Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier
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Galactic cosmic radiation leads to cognitive impairment and increased aβ plaque accumulation in a mouse model of Alzheimer's diseaseImmune responses at brain barriers and implications for brain development and neurological function in later lifeFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic ratsFundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's diseaseOvernutrition Determines LPS Regulation of Mycotoxin Induced Neurotoxicity in Neurodegenerative DiseasesPlasma exosomal α-synuclein is likely CNS-derived and increased in Parkinson's disease.Inflammation-induced dysfunction of the low-density lipoprotein receptor-related protein-1 at the blood-brain barrier: protection by the antioxidant N-acetylcysteine.Apolipoprotein E isoform-specific effects on lipoprotein receptor processing.Impairment of paravascular clearance pathways in the aging brain.Cannabinoid receptor type-2 stimulation, blockade, and deletion alter the vascular inflammatory responses to traumatic brain injury.Detection of bacterial antigens and Alzheimer's disease-like pathology in the central nervous system of BALB/c mice following intranasal infection with a laboratory isolate of Chlamydia pneumoniae.Multi-target action of the novel anti-Alzheimer compound CHF5074: in vivo study of long term treatment in Tg2576 mice.Histamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibilityPeriodontal disease associates with higher brain amyloid load in normal elderly.Systemic inflammation in early neonatal mice induces transient and lasting neurodegenerative effects.Disruption in the Blood-Brain Barrier: The Missing Link between Brain and Body Inflammation in Bipolar Disorder?Blood-brain barrier dysfunction as a cause and consequence of Alzheimer's disease.Efflux transport of serum amyloid P component at the blood-brain barrier.Modulation of lipopolysaccharide-induced memory insult, γ-secretase, and neuroinflammation in triple transgenic mice by 5-lipoxygenase.Peroxisome proliferator-activated receptors and Alzheimer's disease: hitting the blood-brain barrier.The blood-brain barrier in neuroimmunology: Tales of separation and assimilation.Drug access to the central nervous system in Alzheimer's disease: preclinical and clinical insights.Actin filament-associated protein 1 (AFAP-1) is a key mediator in inflammatory signaling-induced rapid attenuation of intrinsic P-gp function in human brain capillary endothelial cells.Association Between Periodontitis and Amyloid-β Peptide in Elderly People With and Without Cognitive Impairment.Lipopolysaccharide-induced blood-brain barrier disruption: roles of cyclooxygenase, oxidative stress, neuroinflammation, and elements of the neurovascular unit.Age-associated physiological and pathological changes at the blood-brain barrier: A review.Gram-negative bacterial molecules associate with Alzheimer disease pathology.The effect of systemic inflammation on human brain barrier functionPeripheral Inflammation, Apolipoprotein E4, and Amyloid-β Interact to Induce Cognitive and Cerebrovascular Dysfunction.Blood-Brain Barriers in Obesity.Inflammation Combined with Ischemia Produces Myelin Injury and Plaque-Like Aggregates of Myelin, Amyloid-β and AβPP in Adult Rat Brain.Ceruloplasmin potentiates nitric oxide synthase activity and cytokine secretion in activated microglia.Effect of Linguizhugan decoction on neuroinflammation and expression disorder of the amyloid β‑related transporters RAGE and LRP‑1 in a rat model of Alzheimer's disease.P-glycoprotein (ABCB1) and Oxidative Stress: Focus on Alzheimer's Disease.Tau Proteins Cross the Blood-Brain Barrier.Lipopolysaccharide Associates with Amyloid Plaques, Neurons and Oligodendrocytes in Alzheimer's Disease Brain: A Review.Environmental Subconcussive Injury, Axonal Injury, and Chronic Traumatic Encephalopathy.Neuroimmune Axes of the Blood-Brain Barriers and Blood-Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions.Inhibition of 2-AG hydrolysis differentially regulates blood brain barrier permeability after injury.Lipopolysaccharide endotoxemia induces amyloid-β and p-tau formation in the rat brain.
P2860
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P2860
Lipopolysaccharide impairs amyloid β efflux from brain: altered vascular sequestration, cerebrospinal fluid reabsorption, peripheral clearance and transporter function at the blood-brain barrier
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@en
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@nl
type
label
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@en
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@nl
prefLabel
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@en
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@nl
P2093
P2860
P50
P921
P356
P1476
Lipopolysaccharide impairs amy ...... ion at the blood-brain barrier
@en
P2093
D Allan Butterfield
Joshua B Owen
Pehr E Hartvigson
P2860
P2888
P356
10.1186/1742-2094-9-150
P577
2012-06-29T00:00:00Z
P5875
P6179
1050310464