NO-independent stimulation or activation of soluble guanylyl cyclase during early reperfusion limits infarct size.
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Widening and Elaboration of Consecutive Research into Therapeutic Antioxidant Enzyme DerivativesPostconditioning signalling in the heart: mechanisms and translatabilityCardioprotective effects of PKG activation by soluble GC activator, BAY 60-2770, in ischemia-reperfusion-injured rat hearts.Soluble guanylate cyclase activation during ischemic injury in mice protects against postischemic inflammation at the mitochondrial level.Novel approaches and opportunities for cardioprotective signaling through 3',5'-cyclic guanosine monophosphate manipulation.The cGMP/PKG pathway as a common mediator of cardioprotection: translatability and mechanism.Obestatin regulates cardiovascular function and promotes cardioprotection through the nitric oxide pathway.BAY 41-2272 Treatment Improves Acetylcholine-Induced Aortic Relaxation in L-NAME Hypertensive Rats.cGMP at the centre of attention: emerging strategies for activating the cardioprotective PKG pathway.
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P2860
NO-independent stimulation or activation of soluble guanylyl cyclase during early reperfusion limits infarct size.
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
@zh-hk
2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
NO-independent stimulation or ...... perfusion limits infarct size.
@en
type
label
NO-independent stimulation or ...... perfusion limits infarct size.
@en
prefLabel
NO-independent stimulation or ...... perfusion limits infarct size.
@en
P2093
P2860
P50
P356
P1476
NO-independent stimulation or ...... perfusion limits infarct size.
@en
P2093
Gary F Baxter
Justin S Bice
Yvonne Keim
P2860
P304
P356
10.1093/CVR/CVT257
P577
2013-11-20T00:00:00Z