CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
about
RSK-B, a novel ribosomal S6 kinase family member, is a CREB kinase under dominant control of p38alpha mitogen-activated protein kinase (p38alphaMAPK)Reactive oxygen species-mediated p38 MAPK regulates carbon nanotube-induced fibrogenic and angiogenic responsesMitogen- and stress-activated protein kinase-1 (MSK1) is directly activated by MAPK and SAPK2/p38, and may mediate activation of CREBPRAK, a novel protein kinase regulated by the p38 MAP kinaseA family of LIM-only transcriptional coactivators: tissue-specific expression and selective activation of CREB and CREMMSK1 and MSK2 are required for the mitogen- and stress-induced phosphorylation of CREB and ATF1 in fibroblastsMAP kinase pathways in the yeast Saccharomyces cerevisiaep53 recruitment of CREB binding protein mediated through phosphorylated CREB: a novel pathway of tumor suppressor regulationUV-Induced stabilization of c-fos and other short-lived mRNAsp53-inducible wip1 phosphatase mediates a negative feedback regulation of p38 MAPK-p53 signaling in response to UV radiationRegulation of PRAK subcellular location by p38 MAP kinasesRepressors and upstream repressing sequences of the stress-regulated ENA1 gene in Saccharomyces cerevisiae: bZIP protein Sko1p confers HOG-dependent osmotic regulation.Inactivation of protein-tyrosine phosphatases as mechanism of UV-induced signal transductionRas participates in the activation of p38 MAPK by interleukin-1 by associating with IRAK, IRAK2, TRAF6, and TAK-1Deficiency of the stress kinase p38alpha results in embryonic lethality: characterization of the kinase dependence of stress responses of enzyme-deficient embryonic stem cellsp38 mitogen-activated kinase is a bidirectional regulator of human fibroblast collagenase-1 induction by three-dimensional collagen latticesGLUT4 translocation precedes the stimulation of glucose uptake by insulin in muscle cells: potential activation of GLUT4 via p38 mitogen-activated protein kinaseRequirement of mitogen-activated protein kinase kinase 3 (MKK3) for tumor necrosis factor-induced cytokine expressionProinsulin C-peptide activates cAMP response element-binding proteins through the p38 mitogen-activated protein kinase pathway in mouse lung capillary endothelial cellsReduction of spermatogenesis but not fertility in Creb3l4-deficient miceMAPKAP kinase 2 phosphorylates serum response factor in vitro and in vivoArachidonic acid activates mitogen-activated protein (MAP) kinase-activated protein kinase 2 and mediates adhesion of a human breast carcinoma cell line to collagen type IV through a p38 MAP kinase-dependent pathwayRsk-2 activity is necessary for epidermal growth factor-induced phosphorylation of CREB protein and transcription of c-fos geneStress-induced stimulation of early growth response gene-1 by p38/stress-activated protein kinase 2 is mediated by a cAMP-responsive promoter element in a MAPKAP kinase 2-independent mannerSelective activation of p38 mitogen-activated protein (MAP) kinase isoforms by the MAP kinase kinases MKK3 and MKK6FR167653 inhibits fibronectin expression and apoptosis in diabetic glomeruli and in high-glucose-stimulated mesangial cells.CREB1/ATF1 activation in photoreceptor degeneration and protection.Ionizing radiation induces cellular senescence of articular chondrocytes via negative regulation of SIRT1 by p38 kinase.Environmental factors as regulators and effectors of multistep carcinogenesis.p53-dependent elevation of p21Waf1 expression by UV light is mediated through mRNA stabilization and involves a vanadate-sensitive regulatory system.Regulation of lens gap junctions by Transforming Growth Factor beta.pp60(v-src) induction of cyclin D1 requires collaborative interactions between the extracellular signal-regulated kinase, p38, and Jun kinase pathways. A role for cAMP response element-binding protein and activating transcription factor-2 in pp60(v-Targeted deletion of Capn4 in cells of the chondrocyte lineage impairs chondrocyte proliferation and differentiationTGF-beta induces fibronectin synthesis through a c-Jun N-terminal kinase-dependent, Smad4-independent pathway.Induction of the angiogenic modulator fibroblast growth factor-binding protein by epidermal growth factor is mediated through both MEK/ERK and p38 signal transduction pathways.BRCA1 physically and functionally interacts with ATF1.Loss of epidermal p38α signaling prevents UVR-induced inflammation via acute and chronic mechanisms.Recruitment of an RNA polymerase II complex is mediated by the constitutive activation domain in CREB, independently of CREB phosphorylationCREB and the CRTC co-activators: sensors for hormonal and metabolic signalsHyperactive variants of p38alpha induce, whereas hyperactive variants of p38gamma suppress, activating protein 1-mediated transcription.
P2860
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P2860
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@en
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@nl
type
label
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@en
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@nl
prefLabel
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@en
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@nl
P2093
P2860
P356
P1433
P1476
CREB is activated by UVC through a p38/HOG-1-dependent protein kinase.
@en
P2093
C Sachsenmaier
H J Rahmsdorf
M Iordanov
P Herrlich
P2860
P304
P356
10.1093/EMBOJ/16.5.1009
P407
P577
1997-03-01T00:00:00Z