Regulation of ATP supply during muscle contraction: theoretical studies.
about
A biophysical model of the mitochondrial respiratory system and oxidative phosphorylationEffect of 'binary mitochondrial heteroplasmy' on respiration and ATP synthesis: implications for mitochondrial diseasesInterrelations of ATP synthesis and proton handling in ischaemically exercising human forearm muscle studied by 31P magnetic resonance spectroscopyIntracellular energetic units in red muscle cellsComparative transcriptome analysis to investigate the high starch accumulation of duckweed (Landoltia punctata) under nutrient starvationBuilding the power house: recent advances in mitochondrial studies through proteomics and systems biology.An integrated model of cardiac mitochondrial energy metabolism and calcium dynamics.Permeabilized rat cardiomyocyte response demonstrates intracellular origin of diffusion obstaclesMatching ATP supply and demand in mammalian heart: in vivo, in vitro, and in silico perspectives.Effect of calcium on the oxidative phosphorylation cascade in skeletal muscle mitochondriaHeterogeneity of ADP diffusion and regulation of respiration in cardiac cellsAnalysis of functional coupling: mitochondrial creatine kinase and adenine nucleotide translocase.Systematic construction of kinetic models from genome-scale metabolic networks.'Idealized' state 4 and state 3 in mitochondria vs. rest and work in skeletal muscleA computational model of skeletal muscle metabolism linking cellular adaptations induced by altered loading states to metabolic responses during exercise.Mechanisms of Attenuation of Pulmonary V'O2 Slow Component in Humans after Prolonged Endurance Training.Computational Model of Cellular Metabolic Dynamics in Skeletal Muscle Fibers during Moderate Intensity Exercise.Model analysis of the relationship between intracellular PO2 and energy demand in skeletal muscleRole of NADH/NAD+ transport activity and glycogen store on skeletal muscle energy metabolism during exercise: in silico studiesPhilosophical basis and some historical aspects of systems biology: from Hegel to Noble - applications for bioenergetic research.Intracellular energetic units in healthy and diseased heartsRegulation of mitochondrial Ca2+ and its effects on energetics and redox balance in normal and failing heart.Slow VO₂ kinetics during moderate-intensity exercise as markers of lower metabolic stability and lower exercise tolerance.Mechanistic modeling of aberrant energy metabolism in human disease.The control of brain mitochondrial energization by cytosolic calcium: the mitochondrial gas pedal.Each-step activation of oxidative phosphorylation is necessary to explain muscle metabolic kinetic responses to exercise and recovery in humans.Faster and stronger manifestation of mitochondrial diseases in skeletal muscle than in heart related to cytosolic inorganic phosphate (Pi) accumulation.Contribution of proton leak to oxygen consumption in skeletal muscle during intense exercise is very low despite large contribution at rest.Phosphocreatine recovery overshoot after high intensity exercise in human skeletal muscle is associated with extensive muscle acidification and a significant decrease in phosphorylation potential.Absence of mitochondrial activation during levosimendan inotropic action in perfused paced guinea pig hearts as demonstrated by modular control analysis.Influence of rapid changes in cytosolic pH on oxidative phosphorylation in skeletal muscle: theoretical studies.Training-induced adaptation of oxidative phosphorylation in skeletal muscles.Regulation of oxidative phosphorylation in different muscles and various experimental conditions.Factors determining the oxygen consumption rate (VO2) on-kinetics in skeletal muscles.Control over the contribution of the mitochondrial membrane potential (DeltaPsi) and proton gradient (DeltapH) to the protonmotive force (Deltap). In silico studies.Slow VO2 off-kinetics in skeletal muscle is associated with fast PCr off-kinetics--and inversely.Flux control of sulphate assimilation in Arabidopsis thaliana: adenosine 5'-phosphosulphate reductase is more susceptible than ATP sulphurylase to negative control by thiols.Regulation of oxidative phosphorylation during work transitions results from its kinetic properties.Muscle glycogen depletion and increased oxidative phosphorylation following status epilepticus.Reaction-diffusion constraints in living tissue: effectiveness factors in skeletal muscle design.
P2860
Q24812608-86032808-77F7-466B-8235-9C05980FCE11Q28344441-2BF22F16-F1EE-490D-826D-831E8EB73305Q28354140-56466012-5D3E-4FCF-A799-BC8B3B967759Q28365640-BFE28991-0ED0-4058-965F-CFFD473E2F71Q28703615-5F46A8AA-C68B-4403-A41F-DAB9A0B8A9E7Q33252676-FF253A42-4667-4431-84DE-94DE24A69AF1Q33947602-3C0A58FD-E56F-44B8-A9C4-D6AA6CECF749Q34069998-31BD0D8E-A691-4ADB-AC8E-06AFC4029DE3Q34144264-615CDE55-DB11-4ECA-8E07-4982F9C344ECQ34147741-736272AF-49F4-4382-BE90-0C84DF551AC9Q34181234-6B6DEF3C-2D11-452B-9775-73205B411673Q34186386-4D16476A-914E-4D15-A712-642DAD63EF91Q35063614-0D8B2843-A10D-476A-9046-E7908E4E4032Q35554449-B7E5DC00-43EC-4C92-BC03-4E7A3CDDCF7FQ35793902-2C912DF9-104F-4814-9304-47002DEA7B77Q35996773-37C354E2-5EC9-49CA-B1B9-C409124BDA54Q36199811-C06E83B0-E24B-4743-A8F7-C00C03189FCEQ36656343-C645788C-CA73-4DA2-A179-9DE0732EFD96Q37086464-406D1549-47BF-44C2-90FD-26F6B5E8109BQ37166324-51B8B871-1B1C-4D54-ADA9-9B8B7E6C38C2Q37276512-7895D629-751A-4F1F-8C91-A4AC08AD6216Q37458334-C1793EA8-80DF-4D28-B4E0-F2F945647F25Q37785875-A801CD08-99B5-4EFF-8D00-245A27207E0CQ38056722-B6D58580-6FB1-4E82-8276-9B7774CB857AQ38080810-7D452671-C221-4F02-91B8-CE80468B16C4Q40390777-8E064294-3EA6-4B0E-B17B-DFADE81062C6Q40990028-F8D444BC-9C8F-467E-AFED-ECD2173159D4Q42680474-2FEDE9B3-8438-41B5-9111-42871E3999A8Q42993055-96AAE759-9736-4103-8586-920F13893C1BQ42997884-D7A17798-D22E-4F0A-A8AF-E921BDDA9E3BQ43001972-90557B66-5DE5-4DBF-B7E8-49C3AA89C50BQ43003054-4294F49B-56A9-4FA0-812A-264E600154CBQ43003213-A3CBC0D1-92E1-4172-98E0-C1940C1D2A30Q43003719-1AD69530-5E87-45F4-ABD7-17EFED157F47Q43154648-83ECE189-FED5-420A-935E-203D001E5658Q43961304-44D161A9-13C0-4915-9914-47AFC88F6293Q44130632-24EE9F67-0966-4BC6-9345-F5186D16423AQ44690516-405D7D3D-2F80-4CC3-8136-FFD8B371A567Q44736772-CD79557D-1924-4256-9FB8-D77B0AA63B9CQ45923545-51B3AD0B-8202-44BB-8CEC-B4AF37BFF479
P2860
Regulation of ATP supply during muscle contraction: theoretical studies.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Regulation of ATP supply during muscle contraction: theoretical studies.
@en
type
label
Regulation of ATP supply during muscle contraction: theoretical studies.
@en
prefLabel
Regulation of ATP supply during muscle contraction: theoretical studies.
@en
P2860
P356
P1433
P1476
Regulation of ATP supply during muscle contraction: theoretical studies.
@en
P2860
P304
P356
10.1042/BJ3301189
P407
P478
330 ( Pt 3)
P577
1998-03-01T00:00:00Z