Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats. - Wikidata - awgldk - TriplyDB

Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats.

Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats.

http://www.wikidata.org/entity/Q42062492

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Neuroprotection for stroke: current status and future perspectives Capillary pericytes regulate cerebral blood flow in health and disease.Effect of proline analogues on activity of human prolyl hydroxylase and the regulation of HIF signal transduction pathway.Ischemia-reperfusion injury in stroke Dimethyloxaloylglycine improves angiogenic activity of bone marrow stromal cells in the tissue-engineered bone.The effect of right vagus nerve stimulation on focal cerebral ischemia: an experimental study in the rat.Roles of individual prolyl-4-hydroxylase isoforms in the first 24 hours following transient focal cerebral ischaemia: insights from genetically modified mice.Renal protection in chronic kidney disease: hypoxia-inducible factor activation vs. angiotensin II blockade Training the brain to survive stroke Method parameters' impact on mortality and variability in rat stroke experiments: a meta-analysis.Antihelminthic benzimidazoles are novel HIF activators that prevent oxidative neuronal death via binding to tubulin.Prophylactic neuroprotection against stroke: low-dose, prolonged treatment with deferoxamine or deferasirox establishes prolonged neuroprotection independent of HIF-1 function.HIF-1α Mediates Isoflurane-Induced Vascular Protection in Subarachnoid Hemorrhage PHDs inhibitor DMOG promotes the vascularization process in the AV loop by HIF-1a up-regulation and the preliminary discussion on its kinetics in rat.Risk of Bias in Reports of In Vivo Research: A Focus for Improvement.Pharmacological stimulation of hypoxia inducible factor-1α facilitates the corticosterone response to a mild acute stressor.Hypoxia-inducible factor prolyl hydroxylase inhibition: robust new target or another big bust for stroke therapeutics?Characterization of bone marrow-derived mesenchymal stem cells from dimethyloxallyl glycine-preconditioned mice: Evaluation of the feasibility of dimethyloxallyl glycine as a mobilization agent.Dimethyloxaloylglycine Promotes the Angiogenic Activity of Mesenchymal Stem Cells Derived from iPSCs via Activation of the PI3K/Akt Pathway for Bone Regeneration.Alteplase treatment does not increase brain injury after mechanical middle cerebral artery occlusion in the rat.Dimethyloxaloylglycine increases bone repair capacity of adipose-derived stem cells in the treatment of osteonecrosis of the femoral head.Dimethyloxaloylglycine increases the bone healing capacity of adipose-derived stem cells by promoting osteogenic differentiation and angiogenic potential.Cerebral blood flow alteration in neuroprotection following cerebral ischaemia.Hypoxia inducible factor-1: its potential role in cerebral ischemia.Hypoxia-inducible factor prolyl hydroxylases as targets for neuroprotection by "antioxidant" metal chelators: From ferroptosis to stroke.Prolyl 4-hydroxylases, master regulators of the hypoxia response.Recent Advances in Developing Inhibitors for Hypoxia-Inducible Factor Prolyl Hydroxylases and Their Therapeutic Implications.Hypoxia-inducible factors as neuroprotective agent in Alzheimer's disease.HIF prolyl hydroxylase inhibition prior to transient focal cerebral ischaemia is neuroprotective in mice.In vitro ischemia suppresses hypoxic induction of hypoxia-inducible factor-1α by inhibition of synthesis and not enhanced degradation.Multi-modal assessment of neurovascular coupling during cerebral ischaemia and reperfusion using remote middle cerebral artery occlusion.Inhibition of HIF prolyl-4-hydroxylases by FG-4497 reduces brain tissue injury and edema formation during ischemic stroke.Inhibition of HIF-prolyl-4-hydroxylases prevents mitochondrial impairment and cell death in a model of neuronal oxytosis.Nitric oxide modulates hypoxia-inducible factor-1 and poly(ADP-ribose) polymerase-1 cross talk in response to hypobaric hypoxia.A role for prolyl hydroxylase domain proteins in hippocampal synaptic plasticity.Sestrin2 induced by hypoxia inducible factor1 alpha protects the blood-brain barrier via inhibiting VEGF after severe hypoxic-ischemic injury in neonatal rats.Neuroprotection in ischemic stroke: what does the future hold?Pre- and post-conditional inhibition of prolyl-4-hydroxylase domain enzymes protects the heart from an ischemic insult.Neuronal HIF-1α and HIF-2α deficiency improves neuronal survival and sensorimotor function in the early acute phase after ischemic stroke.Dimethyloxalylglycine preserves the intestinal microvasculature and protects against intestinal injury in a neonatal mouse NEC model: role of VEGF signaling.

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Neuroprotection by dimethyloxalylglycine following permanent and transient focal cerebral ischemia in rats.

http://www.wikidata.org/entity/Q42062492

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2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

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2010年论文

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name

Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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type

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Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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Neuroprotection by dimethyloxa ...... cal cerebral ischemia in rats.

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Journal of Cerebral Blood Flow & Metabolism

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Neuroprotection by dimethyloxa ...... ocal cerebral ischemia in rats

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Daniel Gallichan

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Keith J Brooks

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Lisa C Hoyte

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Michalis Papadakis

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Nicola R Sibson

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Ruoli Chen

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Simon Nagel

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P2860

A conserved family of prolyl-4-hydroxylases that modify HIF

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

Expanding chemical biology of 2-oxoglutarate oxygenases

Heterozygous deficiency of PHD2 restores tumor oxygenation and inhibits metastasis via endothelial normalization.

Reperfusion injury following cerebral ischemia: pathophysiology, MR imaging, and potential therapies.

MRI reveals that early changes in cerebral blood volume precede blood-brain barrier breakdown and overt pathology in MS-like lesions in rat brain.

Early postischemic hyperperfusion: pathophysiologic insights from positron emission tomography.

Activation of hypoxia-inducible factors in hyperoxia through prolyl 4-hydroxylase blockade in cells and explants of primate lung

Erythropoietin-mediated neuroprotection involves cross-talk between Jak2 and NF-kappaB signalling cascades.

Hypoxia-inducible factor 1: control of oxygen homeostasis in health and disease.

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132-143

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10.1038/JCBFM.2010.60

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1

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31

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Alastair Buchan

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2010-04-21T00:00:00Z

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43227354

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20407463

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cerebral infarction

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3049478

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