Stressing HbF synthesis: role of translation? - Wikidata - awgldk - TriplyDB

Stressing HbF synthesis: role of translation?

Stressing HbF synthesis: role of translation?

http://www.wikidata.org/entity/Q42115290

about

Translational control by heme-regulated eIF2α kinase during erythropoiesis Therapeutic interventions to disrupt the protein synthetic machinery in melanoma Therapeutic fetal-globin inducers reduce transcriptional repression in hemoglobinopathy erythroid progenitors through distinct mechanisms.

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Stressing HbF synthesis: role of translation?

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description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Stressing HbF synthesis: role of translation?

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Stressing HbF synthesis: role of translation?

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type

label

Stressing HbF synthesis: role of translation?

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Stressing HbF synthesis: role of translation?

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prefLabel

Stressing HbF synthesis: role of translation?

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Stressing HbF synthesis: role of translation?

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BLOOD-2013-06-506139

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Stressing HbF synthesis: role of translation?

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Jane-Jane Chen

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Susan Perrine

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P2860

A selective inhibitor of eIF2alpha dephosphorylation protects cells from ER stress

The unfolded protein response: from stress pathway to homeostatic regulation

Butyrate increases the efficiency of translation of gamma-globin mRNA.

Regulation of protein synthesis by the heme-regulated eIF2alpha kinase: relevance to anemias.

Heme-regulated eIF2α kinase activated Atf4 signaling pathway in oxidative stress and erythropoiesis.

Neither DNA hypomethylation nor changes in the kinetics of erythroid differentiation explain 5-azacytidine's ability to induce human fetal hemoglobin

Eukaryotic initiation factor 2α phosphorylation mediates fetal hemoglobin induction through a post-transcriptional mechanism

Fetal globin gene inducers: novel agents and new potential.

Haem-regulated eIF2alpha kinase is necessary for adaptive gene expression in erythroid precursors under the stress of iron deficiency.

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467-468

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scholarly article

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10.1182/BLOOD-2013-06-506139

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4

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