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Emerging Roles of BAI Adhesion-GPCRs in Synapse Development and PlasticityThe small GTPase RhoA is required for proper locomotor circuit assemblyMechanisms for spatiotemporal regulation of Rho-GTPase signaling at synapsesThe Rac1-GEF Tiam1 couples the NMDA receptor to the activity-dependent development of dendritic arbors and spinesA new pathway for synthesis of phosphatidylinositol-4,5-bisphosphateType I phosphatidylinositol-4-phosphate 5-kinases synthesize the novel lipids phosphatidylinositol 3,5-bisphosphate and phosphatidylinositol 5-phosphatePolarized signaling endosomes coordinate BDNF-induced chemotaxis of cerebellar precursors.Dynamic control of excitatory synapse development by a Rac1 GEF/GAP regulatory complexThe small GTPases RhoA and Rac1 regulate cerebellar development by controlling cell morphogenesis, migration and foliation.BTK regulates PtdIns-4,5-P2 synthesis: importance for calcium signaling and PI3K activity.αvβ8 integrin interacts with RhoGDI1 to regulate Rac1 and Cdc42 activation and drive glioblastoma cell invasion.Control of synapse development and plasticity by Rho GTPase regulatory proteins.Rac homologues and compartmentalized phosphatidylinositol 4, 5-bisphosphate act in a common pathway to regulate polar pollen tube growth.The Rac-GAP Bcr is a novel regulator of the Par complex that controls cell polarity.Molecular Mechanisms of Dendritic Spine Development and Plasticity.RhoA-ROCK Inhibition Reverses Synaptic Remodeling and Motor and Cognitive Deficits Caused by Traumatic Brain Injury.Cytoskeletal mechanisms for synaptic potentiation.Signal transduction pathways involving the small G proteins rac and Cdc42 and phosphoinositide kinases.Memantine prevents acute radiation-induced toxicities at hippocampal excitatory synapses.RhoGTPases Spread the Word for Synaptic Crosstalk.Type Iα phosphatidylinositol-4-phosphate 5-kinase mediates Rac-dependent actin assemblyLipid kinases are novel effectors of the GTPase Rac1The adhesion-GPCR BAI1 shapes dendritic arbors via Bcr-mediated RhoA activation causing late growth arrestCell type composition and circuit organization of clonally related excitatory neurons in the juvenile mouse neocortexAn autism-linked missense mutation in SHANK3 reveals the modularity of Shank3 functionThe Adhesion-GPCR BAI1 Promotes Excitatory Synaptogenesis by Coordinating Bidirectional Trans-synaptic SignalingThe expanding functional roles and signaling mechanisms of adhesion G protein-coupled receptors
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description
researcher
@en
wetenschapper
@nl
name
K F Tolias
@en
K F Tolias
@nl
type
label
K F Tolias
@en
K F Tolias
@nl
prefLabel
K F Tolias
@en
K F Tolias
@nl
P106
P31
P496
0000-0002-2092-920X