about
Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthaseDecreased SGK1 Expression and Function Contributes to Behavioral Deficits Induced by Traumatic StressAn uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition poreA Bcl-xL-Drp1 complex regulates synaptic vesicle membrane dynamics during endocytosisThe transcription factor orthodenticle homeobox 2 influences axonal projections and vulnerability of midbrain dopaminergic neurons.The mitochondrial complex V-associated large-conductance inner membrane current is regulated by cyclosporine and dexpramipexoleBcl-xL is necessary for neurite outgrowth in hippocampal neuronsBcl-xL regulates mitochondrial energetics by stabilizing the inner membrane potential.Transcriptional regulation of mesencephalic dopaminergic neurons: the full circle of life and death.Linkage of cDNA expression profiles of mesencephalic dopaminergic neurons to a genome-wide in situ hybridization database.Recombinant adeno-associated virus type 2 pseudotypes: comparing safety, specificity, and transduction efficiency in the primate striatum. Laboratory investigation.Transcriptional regulation of their survival: the Engrailed homeobox genes.The lifelong maintenance of mesencephalic dopaminergic neurons by Nurr1 and engrailedBcl-xL in neuroprotection and plasticity.Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.The Mitochondrial Permeability Transition Pore and ATP Synthase.Analysis of gene expression changes in the rat hippocampus after deep brain stimulation of the anterior thalamic nucleus.Iron homeostasis and pulmonary hypertension: iron deficiency leads to pulmonary vascular remodeling in the rat.PrePhyloPro: phylogenetic profile-based prediction of whole proteome linkages.F1FO ATPase vesicle preparation and technique for performing patch clamp recordings of submitochondrial vesicle membranes.Isolation, culture and long-term maintenance of primary mesencephalic dopaminergic neurons from embryonic rodent brains.Physiological roles of the mitochondrial permeability transition pore.The neuregulin receptor, ErbB4, is not required for normal development and adult maintenance of the substantia nigra pars compacta.Inhibition of Bcl-xL prevents pro-death actions of ΔN-Bcl-xL at the mitochondrial inner membrane during glutamate excitotoxicity.Elevated P75NTR expression causes death of engrailed-deficient midbrain dopaminergic neurons by Erk1/2 suppression.Phylogenetic Profiling of Mitochondrial Proteins and Integration Analysis of Bacterial Transcription Units Suggest Evolution of F1Fo ATP Synthase from Multiple Modules.Erratum to: The Mitochondrial Permeability Transition Pore and ATP Synthase.Parkinson's disease protein DJ-1 regulates ATP synthase protein components to increase neuronal process outgrowth.The mitochondrial metabolic function of DJ-1 is modulated by 14-3-3βPhySpeTree: an automated pipeline for reconstructing phylogenetic species treesPost mortem examination of Parkinson's disease brains suggests decline in mitochondrial biomass, reversed by deep brain stimulation of subthalamic nucleus
P50
Q26823103-9DCA24AF-C2B7-4BD2-BEAF-52994D728957Q27346522-D4FC0B21-3DF2-4D6B-8525-6C165A8B5790Q28243052-E551A7E5-F8FA-4CE6-8101-ADD842603849Q28909766-ECA2A5E6-8840-44C6-A21F-ABD2E1C5A4C0Q33944226-E76B30A7-F0F9-4389-9824-CB77BD2DA294Q34785470-8DF4D066-4F38-4EB3-B768-FFDC90197D97Q34802591-E339BF4E-48B6-4D2D-88AF-5B39682251C3Q35406465-0CA209D2-5BAA-4145-9006-8E03BEED5FEBQ37017541-2B5C1C34-7320-4105-ACED-91A003D6D2C5Q37087104-5422BC63-F39E-40DB-8E95-7D13B71EDACAQ37171255-98223BD0-5AA2-4F4F-8DB0-A2C1D80777B8Q37592104-83704C1D-237F-4571-9881-A2E182F2E9F4Q38200738-3D4C8009-1E59-4CAA-814D-E0129880C70FQ38256367-C5BEDE6C-E67D-4B29-85B3-63DFCB2E13E9Q38626077-4D2DC832-810B-4334-B226-F5A2898EACBCQ38945146-9CF61013-6F49-4BDB-AD07-88B980F467E5Q39739174-DCE29A9C-B7CC-47C2-982F-297E14C4E8FCQ41231467-3E07CDEF-AA97-4142-A5EA-4B90B2F96333Q41575762-2C557C06-23B8-439E-A740-CCD0E7B96CEFQ41876252-33370B36-B92B-42D2-A6EA-45DB725BB3B4Q41983682-DBF76CE5-996C-4FF6-93D5-F05F94AF2B24Q42363155-296EF61F-DFB6-4E70-9BC0-CCE9001D1EF6Q42471488-E423CD80-9265-4ADF-B398-121262AF5638Q42706132-F9D54229-9BB9-48D8-9600-BF06F2DC61C0Q43147443-4AC05D1C-A905-4D2C-BEA7-69A3183C7DE4Q47280089-C66F02A3-6EE6-471C-9FCF-45C9664FCDDFQ48407895-52AB1FD0-2D6E-4C75-A560-08A9678FACC2Q64901146-B86BF8E2-EE37-43E0-8DC3-5D816C95573AQ91606425-363654D8-EE58-4169-BF3D-11583BEAA0F3Q91610711-E09D420B-DEBF-4F32-9579-E0C6C584FBC7Q92307176-F495E94A-BB76-4044-8B2E-FA7246BD72E7
P50
description
Neuroscientist
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wetenschapper
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հետազոտող
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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type
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Kambiz N. Alavian
@ast
Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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Kambiz N. Alavian
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P69
P1053
O-2831-2017
P1153
6504447840
P2798
P31
P3829
P3835
kambiz-alavian
P496
0000-0002-0653-3057