Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodelling in mice. - Wikidata - awgldk - TriplyDB

Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodelling in mice.

Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodelling in mice.

http://www.wikidata.org/entity/Q41941377

about

Redox modification of nuclear actin by MICAL-2 regulates SRF signaling Local delivery of novel MRTF/SRF inhibitors prevents scar tissue formation in a preclinical model of fibrosis.Genome-wide RNA-Sequencing analysis identifies a distinct fibrosis gene signature in the conjunctiva after glaucoma surgery RPEL proteins are the molecular targets for CCG-1423, an inhibitor of Rho signaling.Myocardin regulates vascular smooth muscle cell inflammatory activation and disease Megakaryoblastic leukemia-1 is required for the development of bleomycin-induced pulmonary fibrosis A Role of Myocardin Related Transcription Factor-A (MRTF-A) in Scleroderma Related Fibrosis G Protein-Coupled Receptor and RhoA-Stimulated Transcriptional Responses: Links to Inflammation, Differentiation, and Cell Proliferation.Stereospecific Inhibitory Effects of CCG-1423 on the Cellular Events Mediated by Myocardin-Related Transcription Factor A.Loss of the mechanotransducer zyxin promotes a synthetic phenotype of vascular smooth muscle cells.Receptor-targeted liposome-peptide-siRNA nanoparticles represent an efficient delivery system for MRTF silencing in conjunctival fibrosis.Extracellular matrix synthesis in vascular disease: hypertension, and atherosclerosis.Myocardin in biology and disease.Divergent Regulation of Actin Dynamics and Megakaryoblastic Leukemia-1 and -2 (Mkl1/2) by cAMP in Endothelial and Smooth Muscle Cells.MRTFs- master regulators of EMT.Injury induced expression of caveolar proteins in human kidney tubules - role of megakaryoblastic leukemia 1.Myocardin-related transcription factor A cooperates with brahma-related gene 1 to activate P-selectin transcription.Lysophosphatidic acid signaling through its receptor initiates profibrotic epithelial cell fibroblast communication mediated by epithelial cell derived connective tissue growth factor.Pro-B-type natriuretic peptide is cleaved intracellularly: impact of distance between O-glycosylation and cleavage sites.HADC5 deacetylates MKL1 to dampen TNF-α induced pro-inflammatory gene transcription in macrophages.Loss of SMAD3 Promotes Vascular Remodeling in Pulmonary Arterial Hypertension via MRTF Disinhibition.Genotype-Phenotype Associations of IL6 and PRG4 With Conjunctival Fibrosis After Glaucoma Surgery.Nexilin/NEXN controls actin polymerization in smooth muscle and is regulated by myocardin family coactivators and YAP

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P2860

Reciprocal expression of MRTF-A and myocardin is crucial for pathological vascular remodelling in mice.

http://www.wikidata.org/entity/Q41941377

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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name

Reciprocal expression of MRTF- ...... vascular remodelling in mice.

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Reciprocal expression of MRTF- ...... vascular remodelling in mice.

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Reciprocal expression of MRTF- ...... vascular remodelling in mice.

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The EMBO Journal

P1476

Reciprocal expression of MRTF- ...... l vascular remodelling in mice

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P2093

Akinori Kimura

http://www.w3.org/2001/XMLSchema#string

Chinatsu Yamada

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Hideyuki Kinoshita

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Junko Shibata

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Kazuhiro Nakao

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Kazuwa Nakao

http://www.w3.org/2001/XMLSchema#string

Kenji Sobue

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Kenji Ueshima

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Koichiro Kuwahara

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Masataka Sata

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P2860

Potentiation of serum response factor activity by a family of myocardin-related transcription factors.

Muscle-specific signaling mechanism that links actin dynamics to serum response factor.

Modulation of adverse cardiac remodeling by STARS, a mediator of MEF2 signaling and SRF activity

Myocardin is a master regulator of smooth muscle gene expression

MicroRNA-1 inhibits myocardin-induced contractility of human vascular smooth muscle cells

Induction of microRNA-1 by myocardin in smooth muscle cells inhibits cell proliferation

DeltaEF1 mediates TGF-beta signaling in vascular smooth muscle cell differentiation

Identification of a novel transcriptional activator, BSAC, by a functional cloning to inhibit tumor necrosis factor-induced cell death

Activation of cardiac gene expression by myocardin, a transcriptional cofactor for serum response factor

The serum response factor coactivator myocardin is required for vascular smooth muscle development

P304

4428-4440

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10.1038/EMBOJ.2012.296

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23

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31

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Hiroyasu Nakano

Yutaka Kawahito

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2012-10-26T00:00:00Z

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232720251

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23103763

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3512386

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