Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.
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Inhibition of the Mitochondrial Permeability Transition for Cytoprotection: Direct versus Indirect MechanismsThe novel BH3 α-helix mimetic JY-1-106 induces apoptosis in a subset of cancer cells (lung cancer, colon cancer and mesothelioma) by disrupting Bcl-xL and Mcl-1 protein-protein interactions with BakRole of the c subunit of the FO ATP synthase in mitochondrial permeability transitionBcl-2 family proteins as regulators of cancer cell invasion and metastasis: a review focusing on mitochondrial respiration and reactive oxygen speciesCell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthaseRegulation of the intrinsic apoptosis pathway by reactive oxygen speciesThe mitochondrial permeability transition pore and cancer: molecular mechanisms involved in cell deathMitochondria and mitophagy: the yin and yang of cell death controlRegulation of mitochondrial ATP synthase in cardiac pathophysiologyAn uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition poreEnergy failure: does it contribute to neurodegeneration?Bcl-xL-mediated remodeling of rod and cone synaptic mitochondria after postnatal lead exposure: electron microscopy, tomography and oxygen consumptionABT-263 enhances sensitivity to metformin and 2-deoxyglucose in pediatric glioma by promoting apoptotic cell deathA Bcl-xL-Drp1 complex regulates synaptic vesicle membrane dynamics during endocytosisHumanin Derivatives Inhibit Necrotic Cell Death in Neurons.Deregulation of mitochondrial F1FO-ATP synthase via OSCP in Alzheimer's disease.Mitochondrial flashes regulate ATP homeostasis in the heart.Multiple functions of BCL-2 family proteins.The BCL2L1 and PGAM5 axis defines hypoxia-induced receptor-mediated mitophagy.Drosophila larvae lacking the bcl-2 gene, buffy, are sensitive to nutrient stress, maintain increased basal target of rapamycin (Tor) signaling and exhibit characteristics of altered basal energy metabolism.Generation of human striatal neurons by microRNA-dependent direct conversion of fibroblastsRegulation of the H+-ATP synthase by IF1: a role in mitohormesis.PPARγ activation attenuates glycated-serum induced pancreatic beta-cell dysfunction through enhancing Pdx1 and Mafa protein stability.The mitochondrial complex V-associated large-conductance inner membrane current is regulated by cyclosporine and dexpramipexoleBcl-xL is necessary for neurite outgrowth in hippocampal neuronsExpression of animal anti-apoptotic gene Ced-9 enhances tolerance during Glycine max L.-Bradyrhizobium japonicum interaction under saline stress but reduces nodule formation.Bcl-xL regulates mitochondrial energetics by stabilizing the inner membrane potential.Cell death in genome evolutionO2 and Ca(2+) fluxes as indicators of apoptosis induced by rose bengal-mediated photodynamic therapy in human oral squamous carcinoma cells.Intermediate progenitors are increased by lengthening of the cell cycle through calcium signaling and p53 expression in human neural progenitorsNon-apoptotic functions of apoptosis-regulatory proteins.Interaction of cellular proteins with BCL-xL targeted to cytoplasmic inclusion bodies in adenovirus infected cells.Distinct roles of mitochondria- and ER-localized Bcl-xL in apoptosis resistance and Ca2+ homeostasisIntracellular and extracellular ATP coordinately regulate the inverse correlation between osteoclast survival and bone resorptionDelving deeper: MCL-1's contributions to normal and cancer biology.Programmed cell death in plants: lessons from bacteria?Functions of BCL-X L at the Interface between Cell Death and MetabolismAMPK is critical for mitochondrial function during reperfusion after myocardial ischemia.Mitochondria-mediated energy adaption in cancer: the H(+)-ATP synthase-geared switch of metabolism in human tumors.An interaction between Bcl-xL and the voltage-dependent anion channel (VDAC) promotes mitochondrial Ca2+ uptake.
P2860
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P2860
Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Bcl-xL regulates metabolic eff ...... tochondrial F1FO ATP synthase.
@en
type
label
Bcl-xL regulates metabolic eff ...... tochondrial F1FO ATP synthase.
@en
prefLabel
Bcl-xL regulates metabolic eff ...... tochondrial F1FO ATP synthase.
@en
P2093
P2860
P50
P356
P1433
P1476
Bcl-xL regulates metabolic eff ...... itochondrial F1FO ATP synthase
@en
P2093
Benjamin Flaherty
Christoph Rahner
Elizabeth A Jonas
Emma Lazrove
Ewan McNay
Gordon C Shore
Hongmei Li
J Marie Hardwick
Leon Collis
P2860
P2888
P304
P356
10.1038/NCB2330
P577
2011-09-18T00:00:00Z
P5875
P6179
1042317501