CD8 T cells require gamma interferon to clear borna disease virus from the brain and prevent immune system-mediated neuronal damage.
about
CXCR4 antagonism increases T cell trafficking in the central nervous system and improves survival from West Nile virus encephalitisT cells facilitate recovery from Venezuelan equine encephalitis virus-induced encephalomyelitis in the absence of antibody.Chronic cortical and subcortical pathology with associated neurological deficits ensuing experimental herpes encephalitis.Mice deficient in interferon-gamma or interferon-gamma receptor 1 have distinct inflammatory responses to acute viral encephalomyelitis.IL-1R1 signaling regulates CXCL12-mediated T cell localization and fate within the central nervous system during West Nile Virus encephalitis.The critical role of antigen-presentation-induced cytokine crosstalk in the central nervous system in multiple sclerosis and experimental autoimmune encephalomyelitis.Synergistic roles of antibody and interferon in noncytolytic clearance of Sindbis virus from different regions of the central nervous system.Immune-mediated loss of transgene expression from virally transduced brain cells is irreversible, mediated by IFNγ, perforin, and TNFα, and due to the elimination of transduced cellsAntiviral CD8 T cells recognize borna disease virus antigen transgenically expressed in either neurons or astrocytesInterferon gamma protects neonatal neural stem/progenitor cells during measles virus infection of the brainNeuroimmunology of central nervous system viral infections: the cells, molecules and mechanisms involved.Noncytolytic clearance of sindbis virus infection from neurons by gamma interferon is dependent on Jak/STAT signaling.Interferon-gamma prevents death of bystander neurons during CD8 T cell responses in the brain.Interferon gamma modulation of disease manifestation and the local antibody response to alphavirus encephalomyelitis.Pathogenic potential of borna disease virus lacking the immunodominant CD8 T-cell epitope.Cytolytic effector pathways and IFN-γ help protect against Japanese encephalitis.Assessment of listing and categorisation of animal diseases within the framework of the Animal Health Law (Regulation (EU) No 2016/429): Borna diseaseThe neonatal anti-viral response fails to control measles virus spread in neurons despite interferon-gamma expression and a Th1-like cytokine profile.
P2860
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P2860
CD8 T cells require gamma interferon to clear borna disease virus from the brain and prevent immune system-mediated neuronal damage.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
CD8 T cells require gamma inte ...... stem-mediated neuronal damage.
@en
type
label
CD8 T cells require gamma inte ...... stem-mediated neuronal damage.
@en
prefLabel
CD8 T cells require gamma inte ...... stem-mediated neuronal damage.
@en
P2093
P2860
P1433
P1476
CD8 T cells require gamma inte ...... stem-mediated neuronal damage.
@en
P2093
Axel Pagenstecher
Hanns-Joachim Rziha
Jürgen Hausmann
Karen Baur
Kirsten Richter
Peter Staeheli
P2860
P304
13509-13518
P356
10.1128/JVI.79.21.13509-13518.2005
P407
P577
2005-11-01T00:00:00Z