylation and actin cytoskeletal dynamics regulated by rho- and Cdc42-activated LIM-kinase 2

Rho activation patterns after spinal cord injury and the role of activated Rho in apoptosis in the central nervous system

Hippocampal Neurophysiologic Changes after Mild Traumatic Brain Injury and Potential Neuromodulation Treatment Approaches

Recovery after brain injury: mechanisms and principles

Animal models of traumatic brain injury

Rho family GTPases: key players in neuronal development, neuronal survival, and neurodegeneration

The small GTPase RhoA is required for proper locomotor circuit assembly

Mechanisms for spatiotemporal regulation of Rho-GTPase signaling at synapses

P2888

s41598-017-11113-3

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scholarly article

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10.1038/S41598-017-11113-3

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English

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K F Tolias

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2017-09-06T00:00:00Z

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28878396

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traumatic brain injury

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5587534

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