Akt isoforms differentially protect against stroke-induced neuronal injury by regulating mTOR activities.
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PRAS40 plays a pivotal role in protecting against stroke by linking the Akt and mTOR pathwaysPKBγ/AKT3 loss-of-function causes learning and memory deficits and deregulation of AKT/mTORC2 signaling: Relevance for schizophrenia.FGF2/FGFR1 regulates autophagy in FGFR1-amplified non-small cell lung cancer cells.Deficiency of Akt1, but not Akt2, attenuates the development of pulmonary hypertension.Blood brain barrier and neuroinflammation are critical targets of IGF-1-mediated neuroprotection in stroke for middle-aged female rats.Circulating miRNA profiles provide a biomarker for severity of stroke outcomes associated with age and sex in a rat model.MYC Drives Pten/Trp53-Deficient Proliferation and Metastasis due to IL6 Secretion and AKT Suppression via PHLPP2.Exogenous progesterone is neuroprotective following injury to the male zebra finch brain.Specific inhibition of mTOR pathway induces anti-proliferative effect and decreases the hormone secretion in cultured pituitary adenoma cells.Ser/Thr protein kinase Bβ-NADPH oxidase 2 signaling in thromboinflammation.The role of PKA in the translational response to heat stress in Saccharomyces cerevisiae.ConBr, A Lectin Purified from the Seeds of Canavalia brasiliensis, Protects Against Ischemia in Organotypic Culture of Rat Hippocampus: Potential Implication of Voltage-Gated Calcium Channels.The Wnt signaling pathway is differentially expressed during the bovine herpesvirus 1 latency-reactivation cycle: evidence that two protein kinases associated with neuronal survival (Akt3 and bone morphogenetic protein receptor 2) are expressed at hChromium supplementation improved post-stroke brain infarction and hyperglycemia.PUMA mediates the anti-cancer effect of osimertinib in colon cancer cells.Hypoxia Inducible Factor 1α Plays a Key Role in Remote Ischemic Preconditioning Against Stroke by Modulating Inflammatory Responses in Rats.The mTOR cell signaling pathway is crucial to the long-term protective effects of ischemic postconditioning against stroke.
P2860
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P2860
Akt isoforms differentially protect against stroke-induced neuronal injury by regulating mTOR activities.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Akt isoforms differentially pr ...... by regulating mTOR activities.
@en
Akt isoforms differentially pr ...... by regulating mTOR activities.
@nl
type
label
Akt isoforms differentially pr ...... by regulating mTOR activities.
@en
Akt isoforms differentially pr ...... by regulating mTOR activities.
@nl
prefLabel
Akt isoforms differentially pr ...... by regulating mTOR activities.
@en
Akt isoforms differentially pr ...... by regulating mTOR activities.
@nl
P2093
P2860
P356
P1476
Akt isoforms differentially pr ...... by regulating mTOR activities.
@en
P2093
Marcel Daadi
Michelle Cheng
Robert M Sapolsky
P2860
P304
P356
10.1038/JCBFM.2013.132
P577
2013-08-14T00:00:00Z