Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

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Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

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2015年论文

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2015年论文

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name

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@en

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

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label

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@en

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@nl

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Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@en

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@nl

P1476

Diabetic cardiomyopathy: Role of epidermal growth factor receptor tyrosine kinase.

@en

P2093

Lu Cai

http://www.w3.org/2001/XMLSchema#string

Zheng Xu

http://www.w3.org/2001/XMLSchema#string

P2860

Epidermal growth factor receptor inhibition slows progression of diabetic nephropathy in association with a decrease in endoplasmic reticulum stress and an increase in autophagy

Diabetes and microvascular pathophysiology: role of epidermal growth factor receptor tyrosine kinase.

High glucose promotes pancreatic cancer cell proliferation via the induction of EGF expression and transactivation of EGFR

Activation of EGFR/ERBB2 via pathways involving ERK1/2, P38 MAPK, AKT and FOXO enhances recovery of diabetic hearts from ischemia-reperfusion injury

Activation of ErbB2 and Downstream Signalling via Rho Kinases and ERK1/2 Contributes to Diabetes-Induced Vascular Dysfunction.

EGFR signaling promotes TGFβ-dependent renal fibrosis

Chronic pharmacologic inhibition of EGFR leads to cardiac dysfunction in C57BL/6J mice.

Elevated epidermal growth factor receptor phosphorylation induces resistance artery dysfunction in diabetic db/db mice.

A novel role for epidermal growth factor receptor tyrosine kinase and its downstream endoplasmic reticulum stress in cardiac damage and microvascular dysfunction in type 1 diabetes mellitus.

Molecular mechanisms of diabetic cardiomyopathy.

P304

10-12

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P31

scholarly article

editorial

P356

10.1016/J.YJMCC.2015.04.002

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-04-10T00:00:00Z

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25865396

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5288810

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