Cell-derived anaphylatoxins as key mediators of antibody-dependent type II autoimmunity in mice.
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Mechanisms involved in antibody- and complement-mediated allograft rejectionThe perfect storm: HLA antibodies, complement, FcγRs, and endothelium in transplant rejectionComplement: a key system for immune surveillance and homeostasisNK-cell activation and antibody-dependent cellular cytotoxicity induced by rituximab-coated target cells is inhibited by the C3b component of complement.Mechanisms of action of intravenous immunoglobulins.Siglec-5 and Siglec-14 are polymorphic paired receptors that modulate neutrophil and amnion signaling responses to group B Streptococcus.The role of the anaphylatoxins in health and disease.Regulation of human neutrophil Fcγ receptor IIa by C5a receptor promotes inflammatory arthritis in mice.Differential regulation of proinflammatory mediators following LPS- and ATP-induced activation of monocytes from patients with antiphospholipid syndrome.An essential role for Stat3 in regulating IgG immune complex-induced pulmonary inflammation.Genomic regulation of senescence and innate immunity signaling in the retinal pigment epithelium.C5a-regulated CCAAT/enhancer-binding proteins β and δ are essential in Fcγ receptor-mediated inflammatory cytokine and chemokine production in macrophages.Protection against inflammation- and autoantibody-caused fetal loss by the chemokine decoy receptor D6.The role of complement in inflammatory diseases from behind the scenes into the spotlightCooperative and alternate functions for STIM1 and STIM2 in macrophage activation and in the context of inflammationNew concepts of complement in allorecognition and graft rejectionThe C5a receptor has a key role in immune complex glomerulonephritis in complement factor H-deficient miceExtravasations and emigration of neutrophils to the inflammatory site depend on the interaction of immune-complex with Fcgamma receptors and can be effectively blocked by decoy Fcgamma receptors.Neutrophils orchestrate their own recruitment in murine arthritis through C5aR and FcγR signaling.Autoimmune hemolytic anaemia in the antiphospholipid syndrome.Use of mouse models to study the mechanisms and consequences of RBC clearance.IFN-gamma and IL-17 production in experimental autoimmune encephalomyelitis depends on local APC-T cell complement productionComplement in immune and inflammatory disorders: pathophysiological mechanismsC5a is not involved in experimental autoimmune myasthenia gravis pathogenesis.Autoantibody-mediated arthritis in the absence of C3 and activating Fcγ receptors: C5 is activated by the coagulation cascade.An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.Abrogation of immune complex glomerulonephritis by native carboxypeptidase and pharmacological antagonism of the C5a receptor.Fcγ receptor upregulation is associated with immune complex inflammation in the mouse retina and early age-related macular degeneration.The other side of immunoglobulin G: suppressor of inflammation.Epidermolysis bullosa acquisita.Molecular basis for downregulation of C5a-mediated inflammation by IgG1 immune complexes in allergy and asthma.Update on crescentic glomerulonephritis.A Complement-Optimized EGFR Antibody Improves Cytotoxic Functions of Polymorphonuclear Cells against Tumor Cells.Innate immune mediators in cancer: between defense and resistance.Lack of Fc Gamma Receptor IIIA Promotes Rather than Suppresses Humoral and Cellular Immune Responses after Mucosal or Parenteral Immunization with Antigen and Adjuvants.S100A8 causes a shift toward expression of activatory Fcγ receptors on macrophages via toll-like receptor 4 and regulates Fcγ receptor expression in synovium during chronic experimental arthritis.Characterization of the murine IgG Fc receptor III and IIB gene promoters: a single two-nucleotide difference determines their inverse responsiveness to C5a.Orai1 controls C5a-induced neutrophil recruitment in inflammation.C5a receptor enables participation of mast cells in immune complex arthritis independently of Fcγ receptor modulation.C5a and Fcgamma receptors: a mutual admiration society.
P2860
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P2860
Cell-derived anaphylatoxins as key mediators of antibody-dependent type II autoimmunity in mice.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Cell-derived anaphylatoxins as ...... type II autoimmunity in mice.
@en
type
label
Cell-derived anaphylatoxins as ...... type II autoimmunity in mice.
@en
prefLabel
Cell-derived anaphylatoxins as ...... type II autoimmunity in mice.
@en
P2093
P2860
P356
P1476
Cell-derived anaphylatoxins as ...... type II autoimmunity in mice.
@en
P2093
J Engelbert Gessner
J Sjef Verbeek
Jörg Zwirner
Reinhold E Schmidt
Stephanie Konrad
Syed R Ali
Varsha Kumar
P2860
P304
P356
10.1172/JCI25536
P407
P577
2006-02-01T00:00:00Z