p50-NF-kappaB complexes partially compensate for the absence of RelB: severely increased pathology in p50(-/-)relB(-/-) double-knockout mice.
about
Differential utilization of NF-kappaB RELA and RELB in response to extracellular versus intracellular polyIC stimulation in HT1080 cells.Utilization of genetically altered animals in the pharmaceutical industry.On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease.Crucial role for BAFF-BAFF-R signaling in the survival and maintenance of mature B cells.Targeted disruption of NF-{kappa}B1 (p50) augments cigarette smoke-induced lung inflammation and emphysema in mice: a critical role of p50 in chromatin remodelingGenetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.It's got you covered. NF-kappaB in the epidermis.Defective regulation of CXCR2 facilitates neutrophil release from bone marrow causing spontaneous inflammation in severely NF-kappa B-deficient mice.RelB modulation of IkappaBalpha stability as a mechanism of transcription suppression of interleukin-1alpha (IL-1alpha), IL-1beta, and tumor necrosis factor alpha in fibroblasts.NF-kappaB inhibits T-cell activation-induced, p73-dependent cell death by induction of MDM2RelB-p50 NF-kappa B complexes are selectively induced by cytomegalovirus immediate-early protein 1: differential regulation of Bcl-x(L) promoter activity by NF-kappa B family membersIKK alpha on center stage.The anti-apoptotic activities of Rel and RelA required during B-cell maturation involve the regulation of Bcl-2 expression.NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.Requirement for NF-kappaB in osteoclast and B-cell developmentRelB nuclear translocation regulates B cell MHC molecule, CD40 expression, and antigen-presenting cell function.The combined absence of the transcription factors Rel and RelA leads to multiple hemopoietic cell defects.Moderate activation of IKK2-NF-kB in unstressed adult mouse liver induces cytoprotective genes and lipogenesis without apparent signs of inflammation or fibrosis.An essential role for nuclear factor kappaB in promoting double positive thymocyte apoptosis.IKKα-mediated signaling circuitry regulates early B lymphopoiesis during hematopoiesisA role for the IkappaB family member Bcl-3 in the control of central immunologic toleranceYmer acts as a multifunctional regulator in nuclear factor-κB and Fas signaling pathways.Requirement for the NF-kappaB family member RelA in the development of secondary lymphoid organs.Cooperation between classical and alternative NF-κB pathways regulates proinflammatory responses in epithelial cells.NIK-dependent RelB activation defines a unique signaling pathway for the development of V alpha 14i NKT cells.Targeting activation of specific NF-κB subunits prevents stress-dependent atherothrombotic gene expression.The NF-κB p65/miR-23a-27a-24 cluster is a target for leukemia treatmentThe two-faced NF-kappaB in the skin.RelB: an outlier in leukocyte biology.The BLyS family: toward a molecular understanding of B cell homeostasis.Tonic B cell antigen receptor signals supply an NF-kappaB substrate for prosurvival BLyS signaling.NFκB signaling regulates embryonic and adult neurogenesis.NF-κB function in B lymphocytes.NF-κB signaling pathways: role in nervous system physiology and pathology.Nuclear factor-kappa-B signaling in lung development and disease: one pathway, numerous functions.Nuclear factor-kappaB1 (p50) limits the inflammatory and fibrogenic responses to chronic injury.Rel B-modified dendritic cells possess tolerogenic phenotype and functions on lupus splenic lymphocytes in vitro.NF-kappaB family proteins participate in multiple steps of hematopoiesis through elimination of reactive oxygen species.Glutamate-induced NFkappaB activation in the retina.Proteomic analysis of brain protein expression levels in NF-kappabeta p50 -/- homozygous knockout mice
P2860
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P2860
p50-NF-kappaB complexes partially compensate for the absence of RelB: severely increased pathology in p50(-/-)relB(-/-) double-knockout mice.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
p50-NF-kappaB complexes partia ...... elB(-/-) double-knockout mice.
@en
type
label
p50-NF-kappaB complexes partia ...... elB(-/-) double-knockout mice.
@en
prefLabel
p50-NF-kappaB complexes partia ...... elB(-/-) double-knockout mice.
@en
P2093
P2860
P356
P1476
p50-NF-kappaB complexes partia ...... elB(-/-) double-knockout mice.
@en
P2093
P2860
P304
P356
10.1084/JEM.185.7.1359
P407
P577
1997-04-01T00:00:00Z