The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid.

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EVI1 promotes tumor growth via transcriptional repression of MS4A3.Disease-stabilizing treatment based on all-trans retinoic acid and valproic acid in acute myeloid leukemia - identification of responders by gene expression profiling of pretreatment leukemic cells

P2860

Q38299701-7B11D3AC-DCC9-4898-80B3-75E41B01CFBB Q41625772-E9BEDA84-BD7F-4712-9306-A8F81765422A

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The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid.

Item

http://www.wikidata.org/entity/Q41930387

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

The oncogene EVI1 enhances tra ...... ls to all-trans retinoic acid.

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type

Item

label

The oncogene EVI1 enhances tra ...... ls to all-trans retinoic acid.

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prefLabel

The oncogene EVI1 enhances tra ...... ls to all-trans retinoic acid.

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P1476

The oncogene EVI1 enhances tra ...... ls to all-trans retinoic acid.

@en

P2093

Birgit Steinmetz

http://www.w3.org/2001/XMLSchema#string

Eva Slabáková

http://www.w3.org/2001/XMLSchema#string

Ilse Schwarzinger

http://www.w3.org/2001/XMLSchema#string

Itziar Arbesu

http://www.w3.org/2001/XMLSchema#string

Karel Souček

http://www.w3.org/2001/XMLSchema#string

Medhat Shehata

http://www.w3.org/2001/XMLSchema#string

Monika Smějová

http://www.w3.org/2001/XMLSchema#string

P2860

The evi-1 oncoprotein inhibits c-Jun N-terminal kinase and prevents stress-induced cell death

Identification of a retinoic acid responsive element in the retinoic acid receptor beta gene

Oncogenic transcription factor Evi1 regulates hematopoietic stem cell proliferation through GATA-2 expression

MN1 affects expression of genes involved in hematopoiesis and can enhance as well as inhibit RAR/RXR-induced gene expression

Prognostic importance of MN1 transcript levels, and biologic insights from MN1-associated gene and microRNA expression signatures in cytogenetically normal acute myeloid leukemia: a cancer and leukemia group B study

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The impact on outcome of the addition of all-trans retinoic acid to intensive chemotherapy in younger patients with nonacute promyelocytic acute myeloid leukemia: ...

Embryonic retinoic acid synthesis is essential for early mouse post-implantation development

EVI1 is expressed in megakaryocyte cell lineage and enforced expression of EVI1 in UT-7/GM cells induces megakaryocyte differentiation

The corepressor CtBP interacts with Evi-1 to repress transforming growth factor beta signaling

P304

2931-2943

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P31

scholarly article

P356

10.4161/15384101.2014.946869

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Hubert Hackl

Andreas Spittler

Rotraud Wieser

P577

2014-01-01T00:00:00Z

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P5875

269281701

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P698

25486480

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P932

4613657

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The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid.

about

P2860

P2860

The oncogene EVI1 enhances transcriptional and biological responses of human myeloid cells to all-trans retinoic acid.

description

name

type

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P932