Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death.

Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death.

Item
http://www.wikidata.org/entity/Q41819003
The NuA4 complex promotes translesion synthesis (TLS)-mediated DNA damage tolerance.Differential genetic interactions between Sgs1, DNA-damage checkpoint components and DNA repair factors in the maintenance of chromosome stability.Biochemical studies of the Saccharomyces cerevisiae Mph1 helicase on junction-containing DNA structuresRad5-dependent DNA repair functions of the Saccharomyces cerevisiae FANCM protein homolog Mph1.A Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression.PARG dysfunction enhances DNA double strand break formation in S-phase after alkylation DNA damage and augments different cell death pathways.Histone H2B mono-ubiquitylation maintains genomic integrity at stalled replication forks.Sgs1 Binding to Rad51 Stimulates Homology-Directed DNA Repair in Saccharomyces cerevisiae.
P2860

Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death.

Item
http://www.wikidata.org/entity/Q41819003
description
2009 nî lūn-bûn
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2009年の論文
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2009年論文
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2009年論文
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2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
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2009年论文
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2009年论文
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2009年论文
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name
Defects in DNA lesion bypass l ...... ents and increased cell death.
@en
type
label
Defects in DNA lesion bypass l ...... ents and increased cell death.
@en
prefLabel
Defects in DNA lesion bypass l ...... ents and increased cell death.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P932
P356
P1433
P1476
Defects in DNA lesion bypass l ...... ents and increased cell death.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P932