The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
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From rapid to delayed and remote postconditioning: the evolving concept of ischemic postconditioning in brain ischemiaPRAS40 plays a pivotal role in protecting against stroke by linking the Akt and mTOR pathwaysDocosahexaenoic acid signaling modulates cell survival in experimental ischemic stroke penumbra and initiates long-term repair in young and aged rats.T cells contribute to stroke-induced lymphopenia in rats.Early exercise improves cerebral blood flow through increased angiogenesis in experimental stroke rat modelPhosphorylated mitogen-activated protein kinase/extracellular signal-regulated kinase 1/2 may not always represent its kinase activity in a rat model of focal cerebral ischemia with or without ischemic preconditioning.The role of Akt (protein kinase B) and protein kinase C in ischemia-reperfusion injury.TLR5 Activation through NF-κB Is a Neuroprotective Mechanism of Postconditioning after Cerebral Ischemia in Mice.Isoflurane delays the development of early brain injury after subarachnoid hemorrhage through sphingosine-related pathway activation in mice.Ischemic preconditioning reduces endoplasmic reticulum stress and upregulates hypoxia inducible factor-1α in ischemic kidney: the role of nitric oxide.Lithium treatment reduces brain injury induced by focal ischemia with partial reperfusion and the protective mechanisms dispute the importance of akt activity.Akt isoforms differentially protect against stroke-induced neuronal injury by regulating mTOR activities.Impact of acellular hemoglobin-based oxygen carriers on brain apoptosis in rats.Repetitive ischemic preconditioning attenuates inflammatory reaction and brain damage after focal cerebral ischemia in rats: involvement of PI3K/Akt and ERK1/2 signaling pathway.Comparative analysis of the neuroprotective effects of ginsenosides Rg1 and Rb1 extracted from Panax notoginseng against cerebral ischemia.
P2860
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P2860
The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
@en
type
label
The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
@en
prefLabel
The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
@en
P2093
P2860
P1476
The Akt pathway is involved in rapid ischemic tolerance in focal ischemia in Rats.
@en
P2093
Gary Steinberg
Hanfeng Zhang
P2860
P2888
P304
P356
10.1007/S12975-010-0017-5
P577
2010-09-01T00:00:00Z