Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves Bid cleavage. - Wikidata - awgldk - TriplyDB

Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves Bid cleavage.

Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves Bid cleavage.

http://www.wikidata.org/entity/Q42059521

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Protection of Bcl-2 by salubrinal Drug Carrier for Photodynamic Cancer Therapy Photodynamic Efficiency: From Molecular Photochemistry to Cell Death Autophagic death probed by photodynamic therapy Dual roles of nitric oxide in the regulation of tumor cell response and resistance to photodynamic therapy Cathepsin D--many functions of one aspartic protease Cathepsin B contributes to traumatic brain injury-induced cell death through a mitochondria-mediated apoptotic pathway Nanomechanical mechanism for lipid bilayer damage induced by carbon nanotubes confined in intracellular vesicles Novel photosensitizers trigger rapid death of malignant human cells and rodent tumor transplants via lipid photodamage and membrane permeabilization The lysosome-associated apoptosis-inducing protein containing the pleckstrin homology (PH) and FYVE domains (LAPF), representative of a novel family of PH and FYVE domain-containing proteins, induces caspase-independent apoptosis via the lysosomal-m Nanoparticles of Titanium and Zinc Oxides as Novel Agents in Tumor Treatment: a Review.Early apoptosis and cell death induced by ATX-S10Na (II)-mediated photodynamic therapy are Bax- and p53-dependent in human colon cancer cells.Bid is required in NPe6-PDT-induced apoptosis.Febrile-range hyperthermia accelerates caspase-dependent apoptosis in human neutrophils Signaling From Lysosomes Enhances Mitochondria-Mediated Photodynamic Therapy In Cancer Cells.Photosensitization reaction-induced acute electrophysiological cell response of rat myocardial cells in short loading periods of talaporfin sodium or porfimer sodium.Assessing autophagy in the context of photodynamic therapy Enhanced efficacy of photodynamic therapy via a sequential targeting protocol Imaging and photodynamic therapy: mechanisms, monitoring, and optimization.Molecular determinants of photodynamic therapy for lung cancers.Reversible effects of photodamage directed toward mitochondria Evaluation of photodynamic treatment using aluminum phthalocyanine tetrasulfonate chloride as a photosensitizer: new approach.Initiation of autophagy by photodynamic therapy.Susceptibility of myelomonocytic leukemia U937 cells to the induction of apoptosis by the non-peptidic Bcl-2 ligand HA14-1 is cell cycle phase-dependent.Adventures in photodynamic therapy: 1976-2008.A requirement for bid for induction of apoptosis by photodynamic therapy with a lysosome- but not a mitochondrion-targeted photosensitizer.Dysregulation of macrophage-secreted cathepsin B contributes to HIV-1-linked neuronal apoptosis.Nonesterified cholesterol content of lysosomes modulates susceptibility to oxidant-induced permeabilization.Effects of endosomal photodamage on membrane recycling and endocytosis.Chemotherapeutic approaches for targeting cell death pathways.Low-level light therapy potentiates NPe6-mediated photodynamic therapy in a human osteosarcoma cell line via increased ATP.Syntheses and cellular investigations of 17(3)-, 15(2)-, and 13(1)-amino acid derivatives of chlorin e(6).Photodynamic therapy of cancer: an update Destination 'lysosome': a target organelle for tumour cell killing?Photodynamic therapy and cell death pathways.Promotion of Proapoptotic Signals by Lysosomal Photodamage.Bid-cardiolipin interaction at mitochondrial contact site contributes to mitochondrial cristae reorganization and cytochrome C release Structural factors and mechanisms underlying the improved photodynamic cell killing with silicon phthalocyanine photosensitizers directed to lysosomes versus mitochondria Lysosomal signaling enhances mitochondria-mediated photodynamic therapy in A431 cancer cells: role of iron ATG7 deficiency suppresses apoptosis and cell death induced by lysosomal photodamage.

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P2860

Release of cytochrome c and activation of pro-caspase-9 following lysosomal photodamage involves Bid cleavage.

http://www.wikidata.org/entity/Q42059521

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Release of cytochrome c and ac ...... odamage involves Bid cleavage.

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Release of cytochrome c and ac ...... odamage involves Bid cleavage.

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B Chelladurai

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D Kessel

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P2860

tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

BID: a novel BH3 domain-only death agonist

Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity

In vitro photosensitization II. An electron microscopy study of cellular destruction with mono-L-aspartyl chlorin e6 and photofrin II

Dissociation of mitochondrial depolarization from cytochrome c release during apoptosis induced by photodynamic therapy

Apoptosis induced by exposure to a low steady-state concentration of H2O2 is a consequence of lysosomal rupture

Lysosomal release of cathepsin D precedes relocation of cytochrome c and loss of mitochondrial transmembrane potential during apoptosis induced by oxidative stress

The Bcl-2 protein family: arbiters of cell survival

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