Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

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Activation of the receptor tyrosine kinase AXL regulates the immune microenvironment in glioblastoma.

P2860

Q52659572-89292F3E-BBE2-4C3F-8072-A231BA72C17C

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Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

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http://www.wikidata.org/entity/Q41847773

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

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2017年论文

@zh-cn

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Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

@en

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Item

label

Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

@en

prefLabel

Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

@en

P1476

Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL

@en

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Avi Maimon

http://www.w3.org/2001/XMLSchema#string

Ghada Abboud-Jarrous

http://www.w3.org/2001/XMLSchema#string

Mayan Cohen-Elisha

http://www.w3.org/2001/XMLSchema#string

Rakefet Czerninski

http://www.w3.org/2001/XMLSchema#string

Shivam Priya

http://www.w3.org/2001/XMLSchema#string

Stuart Fischman

http://www.w3.org/2001/XMLSchema#string

P2860

axl, a transforming gene isolated from primary human myeloid leukemia cells, encodes a novel receptor tyrosine kinase

AXL mediates resistance to PI3Kα inhibition by activating the EGFR/PKC/mTOR axis in head and neck and esophageal squamous cell carcinomas

Cytokeratin 8/18 expression indicates a poor prognosis in squamous cell carcinomas of the oral cavity

AXL kinase as a novel target for cancer therapy

Identification of the product of growth arrest-specific gene 6 as a common ligand for Axl, Sky, and Mer receptor tyrosine kinases

TAM receptors are pleiotropic inhibitors of the innate immune response

MERTK receptor tyrosine kinase is a therapeutic target in melanoma.

T cell-derived protein S engages TAM receptor signaling in dendritic cells to control the magnitude of the immune response.

Receptor tyrosine kinases, TYRO3, AXL, and MER, demonstrate distinct patterns and complex regulation of ligand-induced activation

AXL mediates resistance to cetuximab therapy

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13986-14002

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scholarly article

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10.18632/ONCOTARGET.14753

http://www.w3.org/2001/XMLSchema#string

P407

English

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Tal Burstyn-Cohen

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2017-02-01T00:00:00Z

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P698

28118606

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P932

5355156

http://www.w3.org/2001/XMLSchema#string

Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

about

P2860

P2860

Protein S drives oral squamous cell carcinoma tumorigenicity through regulation of AXL.

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P1476

P2093

P2860

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P698

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P356

P1433

P1476

P2093

P2860

P304

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P356

P407

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P478

P50

P577

P698

P932