Sources of cells that contribute to atherosclerotic intimal calcification: an in vivo genetic fate mapping study. - Wikidata - awgldk - TriplyDB

Sources of cells that contribute to atherosclerotic intimal calcification: an in vivo genetic fate mapping study.

Sources of cells that contribute to atherosclerotic intimal calcification: an in vivo genetic fate mapping study.

http://www.wikidata.org/entity/Q42172818

about

A role for the endothelium in vascular calcification The short and long of noncoding sequences in the control of vascular cell phenotypes Constrictor prostanoids and uridine adenosine tetraphosphate: vascular mediators and therapeutic targets in hypertension and diabetes Vascular calcifying progenitor cells possess bidirectional differentiation potentials Smooth muscle cell plasticity: fact or fiction?Has our understanding of calcification in human coronary atherosclerosis progressed?Vitamin K-antagonists accelerate atherosclerotic calcification and induce a vulnerable plaque phenotype.Attenuation of chondrogenic transformation in vascular smooth muscle by dietary quercetin in the MGP-deficient mouse model miR-30e targets IGF2-regulated osteogenesis in bone marrow-derived mesenchymal stem cells, aortic smooth muscle cells, and ApoE-/- mice.Runx2 Expression in Smooth Muscle Cells Is Required for Arterial Medial Calcification in Mice.Vascular smooth muscle LRP6 limits arteriosclerotic calcification in diabetic LDLR-/- mice by restraining noncanonical Wnt signals CX3CR1 deficiency promotes muscle repair and regeneration by enhancing macrophage ApoE production Preexisting smooth muscle cells contribute to neointimal cell repopulation at an incidence varying widely among individual lesions.MicroRNAs 29b, 133b, and 211 Regulate Vascular Smooth Muscle Calcification Mediated by High Phosphorus MicroRNA in cardiovascular calcification: focus on targets and extracellular vesicle delivery mechanisms.Urokinase receptor mediates osteogenic differentiation of mesenchymal stem cells and vascular calcification via the complement C5a receptor.Mechanisms of vascular calcification in CKD-evidence for premature ageing?Emerging regulators of vascular smooth muscle cell function in the development and progression of atherosclerosis.Deletion of a Distal RANKL Gene Enhancer Delays Progression of Atherosclerotic Plaque Calcification in Hypercholesterolemic Mice.Arterial Calcification in Diabetes Mellitus: Preclinical Models and Translational Implications.The chronic kidney disease - Mineral bone disorder (CKD-MBD): Advances in pathophysiology.Low-Density Lipoprotein Receptor-Related Proteins in Skeletal Development and Disease.Adventitial MSC-like Cells Are Progenitors of Vascular Smooth Muscle Cells and Drive Vascular Calcification in Chronic Kidney Disease Absence of the Vitamin D Receptor Inhibits Atherosclerotic Plaque Calcification in Female Hypercholesterolemic Mice.Limited miR-17-92 overexpression drives hematologic malignancies Diabetes mellitus accelerates cartilaginous metaplasia and calcification in atherosclerotic vessels of LDLr mutant mice.COMP-lex Mechanics: Matricrine Signaling.Runx2 Deletion in Smooth muscle Cells Inhibits Vascular Osteochondrogenesis and Calcification but not Atherosclerotic Lesion Formation.Osteogenesis of heterotopically transplanted mesenchymal stromal cells in rat models of chronic kidney disease.Calcifying Matrix Vesicles and Atherosclerosis.Diverse cellular architecture of atherosclerotic plaque derives from clonal expansion of a few medial SMCs.Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression.Role of smooth muscle cells in vascular calcification: implications in atherosclerosis and arterial stiffness.Increased Calcific Aortic Valve Disease in response to a diabetogenic, procalcific diet in the LDLr-/-ApoB100/100 mouse model.Effect of vitamin D receptor activator therapy on vitamin D receptor and osteocalcin expression in circulating endothelial progenitor cells of hemodialysis patients

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P2860

Sources of cells that contribute to atherosclerotic intimal calcification: an in vivo genetic fate mapping study.

http://www.wikidata.org/entity/Q42172818

description

2012 nî lūn-bûn

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name

Sources of cells that contribu ...... vo genetic fate mapping study.

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Sources of cells that contribu ...... vo genetic fate mapping study.

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Sources of cells that contribu ...... vo genetic fate mapping study.

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Sources of cells that contribu ...... vo genetic fate mapping study.

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Cardiovascular Research

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Cecilia M Giachelli

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Elizabeth M Leaf

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Hsueh-Ying Yang

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Jie Hong Hu

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Mei Y Speer

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Ngoc B Nguyen

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Veena Naik

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P2860

Mutations in the gene encoding the human matrix Gla protein cause Keutel syndrome

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

Time-course analysis on the differentiation of bone marrow-derived progenitor cells into smooth muscle cells during neointima formation.

Overexpression of urokinase by plaque macrophages causes histological features of plaque rupture and increases vascular matrix metalloproteinase activity in aged apolipoprotein e-null mice

Myeloid CD34+CD13+ precursor cells transdifferentiate into chondrocyte-like cells in atherosclerotic intimal calcification

Spotty calcification typifies the culprit plaque in patients with acute myocardial infarction: an intravascular ultrasound study.

Expression of the SM22alpha promoter in transgenic mice provides evidence for distinct transcriptional regulatory programs in vascular and visceral smooth muscle cells.

Smooth muscle cells give rise to osteochondrogenic precursors and chondrocytes in calcifying arteries.

Assessment and significance of arterial stiffness in patients with chronic kidney disease.

Multimodality molecular imaging identifies proteolytic and osteogenic activities in early aortic valve disease.

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545-554

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10.1093/CVR/CVS126

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3

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94

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2012-03-21T00:00:00Z

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221903448

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22436847

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atherosclerosis

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