Stress fracture healing: fatigue loading of the rat ulna induces upregulation in expression of osteogenic and angiogenic genes that mimic the intramembranous portion of fracture repair.
about
Skeletal Blood Flow in Bone Repair and MaintenanceAngiogenesis and intramembranous osteogenesisMechanistic, mathematical model to predict the dynamics of tissue genesis in bone defects via mechanical feedback and mediation of biochemical factorsBmp2 in osteoblasts of periosteum and trabecular bone links bone formation to vascularization and mesenchymal stem cells.Long bone structure and strength depend on BMP2 from osteoblasts and osteocytes, but not vascular endothelial cells.Direct bone formation during distraction osteogenesis does not require TNFalpha receptors and elevated serum TNFalpha fails to inhibit bone formation in TNFR1 deficient mice.Healing of non-displaced fractures produced by fatigue loading of the mouse ulna.Differential gene expression from microarray analysis distinguishes woven and lamellar bone formation in the rat ulna following mechanical loading.Osteocyte expression of caspase-3, COX-2, IL-6 and sclerostin are spatially and temporally associated following stress fracture initiation.Short-term low-strain vibration enhances chemo-transport yet does not stimulate osteogenic gene expression or cortical bone formation in adult micePeriostin deficiency increases bone damage and impairs injury response to fatigue loading in adult miceAntagonizing the αv β3 integrin inhibits angiogenesis and impairs woven but not lamellar bone formation induced by mechanical loading.HIF-1α regulates bone formation after osteogenic mechanical loading.Restoration of regenerative osteoblastogenesis in aged mice: modulation of TNF.Skeletal anabolism, PTH, and the bone-vascular axis.Chitosan nanofiber scaffold improves bone healing via stimulating trabecular bone production due to upregulation of the Runx2/osteocalcin/alkaline phosphatase signaling pathway.Bmp2 conditional knockout in osteoblasts and endothelial cells does not impair bone formation after injury or mechanical loading in adult mice.Angiogenesis is required for stress fracture healing in rats.Quantification of skeletal blood flow and fluoride metabolism in rats using PET in a pre-clinical stress fracture model.Self-repair of rat cortical bone microdamage after fatigue loading in vivo.Nitric oxide-mediated vasodilation increases blood flow during the early stages of stress fracture healing.Adaptive and Injury Response of Bone to Mechanical Loading.Initiation and early control of tissue regeneration - bone healing as a model system for tissue regeneration.Distinct Osteomimetic Response of Androgen-Dependent and Independent Human Prostate Cancer Cells to Mechanical Action of Fluid Flow: Prometastatic Implications.Comparing histological, vascular and molecular responses associated with woven and lamellar bone formation induced by mechanical loading in the rat ulna.Hedgehog signaling mediates woven bone formation and vascularization during stress fracture healing.Subtraction micro-computed tomography of angiogenesis and osteogenesis during bone repair using synchrotron radiation with a novel contrast agent.Construction of adipose scaffold for bone repair with gene engineering bone cells.Effect of low-intensity whole-body vibration on bone defect repair and associated vascularization in mice.Molecular profile of osteoprogenitor cells seeded on allograft bone.The Interaction of BMP2-Induced Defect Healing in Rat and Fixator Stiffness Modulates Matrix Alignment and Contraction
P2860
Q26825242-B1AC9A0E-5E5D-49E8-876E-14052C0D8CB7Q27025343-91C6E84B-B94C-484A-A9E3-08046DC15F5AQ28540030-7BD6C144-D1B4-4FDB-967B-C396BAE221B2Q30544691-D2EE4B1B-E0C9-4E11-A8A1-0095127373A4Q33623854-A20F53C2-0C41-4526-B542-D0A9E3C8B873Q33636793-812E79E8-9C12-481E-B9F2-25177114B83BQ33872314-77315C9D-F52C-43F5-A902-3E1920205D6AQ34117489-6C5A45EC-A0DF-49CA-AC5F-7363A166AC47Q34171846-596ED613-A77C-402E-AB11-FFFBB98A2BAFQ34574647-23B98E30-B9E1-44F4-995C-E4A871E80FB8Q35027916-3EC549EC-27DA-4C79-B5EB-02791EB287DCQ35068663-B5EDE28F-BCFA-403A-9613-8BE508B16E6FQ35108936-45100C9E-05E9-428C-A827-814566D1AD4DQ35156038-4100D41F-DC0A-414D-A490-EE2181FE00ACQ36082714-13AF3582-34ED-4655-8936-2188289A5901Q36110956-446609DA-EC13-4897-995C-6B11C96298F0Q36268982-0952F2DD-1A7D-4D92-97B7-510A6BEB4F61Q36442977-5F46EFE8-1F03-48B2-8FE4-5BBF60C29156Q36672286-46730DB1-CD31-4086-8652-D1EE013B8A74Q36804806-F6F9E188-AE6F-42DD-90AE-3C865F10673EQ37578244-CA70A761-17F5-4038-A1B3-AD3BFEF146DEQ38090608-B5B2CF0F-9B2D-45EF-89B9-4C2A6BCD948DQ38176443-D63C87E5-265D-4D91-85CF-174F6682DC79Q38733024-29E222A0-1398-406B-B0AD-6B83A6BA7A0CQ42572957-DFFC5125-76BE-4EFE-8DAC-3E17D8DD4874Q42617306-F8C777B5-092F-4DB6-92A1-0C2BDCA57616Q45365859-EE4DF7CA-3CDB-49D4-BEF3-8B0C6191A5ECQ46643522-42C74A18-B76E-4469-97D1-415E91A4AF5FQ47976223-3D1634D2-A756-4951-BE80-091C9397B46EQ54346236-B1D9A47F-D0FB-462F-865A-9DC2CC4DB214Q57146234-5158260D-197F-4B51-AEDC-42D2F1EDD608
P2860
Stress fracture healing: fatigue loading of the rat ulna induces upregulation in expression of osteogenic and angiogenic genes that mimic the intramembranous portion of fracture repair.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Stress fracture healing: fatig ...... us portion of fracture repair.
@en
Stress fracture healing: fatig ...... us portion of fracture repair.
@nl
type
label
Stress fracture healing: fatig ...... us portion of fracture repair.
@en
Stress fracture healing: fatig ...... us portion of fracture repair.
@nl
prefLabel
Stress fracture healing: fatig ...... us portion of fracture repair.
@en
Stress fracture healing: fatig ...... us portion of fracture repair.
@nl
P2860
P1433
P1476
Stress fracture healing: fatig ...... us portion of fracture repair.
@en
P2093
Gregory R Wohl
Matthew J Silva
P2860
P304
P356
10.1016/J.BONE.2008.09.010
P577
2008-10-07T00:00:00Z