Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier - Wikidata - awgldk - TriplyDB

Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier

Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier

http://www.wikidata.org/entity/Q42259826

about

Galactic cosmic radiation leads to cognitive impairment and increased aβ plaque accumulation in a mouse model of Alzheimer's disease Immune responses at brain barriers and implications for brain development and neurological function in later life Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic rats Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease Overnutrition Determines LPS Regulation of Mycotoxin Induced Neurotoxicity in Neurodegenerative Diseases Plasma exosomal α-synuclein is likely CNS-derived and increased in Parkinson's disease.Inflammation-induced dysfunction of the low-density lipoprotein receptor-related protein-1 at the blood-brain barrier: protection by the antioxidant N-acetylcysteine.Apolipoprotein E isoform-specific effects on lipoprotein receptor processing.Impairment of paravascular clearance pathways in the aging brain.Cannabinoid receptor type-2 stimulation, blockade, and deletion alter the vascular inflammatory responses to traumatic brain injury.Detection of bacterial antigens and Alzheimer's disease-like pathology in the central nervous system of BALB/c mice following intranasal infection with a laboratory isolate of Chlamydia pneumoniae.Multi-target action of the novel anti-Alzheimer compound CHF5074: in vivo study of long term treatment in Tg2576 mice.Histamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibility Periodontal disease associates with higher brain amyloid load in normal elderly.Systemic inflammation in early neonatal mice induces transient and lasting neurodegenerative effects.Disruption in the Blood-Brain Barrier: The Missing Link between Brain and Body Inflammation in Bipolar Disorder?Blood-brain barrier dysfunction as a cause and consequence of Alzheimer's disease.Efflux transport of serum amyloid P component at the blood-brain barrier.Modulation of lipopolysaccharide-induced memory insult, γ-secretase, and neuroinflammation in triple transgenic mice by 5-lipoxygenase.Peroxisome proliferator-activated receptors and Alzheimer's disease: hitting the blood-brain barrier.The blood-brain barrier in neuroimmunology: Tales of separation and assimilation.Drug access to the central nervous system in Alzheimer's disease: preclinical and clinical insights.Actin filament-associated protein 1 (AFAP-1) is a key mediator in inflammatory signaling-induced rapid attenuation of intrinsic P-gp function in human brain capillary endothelial cells.Association Between Periodontitis and Amyloid-β Peptide in Elderly People With and Without Cognitive Impairment.Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit.Age-associated physiological and pathological changes at the blood-brain barrier: A review.Gram-negative bacterial molecules associate with Alzheimer disease pathology.The effect of systemic inflammation on human brain barrier function Peripheral Inflammation, Apolipoprotein E4, and Amyloid-β Interact to Induce Cognitive and Cerebrovascular Dysfunction.Blood-Brain Barriers in Obesity.Inflammation Combined with Ischemia Produces Myelin Injury and Plaque-Like Aggregates of Myelin, Amyloid-β and AβPP in Adult Rat Brain.Ceruloplasmin potentiates nitric oxide synthase activity and cytokine secretion in activated microglia.Effect of Linguizhugan decoction on neuroinflammation and expression disorder of the amyloid β‑related transporters RAGE and LRP‑1 in a rat model of Alzheimer's disease.P-glycoprotein (ABCB1) and Oxidative Stress: Focus on Alzheimer's Disease.Tau Proteins Cross the Blood-Brain Barrier.Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer's Disease Brain: A Review.Environmental Subconcussive Injury, Axonal Injury, and Chronic Traumatic Encephalopathy.Neuroimmune Axes of the Blood-Brain Barriers and Blood-Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions.Inhibition of 2-AG hydrolysis differentially regulates blood brain barrier permeability after injury.Lipopolysaccharide endotoxemia induces amyloid-β and p-tau formation in the rat brain.

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P2860

Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier

http://www.wikidata.org/entity/Q42259826

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2012 nî lūn-bûn

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Journal of Neuroinflammation

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Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier

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D Allan Butterfield

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Joshua B Owen

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Pehr E Hartvigson

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P2860

SRF and myocardin regulate LRP-mediated amyloid-beta clearance in brain vascular cells

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Inflammation and Alzheimer's disease

Phosphorylation of LRP1: regulation of transport and signal transduction

Graphical evaluation of blood-to-brain transfer constants from multiple-time uptake data

TLR2 is a primary receptor for Alzheimer's amyloid β peptide to trigger neuroinflammatory activation.

Cytokine and chemokine responses in serum and brain after single and repeated injections of lipopolysaccharide: multiplex quantification with path analysis.

Systemic catabolism of Alzheimer's Abeta40 and Abeta42.

Transport pathways for clearance of human Alzheimer's amyloid beta-peptide and apolipoproteins E and J in the mouse central nervous system

Lipopolysaccharide impairs blood-brain barrier P-glycoprotein function in mice through prostaglandin- and nitric oxide-independent pathways

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Michelle A Erickson

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