Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers - Wikidata - awgldk - TriplyDB

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

http://www.wikidata.org/entity/Q42112477

about

Topical Collagen-Based Biomaterials for Chronic Wounds: Rationale and Clinical Application Cellular and molecular mechanisms of repair in acute and chronic wound healing Palatogenesis and cutaneous repair: A two-headed coin Induction of specific microRNAs inhibits cutaneous wound healing Chronic wound repair and healing in older adults: current status and future research.Clinical challenges of chronic wounds: searching for an optimal animal model to recapitulate their complexity Attenuation of the transforming growth factor beta-signaling pathway in chronic venous ulcers Exploring a Role for Regulatory miRNAs In Wound Healing during Ageing:Involvement of miR-200c in wound repair.Utility of a human-mouse xenograft model and in vivo near-infrared fluorescent imaging for studying wound healing Differential keratin expression during epiboly in a wound model of bioengineered skin and in human chronic wounds Wound edge biopsy sites in chronic wounds heal rapidly and do not result in delayed overall healing of the wounds.Transforming growth factor Beta 3 is required for excisional wound repair in vivo Identification of a transcriptional signature for the wound healing continuum Deregulation of epidermal stem cell niche contributes to pathogenesis of nonhealing venous ulcers Three-dimensional human tissue models that incorporate diabetic foot ulcer-derived fibroblasts mimic in vivo features of chronic wounds.Comparative Genomic, MicroRNA, and Tissue Analyses Reveal Subtle Differences between Non-Diabetic and Diabetic Foot Skin.Advanced oxidative protein products induced human keratinocyte apoptosis through the NOX-MAPK pathway.Biology and Biomarkers for Wound Healing.Acute and impaired wound healing: pathophysiology and current methods for drug delivery, part 1: normal and chronic wounds: biology, causes, and approaches to care.Desmoglein-1/Erbin interaction suppresses ERK activation to support epidermal differentiation.The skinny on Slug.Primary cultured fibroblasts derived from patients with chronic wounds: a methodology to produce human cell lines and test putative growth factor therapy such as GMCSF.Wound repair and regeneration: mechanisms, signaling, and translation Operative debridement of pressure ulcers.Diabetic wound regeneration using peptide-modified hydrogels to target re-epithelialization.Epidemiology and aetiology of C4-6 disease.JMJD3 and NF-κB-dependent activation of Notch1 gene is required for keratinocyte migration during skin wound healing.A bioengineered living cell construct activates an acute wound healing response in venous leg ulcers.Epidermal Differentiation in Barrier Maintenance and Wound Healing.Epithelialization in Wound Healing: A Comprehensive Review.Effects of TLC-Ag dressings on skin inflammation.Inherent differences in keratinocyte function in hidradenitis suppurativa: Evidence for the role of IL-22 in disease pathogenesis.Abnormal connexin expression in human chronic wounds.Keratinocyte galvanotaxis in combined DC and AC electric fields supports an electromechanical transduction sensing mechanism.Topical mevastatin promotes wound healing by inhibiting the transcription factor c-Myc via the glucocorticoid receptor and the long non-coding RNA Gas5.ATP Release and P2 Y Receptor Signaling are Essential for Keratinocyte Galvanotaxis.

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P2860

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

http://www.wikidata.org/entity/Q42112477

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

@en

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

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type

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Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

@en

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

@nl

prefLabel

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

@en

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

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J.1582-4934.2008.00321.X

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Journal of Cellular and Molecular Medicine

P1476

Deregulation of keratinocyte differentiation and activation: a hallmark of venous ulcers

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P2093

Agata Krzyzanowska

http://www.w3.org/2001/XMLSchema#string

Donna Brennan

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Harold Brem

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Irena Pastar

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Marjana Tomic-Canic

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Michael Golinko

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My G Mahoney

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Olivera Stojadinovic

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Sasa Vukelic

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P2860

Suprabasal desmoglein 3 expression in the epidermis of transgenic mice results in hyperproliferation and abnormal differentiation.

Onset of keratin 17 expression coincides with the definition of major epithelial lineages during skin development

Molecular markers in patients with chronic wounds to guide surgical debridement

SERCA2a, phospholamban, sarcolipin, and ryanodine receptors gene expression in children with congenital heart defects

Common ground in the transcriptional profiles of wounds and tumors

The retinoblastoma protein and cell cycle control

Epidermal tight junctions: ZO-1 and occludin are expressed in mature, developing, and affected skin and in vitro differentiating keratinocytes

eIF4E promotes nuclear export of cyclin D1 mRNAs via an element in the 3'UTR

Protocol for the successful treatment of venous ulcers

Direct evidence that involucrin is a major early isopeptide cross-linked component of the keratinocyte cornified cell envelope

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2675-2690

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10.1111/J.1582-4934.2008.00321.X

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18373736

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3828883

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