Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

about

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

Item

http://www.wikidata.org/entity/Q42318744

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@en

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@nl

type

Item

label

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@en

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@nl

prefLabel

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@en

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@nl

P1476

Bringing light into gene regulation in hematopoietic stem cells by the Mediator complex.

@en

P2093

Christian Kosan

http://www.w3.org/2001/XMLSchema#string

Maren Godmann

http://www.w3.org/2001/XMLSchema#string

P2860

MED12 controls the response to multiple cancer drugs through regulation of TGF-β receptor signaling

Genetic and Epigenetic Mechanisms That Maintain Hematopoietic Stem Cell Function

The Mediator complex: a central integrator of transcription

Structure, function, and activator-induced conformations of the CRSP coactivator

Somatic MED12 mutations are associated with poor prognosis markers in chronic lymphocytic leukemia

Evolution and impact of subclonal mutations in chronic lymphocytic leukemia

MED12 Regulates HSC-Specific Enhancers Independently of Mediator Kinase Activity to Control Hematopoiesis.

Hierarchical organization of fetal and adult hematopoietic stem cells.

Sepsis-Induced Thymic Atrophy Is Associated with Defects in Early Lymphopoiesis.

Hematopoietic stem cells reversibly switch from dormancy to self-renewal during homeostasis and repair.

P304

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

editorial

P356

10.21037/SCI.2017.01.04

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-02-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28275640

http://www.w3.org/2001/XMLSchema#string

P932

5334556

http://www.w3.org/2001/XMLSchema#string