Increased amyloidogenic processing of transgenic human APP in X11-like deficient mouse brain. - Wikidata - awgldk - TriplyDB

Increased amyloidogenic processing of transgenic human APP in X11-like deficient mouse brain.

Increased amyloidogenic processing of transgenic human APP in X11-like deficient mouse brain.

http://www.wikidata.org/entity/Q42414078

about

Intracellular trafficking of the amyloid β-protein precursor (APP) regulated by novel function of X11-like.Cytoplasmic fragment of Alcadein α generated by regulated intramembrane proteolysis enhances amyloid β-protein precursor (APP) transport into the late secretory pathway and facilitates APP cleavage.Membrane-microdomain localization of amyloid β-precursor protein (APP) C-terminal fragments is regulated by phosphorylation of the cytoplasmic Thr668 residue.Serotonin 6 receptor controls Alzheimer's disease and depression Hyperpolarization-activated cyclic nucleotide gated channels: a potential molecular link between epileptic seizures and Aβ generation in Alzheimer's disease X11/Mint genes control polarized localization of axonal membrane proteins in vivo The role of lipoprotein receptors on the physiological function of APP.Can insulin signaling pathways be targeted to transport Aβ out of the brain?Multisite tyrosine phosphorylation of the N-terminus of Mint1/X11α by Src kinase regulates the trafficking of amyloid precursor protein Facilitation of brain mitochondrial activity by 5-aminolevulinic acid in a mouse model of Alzheimer's disease.Association of Neuropathological Markers in the Parietal Cortex With Antemortem Cognitive Function in Persons With Mild Cognitive Impairment and Alzheimer Disease.

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Increased amyloidogenic processing of transgenic human APP in X11-like deficient mouse brain.

http://www.wikidata.org/entity/Q42414078

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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name

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@en

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@nl

type

label

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@en

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@nl

prefLabel

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@en

Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@nl

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Molecular Neurodegeneration

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Increased amyloidogenic proces ...... 11-like deficient mouse brain.

@en

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Genzo Itoh

http://www.w3.org/2001/XMLSchema#string

Hidenori Taru

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Maho Kondo

http://www.w3.org/2001/XMLSchema#string

Maki Shiono

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Masahiro Maeda

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Norio Takei

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Saori Hata

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Takahide Matsushima

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Tohru Yamamoto

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Toshiharu Suzuki

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P2860

Interaction of a neuron-specific protein containing PDZ domains with Alzheimer's amyloid precursor protein

X11L2, a new member of the X11 protein family, interacts with Alzheimer's beta-amyloid precursor protein

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Amyloidogenic processing of the Alzheimer beta-amyloid precursor protein depends on lipid rafts

Deletion of Mint proteins decreases amyloid production in transgenic mouse models of Alzheimer's disease

The X11alpha protein slows cellular amyloid precursor protein processing and reduces Abeta40 and Abeta42 secretion

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide

APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy

Amyloid precursor protein trafficking, processing, and function

Generation and maintenance of neuronal polarity: mechanisms of transport and targeting.

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35

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scholarly article

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10.1186/1750-1326-5-35

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2010-09-15T00:00:00Z

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