Evidence that nitric oxide does not mediate the hyperpolarization and relaxation to acetylcholine in the rat small mesenteric artery.
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Nitric oxide suppresses cerebral vasomotion by sGC-independent effects on ryanodine receptors and voltage-gated calcium channelsCharacterization of endothelium-derived hyperpolarizing factor in the human forearm microcirculationRole of arachidonic acid lipoxygenase metabolites in the regulation of vascular toneActivation of endothelial cell IK(Ca) with 1-ethyl-2-benzimidazolinone evokes smooth muscle hyperpolarization in rat isolated mesenteric arteryRelaxation to authentic nitric oxide and SIN-1 in rat isolated mesenteric arteries: variable role for smooth muscle hyperpolarizationDefinitive role for natriuretic peptide receptor-C in mediating the vasorelaxant activity of C-type natriuretic peptide and endothelium-derived hyperpolarising factorRegulation of cellular communication by signaling microdomains in the blood vessel wall.Adaptation of myocardial blood flow to increased metabolic demand is not dependent on endothelial vasodilators in the rat heart.Endothelial cell protein kinase G inhibits release of EDHF through a PKG-sensitive cation channel.Fetal growth and consequences for later life.Vasodilator responses to acetylcholine are not mediated by the activation of soluble guanylate cyclase or TRPV4 channels in the rat.Tranilast increases vasodilator response to acetylcholine in rat mesenteric resistance arteries through increased EDHF participation.Cell-cell communication in the vessel wall.Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase.Release of C-type natriuretic peptide accounts for the biological activity of endothelium-derived hyperpolarizing factor.Vascular smooth muscle phenotypic diversity and functionStatins and selective inhibition of Rho kinase protect small conductance calcium-activated potassium channel function (K(Ca)2.3) in cerebral arteries.Endothelium-derived hyperpolarizing factor: is there a novel chemical mediator?EDHF: spreading the influence of the endothelium.Myoendothelial electrical coupling in arteries and arterioles and its implications for endothelium-derived hyperpolarizing factor.The effect of hyperglycaemia on function of rat isolated mesenteric resistance artery.Evidence that mechanisms dependent and independent of nitric oxide mediate endothelium-dependent relaxation to bradykinin in human small resistance-like coronary arteriesA comparison of EDHF-mediated and anandamide-induced relaxations in the rat isolated mesenteric artery.Role of potassium channels in the nitrergic nerve stimulation-induced vasodilatation in the guinea-pig isolated basilar artery.P2U-receptor mediated endothelium-dependent but nitric oxide-independent vascular relaxation.Mechanisms of relaxations of bovine isolated bronchioles by the nitric oxide donor, GEA 3175Evidence that different mechanisms underlie smooth muscle relaxation to nitric oxide and nitric oxide donors in the rabbit isolated carotid artery.Sex differences in the relative contributions of nitric oxide and EDHF to agonist-stimulated endothelium-dependent relaxations in the rat isolated mesenteric arterial bed.ATP-sensitive K+ channels in smooth muscle cells of guinea-pig mesenteric lymphatics: role in nitric oxide and beta-adrenoceptor agonist-induced hyperpolarizations.Enhanced acetylcholine induced relaxation in small mesenteric arteries from pregnant rats: an important role for endothelium-derived hyperpolarizing factor (EDHF).Endothelium-dependent hyperpolarization in resting and depolarized mammary and coronary arteries of guinea-pigs.Control of vascular tone in isolated mesenteric arterial segments from hypertensive patientsEDHF, NO and a prostanoid: hyperpolarization-dependent and -independent relaxation in guinea-pig arteriesAbnormal activation of K(+) channels in aortic smooth muscle of rats with endotoxic shock: electrophysiological and functional evidence.NO and KATP channels underlie endotoxin-induced smooth muscle hyperpolarization in rat mesenteric resistance arteriesThe pore-forming subunit of the K(ATP) channel is an important molecular target for LPS-induced vascular hyporeactivity in vitro.Muscle KATP channels: recent insights to energy sensing and myoprotectionRole of nitric oxide in the autonomic innervation of smooth muscle.Endothelium-dependent relaxation to acetylcholine in the rabbit basilar artery: importance of membrane hyperpolarization.Multiple pathways underlying endothelium-dependent relaxation in the rabbit isolated femoral artery.
P2860
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P2860
Evidence that nitric oxide does not mediate the hyperpolarization and relaxation to acetylcholine in the rat small mesenteric artery.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Evidence that nitric oxide doe ...... e rat small mesenteric artery.
@en
type
label
Evidence that nitric oxide doe ...... e rat small mesenteric artery.
@en
prefLabel
Evidence that nitric oxide doe ...... e rat small mesenteric artery.
@en
P2860
P1476
Evidence that nitric oxide doe ...... e rat small mesenteric artery.
@en
P2093
C J Garland
G A McPherson
P2860
P304
P356
10.1111/J.1476-5381.1992.TB14270.X
P407
P577
1992-02-01T00:00:00Z